Human LilrB2 Is a β-Amyloid Receptor and Its Murine Homolog PirB Regulates Synaptic Plasticity in an Alzheimer’s Model
暂无分享,去创建一个
Bradley T. Hyman | Carla J. Shatz | K. Christopher Garcia | Michael E. Birnbaum | Maja Djurisic | C. Shatz | B. Hyman | K. Garcia | M. Birnbaum | Christopher M. William | M. Djurisic | Taeho Kim | George S. Vidal | Taeho Kim
[1] R. Malinow,et al. The prion protein as a receptor for amyloid-β , 2010, Nature.
[2] R. Malinow,et al. APP Processing and Synaptic Function , 2003, Neuron.
[3] D. Borchelt,et al. Co-expression of multiple transgenes in mouse CNS: a comparison of strategies. , 2001, Biomolecular engineering.
[4] T. Takai. Paired immunoglobulin‐like receptors and their MHC class I recognition , 2005, Immunology.
[5] L. Mucke,et al. Amyloid-β–induced neuronal dysfunction in Alzheimer's disease: from synapses toward neural networks , 2010, Nature Neuroscience.
[6] M. Bear,et al. Bidirectional synaptic mechanisms of ocular dominance plasticity in visual cortex , 2008, Philosophical Transactions of the Royal Society B: Biological Sciences.
[7] J. Couchman,et al. An Epidermal MicroRNA Regulates Neuronal Migration Through Control of the Cellular Glycosylation State , 2013, Science.
[8] O. Arancio,et al. Preparation of oligomeric beta-amyloid 1-42 and induction of synaptic plasticity impairment on hippocampal slices. , 2010, Journal of visualized experiments : JoVE.
[9] C. Divino,et al. Paired immunoglobin-like receptor-B regulates the suppressive function and fate of myeloid-derived suppressor cells. , 2011, Immunity.
[10] A. Vortmeyer,et al. Alzheimer Amyloid-β Oligomer Bound to Post-Synaptic Prion Protein Activates Fyn to Impair Neurons , 2012, Nature Neuroscience.
[11] Nanxin Li,et al. mTOR-Dependent Synapse Formation Underlies the Rapid Antidepressant Effects of NMDA Antagonists , 2010, Science.
[12] M. Gallagher,et al. A specific amyloid-β protein assembly in the brain impairs memory , 2006, Nature.
[13] F. Kirchhoff,et al. Reduced Spine Density in Specific Regions of CA1 Pyramidal Neurons in Two Transgenic Mouse Models of Alzheimer's Disease , 2011, The Journal of Neuroscience.
[14] R. Huganir,et al. Arc/Arg3.1 Regulates an Endosomal Pathway Essential for Activity-Dependent β-Amyloid Generation , 2011, Cell.
[15] R. Giger,et al. Oligodendrocyte-Myelin Glycoprotein and Nogo Negatively Regulate Activity-Dependent Synaptic Plasticity , 2010, The Journal of Neuroscience.
[16] M. Giustetto,et al. Learning, AMPA receptor mobility and synaptic plasticity depend on n‐cofilin‐mediated actin dynamics , 2010, The EMBO journal.
[17] D. Alkon,et al. Protection against β-Amyloid-induced Apoptosis by Peptides Interacting with β-Amyloid* , 2007, Journal of Biological Chemistry.
[18] Junying Yuan,et al. FcγRIIb mediates amyloid-β neurotoxicity and memory impairment in Alzheimer's disease. , 2013, The Journal of clinical investigation.
[19] Bernardo L Sabatini,et al. Natural Oligomers of the Alzheimer Amyloid-β Protein Induce Reversible Synapse Loss by Modulating an NMDA-Type Glutamate Receptor-Dependent Signaling Pathway , 2007, The Journal of Neuroscience.
[20] C. Shatz,et al. Neuroprotection from Stroke in the Absence of MHCI or PirB , 2012, Neuron.
[21] T. Sakurai,et al. Pyruvate prevents the inhibition of the long-term potentiation induced by amyloid-β through protein phosphatase 2A inactivation. , 2012, Journal of Alzheimer's disease : JAD.
[22] Demetris K. Roumis,et al. Synaptic Plasticity Defect Following Visual Deprivation in Alzheimer's Disease Model Transgenic Mice , 2012, The Journal of Neuroscience.
