Exclusion of exogenous phosphatidylinositol‐3,4,5‐trisphosphate from neutrophil‐polarizing pseudopodia: stabilization of the uropod and cell polarity
暂无分享,去创建一个
[1] R. C. Richards,et al. Phorbol ester induces rapid actin assembly in neutrophil leucocytes independently of changes in [Ca2+]i and pHi , 1986, Journal of Muscle Research & Cell Motility.
[2] D. Cantrell,et al. Sustained and dynamic inositol lipid metabolism inside and outside the immunological synapse , 2002, Nature Immunology.
[3] M. Hallett,et al. Cytosolic free Ca2+ changes and calpain activation are required for β integrin–accelerated phagocytosis by human neutrophils , 2002, The Journal of cell biology.
[4] Marc W. Kirschner,et al. A PtdInsP3- and Rho GTPase-mediated positive feedback loop regulates neutrophil polarity , 2002, Nature Cell Biology.
[5] J. Berg,et al. Myosin X is a downstream effector of PI(3)K during phagocytosis , 2002, Nature Cell Biology.
[6] P. Hawkins,et al. Roles of PI3Ks in leukocyte chemotaxis and phagocytosis. , 2002, Current opinion in cell biology.
[7] F. Maxfield,et al. Cytoskeleton-dependent membrane domain segregation during neutrophil polarization. , 2001, Molecular biology of the cell.
[8] P. Lipp,et al. Phosphatidylinositol 3-phosphate is generated in phagosomal membranes , 2001, Current Biology.
[9] S. Grinstein,et al. Restricted Accumulation of Phosphatidylinositol 3-Kinase Products in a Plasmalemmal Subdomain during Fcγ Receptor-Mediated Phagocytosis , 2001, The Journal of cell biology.
[10] A. Hsu,et al. Phosphoinositide 3-Kinase Facilitates Antigen-stimulated Ca2+ Influx in RBL-2H3 Mast Cells via a Phosphatidylinositol 3,4,5-Trisphosphate-sensitive Ca2+Entry Mechanism* , 2001, The Journal of Biological Chemistry.
[11] Orion D. Weiner,et al. Leukocytes navigate by compass: roles of PI3Kγ and its lipid products , 2000 .
[12] Joseph C. Shope,et al. Intracellular delivery of phosphoinositides and inositol phosphates using polyamine carriers. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[13] V. Niggli,et al. A membrane‐permeant ester of phosphatidylinositol 3,4,5‐trisphosphate (PIP3) is an activator of human neutrophil migration , 2000, FEBS letters.
[14] F. Maxfield,et al. Neutrophil polarity and locomotion are associated with surface redistribution of leukosialin (CD43), an antiadhesive membrane molecule. , 2000, Blood.
[15] F. Maxfield,et al. Ca2+-dependent myosin II activation is required for uropod retraction during neutrophil migration. , 2000, Journal of cell science.
[16] Silvano Sozzani,et al. Central role for G protein-coupled phosphoinositide 3-kinase γ in inflammation , 2000 .
[17] Dianqing Wu,et al. Roles of PLC-β2 and -β3 and PI3Kγ in Chemoattractant-Mediated Signal Transduction , 2000 .
[18] W L Stanford,et al. Function of PI3Kgamma in thymocyte development, T cell activation, and neutrophil migration. , 2000, Science.
[19] J W Sedat,et al. Polarization of chemoattractant receptor signaling during neutrophil chemotaxis. , 2000, Science.
[20] I. Laffafian,et al. Lipid-assisted microinjection: introducing material into the cytosol and membranes of small cells. , 1998, Biophysical journal.
[21] M. Hallett,et al. Release of 'caged' cytosolic Ca2+ triggers rapid spreading of human neutrophils adherent via integrin engagement. , 1998, Journal of cell science.
[22] M. Hallett. Controlling the molecular motor of neutrophil chemotaxis , 1997, BioEssays : news and reviews in molecular, cellular and developmental biology.
[23] L. Koenderman,et al. Tyrosine phosphorylation-dependent activation of phosphatidylinositide 3-kinase occurs upstream of Ca2+-signalling induced by Fcgamma receptor cross-linking in human neutrophils. , 1997, The Biochemical journal.
[24] M. Hallett,et al. Near membrane Ca2+ changes resulting from store release in neutrophils: detection by FFP-18. , 1996, Cell calcium.
[25] I. Laffafian,et al. Does cytosolic free Ca2+ signal neutrophil chemotaxis in response to formylated chemotactic peptide? , 1995, Journal of cell science.
[26] P. Taylor,et al. Transient increase in phosphatidylinositol 3,4-bisphosphate and phosphatidylinositol trisphosphate during activation of human neutrophils. , 1989, The Journal of biological chemistry.
[27] M. Hallett. The Neutrophil: Cellular Biochemistry and Physiology , 1989 .