Emerging role of semaphorin-3A in autoimmune diseases
暂无分享,去创建一个
D. Ye | Xiao-mei Li | H. Pan | Li-Na Liu | Hai-Feng Pan
[1] A. La Cava,et al. Editorial: Innate Immunity Pathways in Autoimmune Diseases , 2019, Front. Immunol..
[2] P. Tak,et al. Class 3 semaphorins modulate the invasive capacity of rheumatoid arthritis fibroblast-like synoviocytes , 2018, Rheumatology.
[3] Z. Vadasz,et al. Semaphorin 3A: Is a key player in the pathogenesis of asthma. , 2017, Clinical immunology.
[4] M. Etemadifar,et al. Decreased expression of Sema3A, an immune modulator, in blood sample of multiple sclerosis patients. , 2017, Gene.
[5] X. Zhang,et al. [Expression and Clinical Significance of Semaphorin 3A in serum and mononuclear cells in patients with systemic lupus erythematosus]. , 2017, Zhonghua yi xue za zhi.
[6] Shiyang Guan,et al. Interleukin-35: a Potential Therapeutic Agent for Autoimmune Diseases , 2016, Inflammation.
[7] D. Ye,et al. Emerging role of adipokines in systemic lupus erythematosus , 2016, Immunologic research.
[8] D. Sikorska,et al. SAT0223 Soluble Semaphorin 3A and Neuropilin-1: New Markers for Dysregulation of Angiogenesis in Systemic Sclerosis? , 2016 .
[9] A. Drosos,et al. SAT0222 B Cell Depletion Therapy in Systemic Sclerosis Associated Interstitial Lung Disease. A Multicenter, Open Label, Comparative Study with A Follow up of 94 Patient-Years , 2016 .
[10] E. Toubi,et al. A2.01 The expansion of CD25highIL-10highFOXP3high B regulatory cells is in association with sle disease activity , 2016 .
[11] P. Tak,et al. A7.09 Class 3 semaphorins modulate the invasive capacity of rheumatoid arthritis fibroblast-like synoviocytes , 2016 .
[12] X. Montalban,et al. Differential expression of sema3A and sema7A in a murine model of multiple sclerosis: Implications for a therapeutic design. , 2016, Clinical immunology.
[13] R. Soares,et al. Decreased expression of neuropilin-1 as a novel key factor contributing to peripheral microvasculopathy and defective angiogenesis in systemic sclerosis , 2015, Annals of the rheumatic diseases.
[14] H. Hartung,et al. Targeting semaphorins in MS as a treatment strategy to promote remyelination: A tale of mice, rats and men , 2015, Multiple sclerosis.
[15] G. Slobodin,et al. The Expansion of CD25highIL-10highFoxP3high B Regulatory Cells Is in Association with SLE Disease Activity , 2015, Journal of immunology research.
[16] S. Cajal,et al. Expression of semaphorin 3A, semaphorin 7A and their receptors in multiple sclerosis lesions , 2015, Multiple sclerosis.
[17] D. Rimar,et al. Semaphorin 3A, a potential immune regulator in Familial Mediterranean Fever , 2015, Pediatric Rheumatology.
[18] S. Kim. Immunological function of Blimp-1 in dendritic cells and relevance to autoimmune diseases , 2015, Immunologic Research.
[19] Manuel A. Friese,et al. Immunopathology of multiple sclerosis , 2015, Nature Reviews Immunology.
[20] T. Rainis,et al. The Involvement of Immune Semaphorins in the Pathogenesis of Inflammatory Bowel Diseases (IBDs) , 2015, PloS one.
[21] D. Rimar,et al. Semaphorin 3A: an immunoregulator in systemic sclerosis , 2015, Rheumatology International.
[22] D. Rimar,et al. A6.14 Semaphorin 3A, an immunoregulator and potential biomarker for disease severity in systemic sclerosis , 2015 .
[23] I. F. D. Larrinoa. Lo mejor del año en lupus eritematoso sistémico , 2015 .
[24] I. F. Larrinoa,et al. What is new in systemic lupus erythematosus. , 2015, Reumatologia clinica.
[25] Í. Rúa-Figueroa Fernández de Larrinoa. What is new in systemic lupus erythematosus. , 2015, Reumatologia clinica.
[26] X. Corbella,et al. Mortality and survival in systemic sclerosis: systematic review and meta-analysis. , 2014, Seminars in arthritis and rheumatism.
[27] D. Rimar,et al. AB0195 Semaphorin 3A as A Possible Immunoregulator in Systemic Sclerosis , 2014 .
