Genome-wide analysis of glucocorticoid receptor-binding sites in myotubes identifies gene networks modulating insulin signaling
暂无分享,去创建一个
T. Speed | T. Kuo | Oleg Mayba | Jen-Chywan Wang | C. Harris | M. Lew
[1] S. Bodine,et al. Muscle sparing in muscle RING finger 1 null mice: response to synthetic glucocorticoids , 2011, The Journal of physiology.
[2] M. Tong,et al. Over-expression of NYGGF4 (PID1) inhibits glucose transport in skeletal myotubes by blocking the IRS1/PI3K/AKT insulin pathway. , 2011, Molecular genetics and metabolism.
[3] Keiichi Fukuda,et al. Crosstalk between glucocorticoid receptor and nutritional sensor mTOR in skeletal muscle. , 2011, Cell metabolism.
[4] B. Walker,et al. Glucocorticoid-Mediated Inhibition of Angiogenic Changes in Human Endothelial Cells Is Not Caused by Reductions in Cell Proliferation or Migration , 2010, PloS one.
[5] Terence P. Speed,et al. Genome-Wide Analysis of Glucocorticoid Receptor Binding Regions in Adipocytes Reveal Gene Network Involved in Triglyceride Homeostasis , 2010, PloS one.
[6] S. Price,et al. FOXO3a mediates signaling crosstalk that coordinates ubiquitin and atrogin‐1/MAFbx expression during glucocorticoid‐induced skeletal muscle atrophy , 2010, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[7] W. Mitch,et al. Endogenous glucocorticoids and impaired insulin signaling are both required to stimulate muscle wasting under pathophysiological conditions in mice. , 2009, The Journal of clinical investigation.
[8] D. Sabatini,et al. DEPTOR Is an mTOR Inhibitor Frequently Overexpressed in Multiple Myeloma Cells and Required for Their Survival , 2009, Cell.
[9] W. Bauman,et al. Dependence of dexamethasone-induced Akt/FOXO1 signaling, upregulation of MAFbx, and protein catabolism upon the glucocorticoid receptor. , 2009, Biochemical and biophysical research communications.
[10] S. Bodine,et al. The glucocorticoid receptor and FOXO1 synergistically activate the skeletal muscle atrophy-associated MuRF1 gene. , 2008, American journal of physiology. Endocrinology and metabolism.
[11] P. Hasselgren,et al. Dexamethasone and corticosterone induce similar, but not identical, muscle wasting responses in cultured L6 and C2C12 myotubes , 2008, Journal of cellular biochemistry.
[12] M. Karin,et al. p53 Target Genes Sestrin1 and Sestrin2 Connect Genotoxic Stress and mTOR Signaling , 2008, Cell.
[13] Pan Du,et al. lumi: a pipeline for processing Illumina microarray , 2008, Bioinform..
[14] M. Birnbaum,et al. The role of FoxO in the regulation of metabolism , 2008, Oncogene.
[15] J. Thissen,et al. Mechanisms of glucocorticoid-induced myopathy. , 2008, The Journal of endocrinology.
[16] L. Gathercole,et al. Glucocorticoid modulation of insulin signaling in human subcutaneous adipose tissue. , 2007, The Journal of clinical endocrinology and metabolism.
[17] Hao Li,et al. Cell- and gene-specific regulation of primary target genes by the androgen receptor. , 2007, Genes & development.
[18] Panayiotis V. Benos,et al. STAMP: a web tool for exploring DNA-binding motif similarities , 2007, Nucleic Acids Res..
[19] M. Scadeng,et al. Bone marrow–specific Cap gene deletion protects against high-fat diet–induced insulin resistance , 2007, Nature Medicine.
[20] L. Ellisen,et al. Dexamethasone Represses Signaling through the Mammalian Target of Rapamycin in Muscle Cells by Enhancing Expression of REDD1* , 2006, Journal of Biological Chemistry.
[21] B. Draznin. Molecular Mechanisms of Insulin Resistance: Serine Phosphorylation of Insulin Receptor Substrate-1 and Increased Expression of p85α , 2006, Diabetes.
[22] Jun S. Song,et al. CEAS: cis-regulatory element annotation system. , 2006, Nucleic acids research.
[23] L. Cantley,et al. Loss of class IA PI3K signaling in muscle leads to impaired muscle growth, insulin response, and hyperlipidemia. , 2006, Cell metabolism.
[24] F. Buttgereit,et al. Non-genomic glucocorticoid effects to provide the basis for new drug developments , 2006, Molecular and Cellular Endocrinology.
[25] C. Kahn,et al. Increased P85α Is a Potent Negative Regulator of Skeletal Muscle Insulin Signaling and Induces in Vivo Insulin Resistance Associated with Growth Hormone Excess* , 2005, Journal of Biological Chemistry.
[26] J. Ofrecio,et al. Increased p85/55/50 expression and decreased phosphotidylinositol 3-kinase activity in insulin-resistant human skeletal muscle. , 2005, Diabetes.
[27] T. Fujita,et al. Dexamethasone inhibits insulin‐induced chondrogenesis of ATDC5 cells by preventing PI3K‐Akt signaling and DNA binding of Runx2 , 2004, Journal of cellular biochemistry.
