Lipid-mediated endothelial dysfunction: a common factor to preeclampsia and chronic vascular disease.

[1]  F. Pipkin What Is the Place of Genetics in the Pathogenesis of Pre-Eclampsia? , 1999 .

[2]  B. Griffin,et al.  Lipoprotein atherogenicity: an overview of current mechanisms , 1999, Proceedings of the Nutrition Society.

[3]  I. Sargent,et al.  Preeclampsia: an excessive maternal inflammatory response to pregnancy. , 1999, American journal of obstetrics and gynecology.

[4]  J. Heinecke Oxidants and antioxidants in the pathogenesis of atherosclerosis: implications for the oxidized low density lipoprotein hypothesis. , 1998, Atherosclerosis.

[5]  L. Weissfeld,et al.  Small low-density lipoproteins and vascular cell adhesion molecule-1 are increased in association with hyperlipidemia in preeclampsia. , 1998, Metabolism: clinical and experimental.

[6]  G. Aykaç-toker,et al.  Imbalance between Lipid Peroxidation and Antioxidant Status in Preeclampsia , 1998, Gynecologic and Obstetric Investigation.

[7]  E. Gratacós,et al.  Lipid peroxide and vitamin E patterns in pregnant women with different types of hypertension in pregnancy. , 1998, American journal of obstetrics and gynecology.

[8]  J. Roberts,et al.  Endothelial Dysfunction in Preeclampsia , 1998, Seminars in reproductive endocrinology.

[9]  C. D. de Groot,et al.  Eicosanoid secretion by human endothelial cells exposed to normal pregnancy and preeclampsia plasma in vitro. , 1998, Prostaglandins, leukotrienes, and essential fatty acids.

[10]  D. Williams,et al.  The pathophysiology of pre-eclampsia , 1997, Intensive Care Medicine.

[11]  K. Furugori,et al.  Increased mitochondrial damage by lipid peroxidation in trophoblast cells of preeclamptic placentas , 1997, Biochemistry and molecular biology international.

[12]  C. Packard,et al.  Lipoprotein Subfraction Concentrations in Preeclampsia: Pathogenic Parallels to Atherosclerosis , 1997, Obstetrics and gynecology.

[13]  H. Ishimoto,et al.  Role of oxygen-derived free radicals in free growth retardation induced by ischemia-reperfusion in rats. , 1997, The American journal of physiology.

[14]  R. Taylor Review: Immunobiology of Preeclampsia , 1997, American journal of reproductive immunology.

[15]  J. Roberts,et al.  Distinct factors in plasma of preeclamptic women increase endothelial nitric oxide or prostacyclin. , 1996, Hypertension.

[16]  M. Stampfer,et al.  Dietary consumption and plasma concentrations of vitamin E in pregnancies complicated by preeclampsia. , 1996, American journal of obstetrics and gynecology.

[17]  E. Gratacós,et al.  Variation in lipid levels during pregnancy in women with different types of hypertension , 1996, Acta obstetricia et gynecologica Scandinavica.

[18]  S. Walsh,et al.  Antioxidant Activities and mRNA Expression of Superoxide Dismutase, Catalase, and Glutathione Peroxidase in Normal and Preeclamptic Placentas , 1996, The Journal of the Society for Gynecologic Investigation: JSGI.

[19]  J. Roberts,et al.  NOS expression is increased in endothelial cells exposed to plasma from women with preeclampsia. , 1995, The American journal of physiology.

[20]  R. Krauss,et al.  Dense low density lipoproteins and coronary artery disease. , 1995, The American journal of cardiology.

[21]  S. Hunt,et al.  Evidence for single gene contributions to hypertension and lipid disturbances: definition, genetics, and clinical significance , 1994, Clinical genetics.

[22]  S. Walsh,et al.  Deficient glutathione peroxidase activity in preeclampsia is associated with increased placental production of thromboxane and lipid peroxides. , 1993, American journal of obstetrics and gynecology.