[28] M. Matucci Cerinic,et al. AB0194 Ll-37: A New Marker for Interstitial Lung Disease (ILD) in Systemic Sclerosis (SSC)? , 2014 .
[29] I. Rosner,et al. A regulatory role for CD72 expression on B cells in systemic lupus erythematosus. , 2014, Seminars in arthritis and rheumatism.
[30] T. Malek,et al. The importance of regulatory T‐cell heterogeneity in maintaining self‐tolerance , 2014, Immunological reviews.
[31] Cong Yu,et al. Diagnostic criteria for systemic lupus erythematosus: a critical review. , 2014, Journal of autoimmunity.
[32] Z. Vadasz,et al. Semaphorins: Their Dual Role in Regulating Immune-Mediated Diseases , 2014, Clinical Reviews in Allergy & Immunology.
[33] S. Zheng,et al. Targeting T-helper 9 cells and interleukin-9 in autoimmune diseases. , 2013, Cytokine & growth factor reviews.
[34] T. Hirano,et al. AB0114 Semaphorin3a and semaphorin4d in rheumatoid arthritis. , 2013 .
[35] Xavier Montalban,et al. Semaphorins 3A and 7A: potential immune and neuroregenerative targets in multiple sclerosis. , 2013, Trends in molecular medicine.
[36] H. Datta,et al. Changes in bone density and bone turnover in patients with rheumatoid arthritis treated with rituximab, a B cell depleting monoclonal antibody (HORUS TRIAL) , 2013, BMC Musculoskeletal Disorders.
[37] S. Zheng,et al. Emerging role of interleukin-22 in autoimmune diseases. , 2013, Cytokine & growth factor reviews.
[38] Y. Goshima,et al. Decreased Semaphorin3A expression correlates with disease activity and histological features of rheumatoid arthritis , 2013, BMC Musculoskeletal Disorders.
[39] H. Takayanagi,et al. Bone cell communication factors and Semaphorins. , 2012, BoneKEy reports.
[40] E. Toubi,et al. Semaphorin 3A – a marker for disease activity and a potential putative disease-modifying treatment in systemic lupus erythematosus , 2012, Lupus.
[41] G. Neufeld,et al. Semaphorin 3A is a marker for disease activity and a potential immunoregulator in systemic lupus erythematosus , 2012, Arthritis Research & Therapy.
[42] R. Ransohoff,et al. Innate immunity in the central nervous system. , 2012, The Journal of clinical investigation.
[43] J. Gran,et al. Differences between rheumatologists and other internists regarding diagnosis and treatment of systemic lupus erythematosus. , 2012, Rheumatology.
[44] Y. Nakatsuji,et al. The role of immune semaphorins in multiple sclerosis , 2011, FEBS letters.
[45] E. Sabo,et al. The involvement of immune semaphorins and neuropilin-1 in lupus nephritis , 2011, Lupus.
[46] J. Carrillo-Ruiz,et al. Multiple sclerosis: An overview of the disease and current concepts of its pathophysiology , 2011 .
[47] P. Ravassard,et al. Class 3 semaphorins influence oligodendrocyte precursor recruitment and remyelination in adult central nervous system. , 2011, Brain : a journal of neurology.
[48] K. Nave,et al. Inhibition of CNS Remyelination by the Presence of Semaphorin 3A , 2011, The Journal of Neuroscience.
[49] M. Petri,et al. Efficacy and safety of belimumab in patients with active systemic lupus erythematosus: a randomised, placebo-controlled, phase 3 trial , 2011, The Lancet.
[50] B. Solomon,et al. Neuropilin-1 attenuates autoreactivity in experimental autoimmune encephalomyelitis , 2011, Proceedings of the National Academy of Sciences.
[51] S. Schneider‐Schaulies,et al. Measles virus modulates dendritic cell/T‐cell communication at the level of plexinA1/neuropilin‐1 recruitment and activity , 2011, European journal of immunology.
[52] D. Hafler,et al. Human regulatory T cells in autoimmune diseases. , 2010, Current opinion in immunology.
[53] A. Catalano. The Neuroimmune Semaphorin-3A Reduces Inflammation and Progression of Experimental Autoimmune Arthritis , 2010, The Journal of Immunology.
[54] D. Attias,et al. Neuropilins and semaphorins - from angiogenesis to autoimmunity. , 2010, Autoimmunity reviews.
