The emerging role of T cells in pemphigus vulgaris: a systematic review
暂无分享,去创建一个
[1] R. Handgretinger,et al. Arginase 1+ IL‐10+ polymorphonuclear myeloid‐derived suppressor cells are elevated in patients with active pemphigus and correlate with an increased Th2/Th1 response , 2021, Experimental dermatology.
[2] Local infiltration of tissue-resident memory CD4+ T cells in lesions of patients with pemphigus and its clinical implications , 2020, Chinese Journal of Dermatology.
[3] R. Pollmann,et al. Immunophenotyping in pemphigus reveals a Th17/Tfh17-dominated immune response promoting desmoglein1/3-specific autoantibody production. , 2020, The Journal of allergy and clinical immunology.
[4] Gang Wang,et al. The role of T cells in pemphigus vulgaris and bullous pemphigoid. , 2020, Autoimmunity reviews.
[5] J. Shen,et al. Functional inhibition of regulatory CD4+CD25+T cells in peripheral blood of patients with pemphigus vulgaris , 2020, Clinical and experimental dermatology.
[6] Azin Mirzazadeh,et al. Pathogenic and protective roles of cytokines in pemphigus: A systematic review. , 2020, Cytokine.
[7] Y. Tsuchida,et al. CD4+CD25+LAG3+ T Cells With a Feature of Th17 Cells Associated With Systemic Lupus Erythematosus Disease Activity , 2019, Front. Immunol..
[8] R. Pollmann,et al. Identification of Autoreactive B Cell Subpopulations in Peripheral Blood of Autoimmune Patients With Pemphigus Vulgaris , 2019, Front. Immunol..
[9] V. Chan,et al. Pathogenic role of tissue-resident memory T cells in autoimmune diseases. , 2018, Autoimmunity reviews.
[10] D. Hafler,et al. Regulatory T cells in autoimmune disease , 2018, Nature Immunology.
[11] Q. Lu,et al. Regulatory effects of Nr4a2 on Th2 cells from patients with pemphigus vulgaris , 2018, Oncotarget.
[12] Vinod K. Sharma,et al. T helper type 1 polarizing γδ T cells and Scavenger receptors contribute to the pathogenesis of Pemphigus vulgaris , 2018, Immunology.
[13] Jie Zheng,et al. Pivotal Role of Lesional and Perilesional T/B Lymphocytes in Pemphigus Pathogenesis. , 2017, The Journal of investigative dermatology.
[14] B. Silva-Santos,et al. IL-17+ γδ T cells as kick-starters of inflammation , 2017, Nature Immunology.
[15] L. Folkersen,et al. CD49a Expression Defines Tissue-Resident CD8+ T Cells Poised for Cytotoxic Function in Human Skin , 2017, Immunity.
[16] C. Hammers,et al. Mechanisms of Disease: Pemphigus and Bullous Pemphigoid. , 2016, Annual review of pathology.
[17] Vinod K. Sharma,et al. Distinctive Treg associated CCR4-CCL22 expression profile with altered frequency of Th17/Treg cell in the immunopathogenesis of Pemphigus Vulgaris. , 2015, Immunobiology.
[18] L. Rashed,et al. Study of T helper 1 and T helper 2 responses in pemphigus vulgaris patients receiving interferon alpha 2a injections in addition to a standard protocol therapy: a randomized controlled trial , 2015, Archives of Dermatological Research.
[19] Jie Zheng,et al. The imbalance of Th17 and regulatory T cells in pemphigus patients. , 2013, European journal of dermatology : EJD.
[20] Scott N. Mueller,et al. The developmental pathway for CD103+CD8+ tissue-resident memory T cells of skin , 2013, Nature Immunology.
[21] D. Zillikens,et al. Pemphigoid diseases , 2013, The Lancet.
[22] J. Craft. Follicular helper T cells in immunity and systemic autoimmunity , 2012, Nature Reviews Rheumatology.
[23] Y. Shirakata,et al. Pathogenic anti-desmoglein 3 mAbs cloned from a paraneoplastic pemphigus patient by phage display. , 2012, The Journal of investigative dermatology.
[24] K. Nakajima. Critical role of the interleukin‐23//T‐helper 17 cell axis in the pathogenesis of psoriasis , 2012, The Journal of dermatology.
[25] W. Uter,et al. Clinical activity of pemphigus vulgaris relates to IgE autoantibodies against desmoglein 3. , 2010, Clinical immunology.
[26] M. Dobre,et al. CD28 T‐cell costimulatory molecule expression in pemphigus vulgaris , 2009, Journal of the European Academy of Dermatology and Venereology : JEADV.
[27] M. Eberl,et al. A Rapid Crosstalk of Human γδ T Cells and Monocytes Drives the Acute Inflammation in Bacterial Infections , 2009, PLoS pathogens.
[28] V. Kuchroo,et al. Induction and effector functions of TH17 cells , 2008, Nature.
[29] T. Nomura,et al. Preferential recruitment of CCR6-expressing Th17 cells to inflamed joints via CCL20 in rheumatoid arthritis and its animal model , 2007, The Journal of experimental medicine.
