Metabolism of preconditioned myocardium: effect of loss and reinstatement of cardioprotection.
暂无分享,去创建一个
R. Jennings | K. Reimer | L. Sebbag | L. Schwartz | M. S. Crago | Robert B. Jennings | Laurent Sebbag | Lisa M. Schwartz | Mark S. Crago
[1] W. Wartman,et al. The Effect of ACTH on Experimental Myocardial Infarcts , 1955, Circulation research.
[2] G. Peirce,et al. Polarographic Oxygen, the Epicardial Electrocardiogram and Muscle Contraction in Experimental Acute Regional Ischemia of the Left Ventricle , 1958, Circulation research.
[3] P. J. Randle,et al. Regulation of glucose uptake by muscle. 3. The effects of insulin, anoxia, salicylate and 2:4-dinitrophenol on membrane transport and intracellular phosphorylation of glucose in the isolated rat heart. , 1959, The Biochemical journal.
[4] Hans Ulrich Bergmeyer,et al. Methods of Enzymatic Analysis , 2019 .
[5] M. W. Slein,et al. d-Glucose: Determination with Hexokinase and Glucose-6-phosphate Dehydrogenase , 1965 .
[6] W. Kübler,et al. Regulation of glycolysis in the ischemic and the anoxic myocardium. , 1970, Journal of molecular and cellular cardiology.
[7] O. H. Lowry,et al. CHAPTER 5 – MEASUREMENT OF ENZYME ACTIVITIES WITH PYRIDINE NUCLEOTIDES , 1972 .
[8] J R Neely,et al. Relationship between carbohydrate and lipid metabolism and the energy balance of heart muscle. , 1974, Annual review of physiology.
[9] R. Kloner,et al. The "no-reflow" phenomenon after temporary coronary occlusion in the dog. , 1974, The Journal of clinical investigation.
[10] D. Hearse,et al. Hypothermic arrest and potassium arrest: metabolic and myocardial protection during elective cardiac arrest. , 1975, Circulation research.
[11] A. Liedtke,et al. Relationship between coronary flow and adenosine triphosphate production from glycolysis and oxidative metabolism. , 1975, Recent advances in studies on cardiac structure and metabolism.
[12] J. Holsinger,et al. Effects on labile metabolites of temporal delay in freezing biopsy samples of dog myocardium in liquid nitrogen. , 1978, Cardiovascular research.
[13] R. Jennings,et al. The Changing Anatomic Reference Base of Evolving Myocardial Infarction: Underestimation of Myocardial Collateral Blood Flow and Overestimation of Experimental Anatomic Infarct Size Due to Tissue Edema, Hemorrhage and Acute Inflammation , 1979, Circulation.
[14] R. Jennings,et al. Prolonged depletion of ATP and of the adenine nucleotide pool due to delayed resynthesis of adenine nucleotides following reversible myocardial ischemic injury in dogs. , 1981, Journal of molecular and cellular cardiology.
[15] R. Jennings,et al. Effect of hypothermia on changes in high-energy phosphate production and utilization in total ischemia. , 1982, Journal of molecular and cellular cardiology.
[16] J. Holsinger,et al. Myocardial metabolism and regional myocardial blood flow in the canine left ventricle following twenty minutes of circumflex artery occlusion and reperfusion. , 1983, Journal of molecular and cellular cardiology.
[17] J. Greenfield,et al. Repetitive episodes of brief ischaemia (12 min) do not produce a cumulative depletion of high energy phosphate compounds. , 1984, Cardiovascular research.
[18] T A Johnson,et al. Effect of serial brief ischemic episodes on extracellular K+, pH, and activation in the pig. , 1985, Circulation.
[19] R. Jennings,et al. Nucleotide metabolism and cellular damage in myocardial ischemia. , 1985, Annual review of physiology.
[20] R. Jennings,et al. Effect of Reperfusion Late in the Phase of Reversible Ischemic Injury: Changes in Cell Volume, Electrolytes, Metabolites, and infrastructure , 1985, Circulation research.
[21] R. Jennings,et al. Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium. , 1986, Circulation.