[28] T. Hiragun,et al. Dexamethasone Suppresses Antigen-Induced Activation of Phosphatidylinositol 3-Kinase and Downstream Responses in Mast Cells , 2004, The Journal of Immunology.
[29] G. Yancopoulos,et al. The IGF-1/PI3K/Akt pathway prevents expression of muscle atrophy-induced ubiquitin ligases by inhibiting FOXO transcription factors. , 2004, Molecular cell.
[30] Marco Sandri,et al. Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle Atrophy , 2004, Cell.
[31] M. White,et al. Insulin receptor substrate proteins and diabetes , 2004, Archives of pharmacal research.
[32] J. Friedman,et al. Human placental growth hormone increases expression of the p85 regulatory unit of phosphatidylinositol 3-kinase and triggers severe insulin resistance in skeletal muscle. , 2004, Endocrinology.
[33] Gordon K Smyth,et al. Statistical Applications in Genetics and Molecular Biology Linear Models and Empirical Bayes Methods for Assessing Differential Expression in Microarray Experiments , 2011 .
[34] J. Klein,et al. Positive and Negative Roles of p85α and p85β Regulatory Subunits of Phosphoinositide 3-Kinase in Insulin Signaling* , 2003, Journal of Biological Chemistry.
[35] K. Yamamoto,et al. Crystallographic analysis of the interaction of the glucocorticoid receptor with DNA , 2003, Nature.
[36] G. Haegeman,et al. The interplay between the glucocorticoid receptor and nuclear factor-kappaB or activator protein-1: molecular mechanisms for gene repression. , 2003, Endocrine reviews.
[37] F. Liu,et al. Grb10 Inhibits Insulin-stimulated Insulin Receptor Substrate (IRS)-Phosphatidylinositol 3-Kinase/Akt Signaling Pathway by Disrupting the Association of IRS-1/IRS-2 with the Insulin Receptor* , 2003, The Journal of Biological Chemistry.
[38] D J Glass,et al. Identification of Ubiquitin Ligases Required for Skeletal Muscle Atrophy , 2001, Science.
[39] D. Botstein,et al. Cluster analysis and display of genome-wide expression patterns. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[40] D. Garrel,et al. Activation of the ubiquitin pathway in rat skeletal muscle by catabolic doses of glucocorticoids. , 1997, The American journal of physiology.
[41] G. Dimitriadis,et al. Effects of glucocorticoid excess on the sensitivity of glucose transport and metabolism to insulin in rat skeletal muscle. , 1997, The Biochemical journal.
[42] J. Wang,et al. Hepatic nuclear factor 3 is an accessory factor required for the stimulation of phosphoenolpyruvate carboxykinase gene transcription by glucocorticoids. , 1996, Molecular endocrinology.
[43] J. Vaughan,et al. Development of Cushing's syndrome in corticotropin-releasing factor transgenic mice. , 1992, Endocrinology.
[44] S. Egginton,et al. Angiogenesis in skeletal and cardiac muscle. , 1992, Physiological reviews.
[45] Young-Sun Lin,et al. GAL4 derivatives function alone and synergistically with mammalian activators in vitro , 1988, Cell.
[46] 中尾 玲子. Ubiquitin ligase Cbl-b is a negative regulator for insulin-like growth factor 1 signaling during muscle atrophy caused by unloading , 2010 .
[47] D. Laber,et al. 11beta-hydroxysteroid dehydrogenase type 1 regulates glucocorticoid-induced insulin resistance in skeletal muscle. , 2009, Diabetes.
[48] Michael Karin,et al. p53 Target Genes Sestrin1 and Sestrin2 Connect Genotoxic Stress and mTOR Signaling , 2009, Cell.
[49] Jae-Hyoo Kim,et al. Apoptosis of skeletal muscle on steroid-induced myopathy in rats. , 2005, The Journal of nutrition.
[50] Y. Le Marchand-Brustel,et al. Positive and negative regulation of insulin signaling through IRS-1 phosphorylation. , 2005, Biochimie.
[51] J. Klein,et al. Positive and negative roles of p85 alpha and p85 beta regulatory subunits of phosphoinositide 3-kinase in insulin signaling. , 2003, The Journal of biological chemistry.
[52] Kohjiro Ueki,et al. Reduced expression of the murine p85alpha subunit of phosphoinositide 3-kinase improves insulin signaling and ameliorates diabetes. , 2002, The Journal of clinical investigation.
[53] C. Wilson,et al. Dexamethasone inhibits insulin-stimulated recruitment of GLUT4 to the cell surface in rat skeletal muscle. , 1998, Metabolism: clinical and experimental.
[54] K. Yamamoto,et al. Regulatory crosstalk at composite response elements. , 1991, Trends in biochemical sciences.
[55] G. Bray,et al. Effects of dexamethasone on glucose transport by skeletal muscles of obese (ob/ob) mice. , 1989, International journal of obesity.
[56] R. Eston,et al. Body composition analysis: a defense of anthropometry in overweight female dieters and controls. , 1989, International journal of obesity.