[30] M. Essakalli,et al. Pemphigus and HLA in Morocco. , 2007, Transfusion clinique et biologique : journal de la Societe francaise de transfusion sanguine.
[31] C. Langford,et al. Distinct Cytokine-Driven Responses of Activated Blood γδ T Cells: Insights into Unconventional T Cell Pleiotropy1 , 2007, The Journal of Immunology.
[32] N. Shibagaki,et al. CD4+CD25high Regulatory T Cells Are Markedly Decreased in Blood of Patients with Pemphigus Vulgaris , 2007, Dermatology.
[33] M. Hussein,et al. Immunohistological analysis of immune cells in blistering skin lesions , 2007, Journal of Clinical Pathology.
[34] Tin Wee Tan,et al. Prediction of desmoglein-3 peptides reveals multiple shared T-cell epitopes in HLA DR4- and DR6- associated Pemphigus vulgaris , 2006, BMC Bioinformatics.
[35] S. Chow,et al. Direct characterization of human T cells in pemphigus vulgaris reveals elevated autoantigen‐specific Th2 activity in association with active disease , 2005, Clinical and experimental dermatology.
[36] M. Dorf,et al. Recruitment of Foxp3+ T regulatory cells mediating allograft tolerance depends on the CCR4 chemokine receptor , 2005, The Journal of experimental medicine.
[37] G. Schuler,et al. Type I Regulatory T Cells Specific for Desmoglein 3 Are More Frequently Detected in Healthy Individuals than in Patients with Pemphigus Vulgaris1 , 2004, The Journal of Immunology.
[38] Wolfgang Uter,et al. T Cell Recognition of Desmoglein 3 Peptides in Patients with Pemphigus Vulgaris and Healthy Individuals1 , 2004, The Journal of Immunology.
[39] W. Uter,et al. Dichotomy of Autoreactive Th1 and Th2 Cell Responses to Desmoglein 3 in Patients with Pemphigus Vulgaris (PV) and Healthy Carriers of PV-Associated HLA Class II Alleles1 , 2003, The Journal of Immunology.
[40] D. Kabelitz,et al. Differentiation of Resting Human Peripheral Blood γδ T Cells toward Th1- or Th2-Phenotype , 2001 .
[41] M. Caproni,et al. Further support for a role for Th2-like cytokines in blister formation of pemphigus. , 2001, Clinical immunology.
[42] R. Eming,et al. Frequency analysis of autoreactive T‐helper 1 and 2 cells in bullous pemphigoid and pemphigus vulgaris by enzyme‐linked immunospot assay , 2000, The British journal of dermatology.
[43] M. Hertl,et al. Analysis of the T Cells that Are Potentially Involved in Autoantibody Production in Pemphigus Vulgaris , 1999, The Journal of dermatology.
[44] R. Dummer,et al. Evidence for cell‐mediated immune mechanisms in the pathology of pemphigus , 1993, The British journal of dermatology.
[45] B. Hazaz,et al. Determination of IgG subclasses in patients with pemphigus with active disease and in remission. , 1989, Archives of dermatology.
[46] N. Mashkilleyson. T- and B-lymphocytes in pemphigus vulgaris. A clinico-immunological follow-up study of fifty patients with pemphigus vulgaris. , 1987, Acta Dermato-Venereologica.
[47] R. Schroff,et al. Production of pemphigus antibodyin vitro and analysis of T-cell subsets , 1983, Journal of Clinical Immunology.
[48] S. Schwartz,et al. Subpopulations of human T lymphocytes. VII. Cellular basis of concanavalin A-induced T cell-mediated suppression of immunoglobulin production by B lymphocytes from normal humans. , 1979, Cellular immunology.
[49] N. Abdou,et al. Suppressor-cell dysfunction in systemic lupus erythematosus. Cells involved and in vitro correction. , 1978, The Journal of clinical investigation.
[50] B. Doft,et al. Increased spontaneous activity of antibody-forming cells in the peripheral blood of patients with active SLE. , 1977, Arthritis and rheumatism.
[51] M. Ferrarini,et al. Subpopulations of human T cells identified by receptors for immunoglobulins and mitogen responsiveness. , 1976, Journal of immunology.
[52] W. O. Porter. On Pemphigus , 1819, Edinburgh medical and surgical journal.
[53] Zhou Zai-gao. Spectratyping analysis of complementarity-determining region 3 of T-cell receptor beta chain variable region in peripheral blood mononuclear cells of patients with pemphigus vulgaris , 2009 .
[54] T. Yamamura,et al. Th17 Cells and autoimmune encephalomyelitis (EAE/MS). , 2008, Allergology international : official journal of the Japanese Society of Allergology.
[55] A. Ahmed,et al. Lymphocyte studies in pemphigus , 2004, Archives of Dermatological Research.
[56] J. Casanova,et al. X-linked neonatal diabetes mellitus, enteropathy and endocrinopathy syndrome is the human equivalent of mouse scurfy , 2001, Nature Genetics.
[57] N. Mashkilleyson,et al. Local involvement of antigen-presenting cells and activated T cells in perilesional and clinically uninvolved skin in pemphigus vulgaris. , 1989, Acta dermato-venereologica.