Vitamin D regulating TGF-β induced epithelial-mesenchymal transition
暂无分享,去创建一个
[1] A. Goldbart,et al. The effect of vitamin D on airway reactivity and inflammation in asthmatic children: A double‐blind placebo‐controlled trial , 2015, Pediatric pulmonology.
[2] D. Agrawal,et al. Vitamin D and inflammatory diseases , 2014, Journal of inflammation research.
[3] Tonya S. King,et al. Effect of vitamin D3 on asthma treatment failures in adults with symptomatic asthma and lower vitamin D levels: the VIDA randomized clinical trial. , 2014, JAMA.
[4] A. Berraies,et al. Link between vitamin D and airway remodeling , 2014, Journal of asthma and allergy.
[5] D. Agrawal,et al. Importins and Exportins Regulating Allergic Immune Responses , 2014, Mediators of inflammation.
[6] O. Eickelberg,et al. Tissue remodelling in chronic bronchial diseases: from the epithelial to mesenchymal phenotype , 2014, European Respiratory Review.
[7] Prue H. Hart,et al. Vitamin D deficiency causes airway hyperresponsiveness, increases airway smooth muscle mass, and reduces TGF‐β expression in the lungs of female BALB/c mice , 2014, Physiological reports.
[8] Toshiyuki Shimizu,et al. A nonclassical vitamin D receptor pathway suppresses renal fibrosis. , 2013, The Journal of clinical investigation.
[9] Q. Hamid,et al. IL-22 contributes to TGF-β1-mediated epithelial-mesenchymal transition in asthmatic bronchial epithelial cells , 2013, Respiratory Research.
[10] X. Xiang,et al. IL4 and IL-17A provide a Th2/Th17-polarized inflammatory milieu in favor of TGF-β1 to induce bronchial epithelial-mesenchymal transition (EMT). , 2013, International journal of clinical and experimental pathology.
[11] Barry Dellinger,et al. Radical-containing ultrafine particulate matter initiates epithelial-to-mesenchymal transitions in airway epithelial cells. , 2013, American journal of respiratory cell and molecular biology.
[12] D. Agrawal,et al. Vitamin D supplementation reduces airway hyperresponsiveness and allergic airway inflammation in a murine model , 2013, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[13] R. Gallo,et al. Vitamin D in allergic disease: shedding light on a complex problem. , 2013, The Journal of allergy and clinical immunology.
[14] B. Tzang,et al. IL‐6 augmented motility of airway epithelial cell BEAS‐2B via Akt/GSK‐3β signaling pathway , 2012, Journal of cellular biochemistry.
[15] D. Agrawal,et al. Vitamin D deficiency decreases the expression of VDR and prohibitin in the lungs of mice with allergic airway inflammation. , 2012, Experimental and molecular pathology.
[16] B. Coulomb,et al. The myofibroblast, multiple origins for major roles in normal and pathological tissue repair , 2012, Fibrogenesis & tissue repair.
[17] L. Akinbami,et al. Trends in asthma prevalence, health care use, and mortality in the United States, 2001-2010. , 2012, NCHS data brief.
[18] D. Agrawal,et al. Calcitriol Decreases Expression of Importin α3 and Attenuates RelA Translocation in Human Bronchial Smooth Muscle Cells , 2012, Journal of Clinical Immunology.
[19] C. Nguyên,et al. Interactions Between β-Catenin and Transforming Growth Factor-β Signaling Pathways Mediate Epithelial-Mesenchymal Transition and Are Dependent on the Transcriptional Co-activator cAMP-response Element-binding Protein (CREB)-binding Protein (CBP)* , 2012, The Journal of Biological Chemistry.
[20] Q. Hamid,et al. Vitamin D Deficiency as a Strong Predictor of Asthma in Children , 2011, International Archives of Allergy and Immunology.
[21] M. Haussler,et al. Vitamin D receptor (VDR)-mediated actions of 1α,25(OH)₂vitamin D₃: genomic and non-genomic mechanisms. , 2011, Best practice & research. Clinical endocrinology & metabolism.
[22] Danielle T. Loughlin,et al. Modification of Collagen by 3-Deoxyglucosone Alters Wound Healing through Differential Regulation of p38 MAP Kinase , 2011, PloS one.
[23] K. Smejda,et al. Vitamin D supplementation in children may prevent asthma exacerbation triggered by acute respiratory infection. , 2011, The Journal of allergy and clinical immunology.
[24] Seung-Hyo Lee,et al. Cigarette smoke exacerbates mouse allergic asthma through Smad proteins expressed in mast cells , 2011, Respiratory research.
[25] G. Krstić. Asthma Prevalence Associated with Geographical Latitude and Regional Insolation in the United States of America and Australia , 2011, PloS one.
[26] M. Morrill. The effects of maternal employment on the health of school-age children. , 2011, Journal of health economics.
[27] Q. Hamid,et al. Role of transforming growth factor-β in airway remodeling in asthma. , 2011, American journal of respiratory cell and molecular biology.
[28] J. Fuxe,et al. Chronic Respiratory Aeroallergen Exposure in Mice Induces Epithelial-Mesenchymal Transition in the Large Airways , 2011, PLoS ONE.