[55] V. Barresi,et al. Increased ratio of vascular endothelial growth factor to semaphorin3A is a negative prognostic factor in human meningiomas , 2010, Neuropathology : official journal of the Japanese Society of Neuropathology.
[56] M. Tessier-Lavigne,et al. Semaphorins guide the entry of dendritic cells into the lymphatics by activating myosin II , 2010, Nature Immunology.
[57] M. Nishioka,et al. Clinical Significance of Serum Levels of Vascular Endothelial Growth Factor, Angiopoietin-1, and Angiopoietin-2 in Patients with Rheumatoid Arthritis , 2010, The Journal of Rheumatology.
[58] T. Randall,et al. Effector and regulatory B cells: modulators of CD4+ T cell immunity , 2010, Nature Reviews Immunology.
[59] A. Kumanogoh,et al. Regulation of immune cell responses by semaphorins and their receptors , 2010, Cellular and Molecular Immunology.
[60] M. Khamashta,et al. Clinical efficacy and side effects of antimalarials in systemic lupus erythematosus: a systematic review , 2008, Annals of the rheumatic diseases.
[61] R. Blasczyk,et al. 27-OR: Soluble semaphorin 3A regulates the immune response , 2009 .
[62] S. Kaveri,et al. Surveillance of antigen-presenting cells by CD4+ CD25+ regulatory T cells in autoimmunity: immunopathogenesis and therapeutic implications. , 2009, The American journal of pathology.
[63] L. Ivashkiv,et al. Expression and function of semaphorin 3A and its receptors in human monocyte-derived macrophages. , 2009, Human immunology.
[64] S. Šatkauskas,et al. The many faces of semaphorins: from development to pathology , 2009, Cellular and Molecular Life Sciences.
[65] Iain B McInnes,et al. Evidence that cytokines play a role in rheumatoid arthritis. , 2008, The Journal of clinical investigation.
[66] A. Chédotal,et al. Plexin-A4 negatively regulates T lymphocyte responses. , 2008, International immunology.
[67] R. Testa,et al. Semaphorin3A signaling controls Fas (CD95)-mediated apoptosis by promoting Fas translocation into lipid rafts. , 2008, Blood.
[68] Atsushi Kumanogoh,et al. Semaphorins and their receptors in immune cell interactions , 2008, Nature Immunology.
[69] H. Kikutani,et al. Immune Semaphorins: Novel Features of Neural Guidance Molecules , 2008, Journal of Clinical Immunology.
[70] Bernard Zalc,et al. Semaphorin 3A and 3F: key players in myelin repair in multiple sclerosis? , 2007, Brain : a journal of neurology.
[71] J. Ménard,et al. Angiogenesis markers (VEGF, soluble receptor of VEGF and angiopoietin-1) in very early arthritis and their association with inflammation and joint destruction. , 2007, Clinical immunology.
[72] T. Medsger,et al. Changes in causes of death in systemic sclerosis, 1972–2002 , 2007, Annals of the rheumatic diseases.
[73] I. Konishi,et al. Expression of semaphorins, vascular endothelial growth factor, and their common receptor neuropilins and alleic loss of semaphorin locus in epithelial ovarian neoplasms: increased ratio of vascular endothelial growth factor to semaphorin is a poor prognostic factor in ovarian carcinomas. , 2006, Human pathology.
[74] S. Chandran,et al. A Novel Role for Sema3A in Neuroprotection from Injury Mediated by Activated Microglia , 2006, The Journal of Neuroscience.
[75] A. Barzilai,et al. Immunosuppressive role of semaphorin‐3A on T cell proliferation is mediated by inhibition of actin cytoskeleton reorganization , 2006, European journal of immunology.
[76] M. Faronato,et al. Semaphorin-3A is expressed by tumor cells and alters T-cell signal transduction and function. , 2006, Blood.
[77] Kazuhiro Suzuki,et al. Semaphorins in the Immune System , 2005 .
[78] Y. Kanakura,et al. Negative regulation of platelet function by a secreted cell repulsive protein, semaphorin 3A. , 2004, Blood.
[79] Till Acker,et al. Uncontrolled Expression of Vascular Endothelial Growth Factor and Its Receptors Leads to Insufficient Skin Angiogenesis in Patients With Systemic Sclerosis , 2004, Circulation research.
[80] D. Pleasure,et al. Induction of neuropilins-1 and -2 and their ligands, Sema3A, Sema3F, and VEGF, during Wallerian degeneration in the peripheral nervous system , 2003, Experimental Neurology.