[23]  S. Davidge,et al.  Pregnancy and lipid peroxide-induced alterations of eicosanoid-metabolizing enzymes in the aorta of the rat. , 1993, American journal of obstetrics and gynecology.

[24]  J. Witztum Susceptibility of low-density lipoprotein to oxidative modification. , 1993, The American journal of medicine.

[25]  R. Krauss,et al.  Susceptibility of small, dense, low-density lipoproteins to oxidative modification in subjects with the atherogenic lipoprotein phenotype, pattern B. , 1993, The American journal of medicine.

[26]  R. Tuimala,et al.  Findings on lipid peroxidation and antioxidant function in hypertensive complications of pregnancy , 1993, British journal of obstetrics and gynaecology.

[27]  H. Kay,et al.  Placental lipid peroxides and thromboxane are increased and prostacyclin is decreased in women with preeclampsia. , 1992, American journal of obstetrics and gynecology.

[28]  A. Goldfien,et al.  Sera From Preeclamptic Women Specifically Activate Human Umbilical Vein Endothelial Cells In Vitro: Morphological and Biochemical Evidence , 1992, American journal of reproductive immunology.

[29]  U. Das,et al.  Free radical generation, lipid peroxidation and essential fatty acids in uncontrolled essential hypertension. , 1990, Prostaglandins, leukotrienes, and essential fatty acids.

[30]  M. King,et al.  Atherogenic lipoprotein phenotype. A proposed genetic marker for coronary heart disease risk. , 1990, Circulation.

[31]  I. Greer,et al.  Neutrophil activation in pregnancy‐induced hypertension , 1989 .

[32]  T. Khong,et al.  Inadequate maternal vascular response to placentation in pregnancies complicated by pre‐eclampsia and by small‐for‐gestational age infants , 1986 .

[33]  S. Moncada,et al.  Superoxide anion is involved in the breakdown of endothelium-derived vascular relaxing factor , 1986, Nature.

[34]  Y. Sasaguri,et al.  Difference in the inhibitory effect of linoleic acid hydroperoxide on prostacyclin biosynthesis between cultured endothelial cells from human umbilical cord vein and cultured smooth muscle cells from rabbit aorta. , 1985, Biochemistry international.

[35]  W. Lands,et al.  Presence of lipid hydroperoxide in human plasma. , 1985, The Journal of clinical investigation.

[36]  J. Crapo,et al.  Biology of disease: free radicals and tissue injury. , 1982, Laboratory investigation; a journal of technical methods and pathology.

[37]  P. Nestel,et al.  The hyperlipidemia of pregnancy in normal and complicated pregnancies. , 1979, American journal of obstetrics and gynecology.

[38]  I. Brosens Morphological changes in the utero-placental bed in pregnancy hypertension. , 1977, Clinics in obstetrics and gynaecology.

[39]  F. de Wolf,et al.  The ultrastructure of acute atherosis in hypertensive pregnancy. , 1975, American journal of obstetrics and gynecology.

[40]  P. Kuo Current metabolic-genetic interrelationship in human atherosclerosis, with therapeutic considerations. , 1968, Annals of internal medicine.

[41]  E. Gratacós,et al.  Serum and placental lipid peroxides in chronic hypertension during pregnancy with and without superimposed preeclampsia. , 1999, Hypertension in pregnancy.

[42]  J. Roberts,et al.  Plasma of preeclamptic women stimulates and then inhibits endothelial prostacyclin. , 1996, Hypertension.

[43]  T. Henriksen,et al.  FASTING SERUM FREE FATTY ACIDS AND TRIGLYCERIDES ARE INCREASED BEFORE 20 WEEKS OF GESTATION IN WOMEN WHO LATER DEVELOP PREECLAMPSIA , 1994 .

[44]  S. Davidge,et al.  Lipid peroxidation increases arterial cyclooxygenase activity during pregnancy , 1994 .

[45]  B. Halliwell,et al.  Role of free radicals and catalytic metal ions in human disease: an overview. , 1990, Methods in enzymology.