[29] D. Agrawal,et al. Fms-like tyrosine kinase 3 ligand decreases T helper type 17 cells and suppressors of cytokine signaling proteins in the lung of house dust mite-sensitized and -challenged mice. , 2010, American journal of respiratory cell and molecular biology.
[30] Christopher M Waters,et al. Epithelial repair mechanisms in the lung. , 2010, American journal of physiology. Lung cellular and molecular physiology.
[31] S. Teach,et al. High prevalence of vitamin D deficiency among inner-city African American youth with asthma in Washington, DC. , 2010, The Journal of pediatrics.
[32] A. K. Reinhardt,et al. TGF-β Isoform Specific Regulation of Airway Inflammation and Remodelling in a Murine Model of Asthma , 2010, PloS one.
[33] J. Roman,et al. Vitamin D inhibition of pro-fibrotic effects of transforming growth factor β1 in lung fibroblasts and epithelial cells , 2010, The Journal of Steroid Biochemistry and Molecular Biology.
[34] J. Carethers,et al. TGF-beta downregulates PTEN via activation of NF-kappaB in pancreatic cancer cells. , 2010, American journal of physiology. Gastrointestinal and liver physiology.
[35] P. O'Byrne,et al. Effects of budesonide and formoterol on allergen-induced airway responses, inflammation, and airway remodeling in asthma. , 2010, The Journal of allergy and clinical immunology.
[36] G. Jarai,et al. Epithelial-mesenchymal transition in primary human bronchial epithelial cells is Smad-dependent and enhanced by fibronectin and TNF-α , 2010, Fibrogenesis & tissue repair.
[37] Jun Sun,et al. Vitamin D receptor deletion leads to reduced level of I κ B α protein through protein translation, protein-protein interaction, and post-translational modification , 2010 .
[38] D. Postma,et al. House dust mite-promoted epithelial-to-mesenchymal transition in human bronchial epithelium. , 2010, American journal of respiratory cell and molecular biology.
[39] D. Davies. The role of the epithelium in airway remodeling in asthma. , 2009, Proceedings of the American Thoracic Society.
[40] B. Zuraw,et al. TGF-β1 induced epithelial to mesenchymal transition (EMT) in human bronchial epithelial cells is enhanced by IL-1β but not abrogated by corticosteroids , 2009, Respiratory research.
[41] R. Crystal,et al. A SNAIL1–SMAD3/4 transcriptional repressor complex promotes TGF-β mediated epithelial–mesenchymal transition , 2009, Nature Cell Biology.
[42] D. Knight,et al. Induction of epithelial-mesenchymal transition in primary airway epithelial cells from patients with asthma by transforming growth factor-beta1. , 2009, American journal of respiratory and critical care medicine.
[43] Min Zhang,et al. TGF-β1 Induces Human Bronchial Epithelial Cell-to-Mesenchymal Transition in Vitro , 2009, Lung.
[44] G. Hunninghake,et al. Respiratory Epithelial Cells Convert Inactive Vitamin D to Its Active Form: Potential Effects on Host Defense1 , 2008, The Journal of Immunology.
[45] R. Panettieri,et al. Vitamin D and glucocorticoids differentially modulate chemokine expression in human airway smooth muscle cells , 2008, British journal of pharmacology.
[46] A. Litonjua,et al. Is vitamin D deficiency to blame for the asthma epidemic? , 2007, The Journal of allergy and clinical immunology.
[47] R. Murphy,et al. The regulatory role of TGF‐β in airway remodeling in asthma , 2007 .
[48] Giulio Gabbiani,et al. The myofibroblast: one function, multiple origins. , 2007, The American journal of pathology.
[49] M. Hew,et al. Expression and activation of TGF-β isoforms in acute allergen-induced remodelling in asthma , 2007, Thorax.
[50] S. Holgate,et al. The contribution of transforming growth factor-β and epidermal growth factor signalling to airway remodelling in chronic asthma , 2006, European Respiratory Journal.
[51] S. Wenzel,et al. Increased TGF-β2 in severe asthma with eosinophilia , 2005 .
[52] S. Wenzel,et al. Increased TGF-beta2 in severe asthma with eosinophilia. , 2005, The Journal of allergy and clinical immunology.
[53] A. Musani,et al. Bronchoalveolar lavage fluid concentrations of transforming growth factor (TGF)‐β1, TGF‐β2, interleukin (IL)‐4 and IL‐13 after segmental allergen challenge and their effects on α‐smooth muscle actin and collagen III synthesis by primary human lung fibroblasts , 2004, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[54] P. Howarth,et al. Mechanisms of airway epithelial damage: epithelial-mesenchymal interactions in the pathogenesis of asthma , 2003, European Respiratory Journal.
[55] J Bousquet,et al. Asthma. From bronchoconstriction to airways inflammation and remodeling. , 2000, American journal of respiratory and critical care medicine.
[56] R. Martin,et al. Eosinophil-associated TGF-beta1 mRNA expression and airways fibrosis in bronchial asthma. , 1997, American journal of respiratory cell and molecular biology.
[57] P. Howarth,et al. Interleukin-4, -5, and -6 and tumor necrosis factor-alpha in normal and asthmatic airways: evidence for the human mast cell as a source of these cytokines. , 1994, American journal of respiratory cell and molecular biology.