[22] R. Jennings,et al. Effect of repetitive brief episodes of ischemia on cell volume, electrolytes and ultrastructure. , 1986, Journal of the American College of Cardiology.
[23] R. Jennings,et al. Myocardial ischemia revisited. The osmolar load, membrane damage, and reperfusion. , 1986, Journal of molecular and cellular cardiology.
[24] R. Jennings,et al. Therapy to reduce free radicals during early reperfusion does not limit the size of myocardial infarcts caused by 90 minutes of ischemia in dogs. , 1988, Circulation.
[25] J. Fallon,et al. Superoxide dismutase reduces reperfusion arrhythmias but fails to salvage regional function or myocardium at risk in conscious dogs. , 1989, Circulation.
[26] R. Jennings,et al. Total ischemia III: Effect of inhibition of anaerobic glycolysis. , 1989, Journal of molecular and cellular cardiology.
[27] W. Schaper,et al. Ischemic preconditioning and myocardial stunning: related consequences of brief coronary occlusion and reperfusion? , 1990, Advances in cardiology.
[28] R. Jennings,et al. Ischemic preconditioning slows energy metabolism and delays ultrastructural damage during a sustained ischemic episode. , 1990, Circulation research.
[29] J. Downey,et al. Protection Against Infarction Afforded by Preconditioning is Mediated by A1 Adenosine Receptors in Rabbit Heart , 1991, Circulation.
[30] J. Slot,et al. Translocation of the glucose transporter GLUT4 in cardiac myocytes of the rat. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[31] C. Kawai,et al. Ischemic Preconditioning Preserves Creatine Phosphate and Intracellular pH , 1991, Circulation.
[32] G. Grover,et al. Role of Myocardial ATP‐Sensitive Potassium Channels in Mediating Preconditioning in the Dog Heart and Their Possible Interaction With Adenosine A1‐Receptors , 1992, Circulation.
[33] G. Asimakis,et al. Ischemic preconditioning attenuates acidosis and postischemic dysfunction in isolated rat heart. , 1992, The American journal of physiology.
[34] R. Jennings,et al. Effect of Anti‐CD18 Antibody on Myocardial Neutrophil Accumulation and Infarct Size After Ischemia and Reperfusion in Dogs , 1993, Circulation.
[35] R. Sievers,et al. Loss of Myocardial Protection After Preconditioning Correlates With the Time Course of Glycogen Recovery Within the Preconditioned Segment , 1993, Circulation.
[36] M. Hori,et al. Ischemic Preconditioning Increases Adenosine Release and 5′‐Nucleotidase Activity During Myocardial Ischemia and Reperfusion in Dogs Implications for Myocardial Salvage , 1993, Circulation.
[37] J. Downey,et al. Preconditioning: state of the art myocardial protection. , 1993, Cardiovascular research.
[38] D. G. L. Wylen,et al. Effect of ischemic preconditioning on interstitial purine metabolite and lactate accumulation during myocardial ischemia. , 1994 .
[39] J. Downey,et al. Cellular Mechanisms in Ischemic Preconditioning: The Role of Adenosine and Protein Kinase C a , 1994, Annals of the New York Academy of Sciences.
[40] K. Uğurbil,et al. Transmural distribution of 2‐deoxyglucose uptake in normal and post‐ischemic canine myocardium , 1995, NMR in biomedicine.
[41] J. Downey,et al. Role of bradykinin in protection of ischemic preconditioning in rabbit hearts. , 1995, Circulation research.
[42] J. Downey,et al. Attenuated purine production during subsequent ischemia in preconditioned rabbit myocardium is unrelated to the mechanism of protection. , 1996, Journal of molecular and cellular cardiology.
[43] V. P. Chacko,et al. Attenuated glycogenolysis reduces glycolytic catabolite accumulation during ischemia in preconditioned rat hearts. , 1996, Circulation research.
[44] M. Schwaiger,et al. Persistent myocardial ischemia increases GLUT1 glucose transporter expression in both ischemic and non-ischemic heart regions. , 1997, Journal of molecular and cellular cardiology.
[45] R. Jennings,et al. Duration and reinstatement of myocardial protection against infarction by ischemic preconditioning in open chest dogs. , 2001, Journal of molecular and cellular cardiology.