[81] C. Lau,et al. Pathogenesis of systemic lupus erythematosus , 2003, Journal of clinical pathology.
[82] M. Weller,et al. Human malignant glioma cells express semaphorins and their receptors, neuropilins and plexins , 2003, Glia.
[83] A. Püschel,et al. The function of semaphorins during nervous system development. , 2003, Frontiers in bioscience : a journal and virtual library.
[84] G. Villegas,et al. Ontogeny of semaphorins 3A and 3F and their receptors neuropilins 1 and 2 in the kidney. , 2002, Mechanisms of Development.
[85] Y. Konttinen,et al. Increased but imbalanced expression of VEGF and its receptors has no positive effect on angiogenesis in systemic sclerosis skin. , 2002, Clinical and experimental rheumatology.
[86] M. Conforti,et al. Angiogenic and angiostatic factors in systemic sclerosis: increased levels of vascular endothelial growth factor are a feature of the earliest disease stages and are associated with the absence of fingertip ulcers , 2002, Arthritis research.
[87] J. Verhaagen,et al. Injury-Induced Class 3 Semaphorin Expression in the Rat Spinal Cord , 2002, Experimental Neurology.
[88] M. Gassmann,et al. HIF-1 mediated upregulation of VEGF and VEGF-R in systemic sclerosis (SSc): Imbalance with angiostatic factors suggests VEGF as a novel option for the treatment of ischemia in patients with SSc , 2002, Arthritis Research.
[89] T. Yagi,et al. Requirement of neuropilin 1-mediated Sema3A signals in patterning of the sympathetic nervous system. , 2002, Development.
[90] R. Rudick,et al. Premyelinating oligodendrocytes in chronic lesions of multiple sclerosis. , 2002, The New England journal of medicine.
[91] J. Goverman,et al. A Pathogenic Role for Myelin-Specific Cd8+ T Cells in a Model for Multiple Sclerosis , 2001, The Journal of experimental medicine.
[92] A. Elhabazi,et al. Biological Activity of Soluble CD100. II. Soluble CD100, Similarly to H-SemaIII, Inhibits Immune Cell Migration1 2 , 2001, The Journal of Immunology.
[93] M. Taniguchi,et al. Expression of class 3 semaphorins and neuropilin receptors in the developing mouse tooth , 2001, Mechanisms of Development.
[94] V. Gagliardini,et al. Semaphorin III Can Induce Death in Sensory Neurons , 1999, Molecular and Cellular Neuroscience.
[95] R. Kalb,et al. Plexin-Neuropilin-1 Complexes Form Functional Semaphorin-3A Receptors , 1999, Cell.
[96] Jacqueline Palace,et al. The effect of anti-α4 integrin antibody on brain lesion activity in MS , 1999, Neurology.
[97] S. Soker,et al. Neuropilin-1 Mediates Collapsin-1/Semaphorin III Inhibition of Endothelial Cell Motility , 1999, The Journal of cell biology.
[98] M. Poo,et al. Unified Nomenclature for the Semaphorins/Collapsins , 1999, Cell.
[99] A. Koch,et al. Angiogenesis: Implications for rheumatoid arthritis , 1998 .
[100] T. Yagi,et al. Disruption of Semaphorin III/D Gene Causes Severe Abnormality in Peripheral Nerve Projection , 1997, Neuron.
[101] A. Silman,et al. Survival following the onset of scleroderma: results from a retrospective inception cohort study of the UK patient population. , 1996, British journal of rheumatology.
[102] W. Snider,et al. The guidance molecule Semaphorin III is expressed in regions of spinal cord and periphery avoided by growing sensory axons , 1995, The Journal of comparative neurology.
[103] R. Adams,et al. Murine semaphorin D/collapsin is a member of a diverse gene family and creates domains inhibitory for axonal extension , 1995, Neuron.
[104] C. Shatz,et al. Sernaphorin III can function as a selective chemorepellent to pattern sensory projections in the spinal cord , 1995, Neuron.
[105] C. Goodman,et al. The semaphorin genes encode a family of transmembrane and secreted growth cone guidance molecules , 1993, Cell.
[106] D. Raible,et al. Collapsin: A protein in brain that induces the collapse and paralysis of neuronal growth cones , 1993, Cell.
[107] T. Medsger,et al. Scleroderma (systemic sclerosis): classification, subsets and pathogenesis. , 1988, The Journal of rheumatology.
[108] F. Quismorio,et al. T Lymphocyte Subsets in Systemic Lupus Erythematosus , 1983 .