Allele frequencies at microsatellite loci: the stepwise mutation model revisited.
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N. Freimer | M. Slatkin | A. Valdés | N. Freimer | A. Valdes
[1] P. Moran,et al. Wandering distributions and the electrophoretic profile. II. , 1975, Theoretical population biology.
[2] T. Ohta,et al. Distribution of allelic frequencies in a finite population under stepwise production of neutral alleles. , 1975, Proceedings of the National Academy of Sciences of the United States of America.
[3] A. Brown,et al. Profiles of electrophoretic alleles in natural populations. , 1975, Genetical research.
[4] C. Wehrhahn. The evolution of selectively similar electrophoretically detectable alleles in finite natural populations. , 1975, Genetics.
[5] B. Weir. Testing for selective neutrality of electrophoretically detectable protein polymorphisms. , 1976, Genetics.
[6] M. Nei,et al. BOTTLENECK EFFECTS ON AVERAGE HETEROZYGOSITY AND GENETIC DISTANCE WITH THE STEPWISE MUTATION MODEL , 1977, Evolution; international journal of organic evolution.
[7] J. Kingman. A note on multidimensional models of neutral mutation. , 1977, Theoretical population biology.
[8] T. Ohta,et al. Stepwise mutation model and distribution of allelic frequencies in a finite population. , 1978, Proceedings of the National Academy of Sciences of the United States of America.
[9] R. Lewontin,et al. The sensitivity of gel electrophoresis as a detector of genetic variation. , 1979, Genetics.
[10] R. Ferrell,et al. The stepwise mutation model: an experimental evaluation utilizing hemoglobin variants. , 1980, Genetics.
[11] M. Nei,et al. Genetic differentiation of quantitative characters between populations or species: I. Mutation and random genetic drift , 1982 .
[12] J. Dausset. Le centre d'étude du polymorphisme humain. , 1986 .
[13] G. Gutman,et al. Slipped-strand mispairing: a major mechanism for DNA sequence evolution. , 1987, Molecular biology and evolution.
[14] A. Jeffreys,et al. Clustering of hypervariable minisatellites in the proterminal regions of human autosomes. , 1988, Genomics.
[15] J. Weber,et al. Abundant class of human DNA polymorphisms which can be typed using the polymerase chain reaction. , 1989, American journal of human genetics.
[16] R. Wolff,et al. Unequal crossingover between homologous chromosomes is not the major mechanism involved in the generation of new alleles at VNTR loci. , 1989, Genomics.
[17] J. Weber. Informativeness of human (dC-dA)n.(dG-dT)n polymorphisms. , 1990, Genomics.
[18] H. Cann,et al. Centre d'etude du polymorphisme humain (CEPH): collaborative genetic mapping of the human genome. , 1990, Genomics.
[19] R. Ferrell,et al. A population genetic study of six VNTR loci in three ethnically defined populations. , 1991, Genomics.
[20] M. Slatkin,et al. Pairwise comparisons of mitochondrial DNA sequences in stable and exponentially growing populations. , 1991, Genetics.
[21] J. Sutcliffe,et al. Variation of the CGG repeat at the fragile X site results in genetic instability: Resolution of the Sherman paradox , 1991, Cell.
[22] A. Jeffreys,et al. Minisatellite repeat coding as a digital approach to DNA typing , 1991, Nature.
[23] D. Nelson,et al. Triplet repeat mutations in human disease. , 1992, Science.
[24] A Roe. Correlations and interactions in random walks and population genetics. , 1992 .
[25] J. Weber,et al. Survey of human and rat microsatellites. , 1992, Genomics.
[26] D. Ledbetter,et al. Genetic mapping of four dinucleotide repeat loci, DXS453, DXS458, DXS454, and DXS424, on the X chromosome using multiplex polymerase chain reaction. , 1992, Genomics.
[27] T. Hudson,et al. Isolation and chromosomal assignment of 100 highly informative human simple sequence repeat polymorphisms. , 1992, Genomics.
[28] E S Lander,et al. A genetic map of the mouse suitable for typing intraspecific crosses. , 1992, Genetics.
[29] L. Jin,et al. Genetic variation at five trimeric and tetrameric tandem repeat loci in four human population groups. , 1992, Genomics.
[30] D. Tautz,et al. Slippage synthesis of simple sequence DNA. , 1992, Nucleic acids research.
[31] David E. Housman,et al. Molecular basis of myotonic dystrophy: Expansion of a trinucleotide (CTG) repeat at the 3′ end of a transcript encoding a protein kinase family member , 1992, Cell.
[32] J. Haines,et al. Construction of a GT polymorphism map of human 9q. , 1992, Genomics.
[33] Robert I. Richards,et al. Dynamic mutations: A new class of mutations causing human disease , 1992, Cell.