Gene targeting of Desrt, a novel ARID class DNA-binding protein, causes growth retardation and abnormal development of reproductive organs.
暂无分享,去创建一个
I. Kola | L. Jermiin | J. Drago | P. Hertzog | D. Venter | D. D. de Kretser | S. Zavarsek | I. Bertoncello | M. Lahoud | S. Hasthorpe | S. Ristevski | D. M. D. Kretser
[1] R. Saint,et al. The Drosophila dead ringer gene is required for early embryonic patterning through regulation of argos and buttonhead expression. , 1999, Development.
[2] L. Parada,et al. Cryptorchidism in mice mutant for Insl3 , 1999, Nature Genetics.
[3] K. Itakura,et al. The novel Mrf-2 DNA-binding domain recognizes a five-base core sequence through major and minor-groove contacts. , 1999, Biochemical and biophysical research communications.
[4] I. Adham,et al. Targeted disruption of the Insl3 gene causes bilateral cryptorchidism. , 1999, Molecular endocrinology.
[5] L. Moore,et al. The trithorax group gene osa encodes an ARID-domain protein that genetically interacts with the brahma chromatin-remodeling factor to regulate transcription. , 1999, Development.
[6] A. Courey,et al. Dorsal-Mediated Repression Requires the Formation of a Multiprotein Repression Complex at the Ventral Silencer , 1998, Molecular and Cellular Biology.
[7] Yate-Ching Yuan,et al. A novel DNA-binding motif shares structural homology to DNA replication and repair nucleases and polymerases , 1998, Nature Structural Biology.
[8] P. Dallas,et al. p300/CREB Binding Protein-Related Protein p270 Is a Component of Mammalian SWI/SNF Complexes , 1998, Molecular and Cellular Biology.
[9] A. Rustighi,et al. NF-kappaB mediated transcriptional activation is enhanced by the architectural factor HMGI-C. , 1998, Nucleic acids research.
[10] I. Kola,et al. Cloning and Characterization of Soluble and Transmembrane Isoforms of a Novel Component of the Murine Type I Interferon Receptor, IFNAR 2* , 1997, The Journal of Biological Chemistry.
[11] G M Rubin,et al. eyelid antagonizes wingless signaling during Drosophila development and has homology to the Bright family of DNA-binding proteins. , 1997, Genes & development.
[12] N. Ling,et al. Immunosuppressive phenotype of corticotropin-releasing factor transgenic mice is reversed by adrenalectomy. , 1997, Cellular immunology.
[13] R. Saint,et al. Characterization of the dead ringer gene identifies a novel, highly conserved family of sequence-specific DNA-binding proteins , 1996, Molecular and cellular biology.
[14] M. Tymms,et al. Construction of a mouse blastocyst cDNA library by PCR amplification from total RNA , 1996, Molecular reproduction and development.
[15] Dimitris Thanos,et al. Reversal of intrinsic DNA bends in the IFNβ gene enhancer by transcription factors and the architectural protein HMG I(Y) , 1995, Cell.
[16] R. Scheuermann,et al. The immunoglobulin heavy-chain matrix-associating regions are bound by Bright: a B cell-specific trans-activator that describes a new DNA-binding protein family. , 1995, Genes & development.
[17] Xianjin Zhou,et al. Mutation responsible for the mouse pygmy phenotype in the developmentally regulated factor HMGI-C , 1995, Nature.
[18] T. Takeuchi,et al. Gene trap capture of a novel mouse gene, jumonji, required for neural tube formation. , 1995, Genes & development.
[19] Ross A. Overbeek,et al. The genetic data environment an expandable GUI for multiple sequence analysis , 1994, Comput. Appl. Biosci..
[20] J. Thompson,et al. CLUSTAL W: improving the sensitivity of progressive multiple sequence alignment through sequence weighting, position-specific gap penalties and weight matrix choice. , 1994, Nucleic acids research.
[21] K. Chada,et al. Mini-mouse: phenotypic characterization of a transgenic insertional mutant allelic to pygmy. , 1994, Genetical research.
[22] H. Chiba,et al. Two human homologues of Saccharomyces cerevisiae SWI2/SNF2 and Drosophila brahma are transcriptional coactivators cooperating with the estrogen receptor and the retinoic acid receptor. , 1994, Nucleic acids research.
[23] M. Scott,et al. Five SWI/SNF gene products are components of a large multisubunit complex required for transcriptional enhancement. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[24] L. Powell-Braxton,et al. IGF-I is required for normal embryonic growth in mice. , 1993, Genes & development.
[25] J. Baker,et al. Mice carrying null mutations of the genes encoding insulin-like growth factor I (Igf-1) and type 1 IGF receptor (Igf1r) , 1993, Cell.
[26] Elizabeth J. Robertson,et al. Role of insulin-like growth factors in embryonic and postnatal growth , 1993, Cell.
[27] Rudolf Jaenisch,et al. Targeted mutation of the DNA methyltransferase gene results in embryonic lethality , 1992, Cell.
[28] T. Roszman,et al. Immunologic disparity in the hypopituitary dwarf mouse. , 1992, Journal of immunology.
[29] M. Nissen,et al. The A.T-DNA-binding domain of mammalian high mobility group I chromosomal proteins. A novel peptide motif for recognizing DNA structure. , 1990, The Journal of biological chemistry.