Mutations in Long Terminal Repeats κB Transcription Factor Binding Sites in Plasma Virus Among South African People Living with HIV-1.
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[1] Y. Chebloune,et al. Comparative Analysis of Tat-Dependent and Tat-Deficient Natural Lentiviruses , 2015, Veterinary sciences.
[2] A. Rothman,et al. Post-translational intracellular trafficking determines the type of immune response elicited by DNA vaccines expressing Gag antigen of Human Immunodeficiency Virus Type 1 (HIV-1) , 2013, Human vaccines & immunotherapeutics.
[3] A. Mahadevan,et al. Sequence insertions in the HIV type 1 subtype C viral promoter predominantly generate an additional NF-κB binding site. , 2012, AIDS research and human retroviruses.
[4] M. Lenardo,et al. Specification of DNA binding activity of NF-kappaB proteins. , 2009, Cold Spring Harbor perspectives in biology.
[5] D. Robertson,et al. Near full-length clones and reference sequences for subtype C isolates of HIV type 1 from three different continents. , 2001, AIDS research and human retroviruses.
[6] B. Berkhout,et al. Evolution of the Human Immunodeficiency Virus Type 1 Long Terminal Repeat Promoter by Conversion of an NF-κB Enhancer Element into a GABP Binding Site , 1999, Journal of Virology.
[7] C. Williamson,et al. An association between HIV‐1 subtypes and mode of transmission in Cape Town, South Africa , 1997, AIDS.
[8] R. Desrosiers,et al. Efficient transcription and replication of simian immunodeficiency virus in the absence of NF-kappaB and Sp1 binding elements , 1996, Journal of virology.
[9] NeogiUjjwal,et al. Mutational Heterogeneity in p6 Gag Late Assembly (L) Domains in HIV-1 Subtype C Viruses from South Africa , 2016 .