[23] E. Dent,et al. Nucleofection and Primary Culture of Embryonic Mouse Hippocampal and Cortical Neurons , 2011, Journal of visualized experiments : JoVE.
[24] T. Yamashita,et al. Myelin suppresses axon regeneration by PIR‐B/SHP‐mediated inhibition of Trk activity , 2011, The EMBO journal.
[25] M. Vestling,et al. Amyloid-β Oligomer Specificity Mediated by the IgM Isotype – Implications for a Specific Protective Mechanism Exerted by Endogenous Auto-Antibodies , 2010, PloS one.
[26] Bernardo L Sabatini,et al. Synapses and Alzheimer's disease. , 2012, Cold Spring Harbor perspectives in biology.
[27] E. Ward,et al. Inhibitory receptors bind Angptls and support blood stem cells and leukemia development , 2012, Nature.
[28] John W. Gilbert,et al. Cellular Prion Protein Mediates Impairment of Synaptic Plasticity by Amyloid-β Oligomers , 2009, Nature.
[29] Joanna L. Jankowsky,et al. Mutant presenilins specifically elevate the levels of the 42 residue β-amyloid peptide in vivo: evidence for augmentation of a 42-specific γ secretase , 2004 .
[30] Shaomin Li,et al. Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory , 2008, Nature Medicine.
[31] C. Shatz,et al. PirB is a Functional Receptor for Myelin Inhibitors of Axonal Regeneration , 2008, Science.
[32] S. Krupenko,et al. ALDH1L1 Inhibits Cell Motility Via Dephosphorylation of Cofilin by PP1 and PP2A , 2010, Oncogene.
[33] C. Shatz,et al. PirB Restricts Ocular-Dominance Plasticity in Visual Cortex , 2006, Science.
[34] Tara Spires-Jones,et al. Amyloid β Induces the Morphological Neurodegenerative Triad of Spine Loss, Dendritic Simplification, and Neuritic Dystrophies through Calcineurin Activation , 2010, The Journal of Neuroscience.
[35] R. Malinow,et al. AMPAR Removal Underlies Aβ-Induced Synaptic Depression and Dendritic Spine Loss , 2006, Neuron.
[36] Julie Harris,et al. Reversing EphB2 depletion rescues cognitive functions in Alzheimer model , 2011, Nature.
[37] J. Ashby. References and Notes , 1999 .
[38] S. Lesné,et al. Detecting aβ*56 oligomers in brain tissues. , 2011, Methods in molecular biology.
[39] N. Daw,et al. Experience-Driven Plasticity of Visual Cortex Limited by Myelin and Nogo Receptor , 2005, Science.
[40] I Ferrer,et al. Widespread changes in dendritic spines in a model of Alzheimer's disease. , 2009, Cerebral cortex.
[41] Brian J. Bacskai,et al. Characterization of amyloid deposition in the APPswe/PS1dE9 mouse model of Alzheimer disease , 2006, Neurobiology of Disease.
[42] B. Hempstead,et al. NRH2 is a trafficking switch to regulate sortilin localization and permit proneurotrophin‐induced cell death , 2009, The EMBO journal.
[43] W. Freeman,et al. Neuroglial Expression of the MHCI Pathway and PirB Receptor Is Upregulated in the Hippocampus with Advanced Aging , 2012, Journal of Molecular Neuroscience.
[44] Christian Hölscher,et al. The Diabetes Drug Liraglutide Prevents Degenerative Processes in a Mouse Model of Alzheimer's Disease , 2011, The Journal of Neuroscience.
[45] Patrick O Kanold,et al. Multiple periods of functional ocular dominance plasticity in mouse visual cortex , 2005, Nature Neuroscience.
[46] Mark F. Bear,et al. Deprivation-induced synaptic depression by distinct mechanisms in different layers of mouse visual cortex , 2007, Proceedings of the National Academy of Sciences.
[47] J. Bamburg,et al. Actin depolymerizing factor and cofilin phosphorylation dynamics: response to signals that regulate neurite extension. , 1998, Cell motility and the cytoskeleton.
[48] Shaomin Li,et al. Soluble Oligomers of Amyloid β Protein Facilitate Hippocampal Long-Term Depression by Disrupting Neuronal Glutamate Uptake , 2009, Neuron.
[49] T. Yamashita,et al. The p75 receptor mediates axon growth inhibition through an association with PIR-B , 2011, Cell Death and Disease.