Genome-wide association meta-analysis of coronary artery disease and periodontitis reveals a novel shared risk locus
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A. Uitterlinden | J. Erdmann | W. Lieb | J. Wellmann | C. Graetz | A. Teumer | A. Franke | K. Berger | M. Laudes | B. Holtfreter | T. Kocher | L. D. de Groot | P. Hoffmann | N. van der Velde | K. Divaris | B. Loos | Loreto A. Muñoz | A. Bhandari | S. Offenbacher | Stephanie Tennstedt | M. Munz | H. Dommisch | A. Schaefer | S. Jepsen | G. Richter | Y. Jockel-Schneider | C. Bruckmann | I. Staufenbiel | B. Krone | A. Uitterlinden | Bastian Krone
[1] Andrew D. Johnson,et al. Fifteen new risk loci for coronary artery disease highlight arterial-wall-specific mechanisms , 2017, Nature Genetics.
[2] A. Descoteaux,et al. Exploitation of the Host Cell Membrane Fusion Machinery by Leishmania Is Part of the Infection Process , 2016, PLoS pathogens.
[3] Manolis Kellis,et al. HaploReg v4: systematic mining of putative causal variants, cell types, regulators and target genes for human complex traits and disease , 2015, Nucleic Acids Res..
[4] Gabor T. Marth,et al. A global reference for human genetic variation , 2015, Nature.
[5] Sebastian M. Armasu,et al. A comprehensive 1000 Genomes-based genome-wide association meta-analysis of coronary artery disease , 2015, Nature Genetics.
[6] L. Johannes,et al. Shiga toxin stimulates clathrin-independent endocytosis of the VAMP2, VAMP3 and VAMP8 SNARE proteins , 2015, Journal of Cell Science.
[7] Wolf-Tilo Balke,et al. Multidimensional gene search with Genehopper , 2015, Nucleic Acids Res..
[8] G. Kempermann. Faculty Opinions recommendation of Human genomics. The Genotype-Tissue Expression (GTEx) pilot analysis: multitissue gene regulation in humans. , 2015 .
[9] Jun S. Liu,et al. The Genotype-Tissue Expression (GTEx) pilot analysis: Multitissue gene regulation in humans , 2015, Science.
[10] J. Hampe,et al. IL-6 blockade by monoclonal antibodies inhibits apolipoprotein (a) expression and lipoprotein (a) synthesis in humans[S] , 2015, Journal of Lipid Research.
[11] P. Eke,et al. Update on Prevalence of Periodontitis in Adults in the United States: NHANES 2009 to 2012. , 2015, Journal of periodontology.
[12] Peter Kraft,et al. Using genetics to test the causal relationship of total adiposity and periodontitis: Mendelian randomization analyses in the Gene-Lifestyle Interactions and Dental Endpoints (GLIDE) Consortium. , 2015, International journal of epidemiology.
[13] C. Gieger,et al. Genetic Evidence for PLASMINOGEN as a Shared Genetic Risk Factor of Coronary Artery Disease and Periodontitis , 2015, Circulation. Cardiovascular genetics.
[14] Wenjie Chen,et al. GRASP v2.0: an update on the Genome-Wide Repository of Associations between SNPs and phenotypes , 2014, Nucleic Acids Res..
[15] K. North,et al. Chronic Periodontitis Genome-wide Association Studies , 2014, Journal of dental research.
[16] C. Susin,et al. Epidemiology and demographics of aggressive periodontitis. , 2014, Periodontology 2000.
[17] Michael R. Yeaman,et al. Platelets: at the nexus of antimicrobial defence , 2014, Nature Reviews Microbiology.
[18] J. Shendure,et al. A general framework for estimating the relative pathogenicity of human genetic variants , 2014, Nature Genetics.
[19] Peggy Hall,et al. The NHGRI GWAS Catalog, a curated resource of SNP-trait associations , 2013, Nucleic Acids Res..
[20] H. Völzke,et al. Genome-wide association study of chronic periodontitis in a general German population. , 2013, Journal of clinical periodontology.
[21] M. Peters,et al. Systematic identification of trans eQTLs as putative drivers of known disease associations , 2013, Nature Genetics.
[22] Pedro G. Ferreira,et al. Transcriptome and genome sequencing uncovers functional variation in humans , 2013, Nature.
[23] Philip Rosenstiel,et al. The large non-coding RNA ANRIL, which is associated with atherosclerosis, periodontitis and several forms of cancer, regulates ADIPOR1, VAMP3 and C11ORF10. , 2013, Human molecular genetics.
[24] C. Wijmenga,et al. Validation of reported genetic risk factors for periodontitis in a large-scale replication study. , 2013, Journal of clinical periodontology.
[25] A. Olshan,et al. Exploring the genetic basis of chronic periodontitis: a genome-wide association study , 2013, Human molecular genetics.
[26] J. Danesh,et al. Large-scale association analysis identifies new risk loci for coronary artery disease , 2012, Nature Genetics.
[27] A. Olshan,et al. Genome-wide Association Study of Periodontal Pathogen Colonization , 2012, Journal of dental research.
[28] K. Berger. [DHS: The Dortmund health study]. , 2012, Bundesgesundheitsblatt, Gesundheitsforschung, Gesundheitsschutz.
[29] Panos N Papapanou,et al. Periodontal disease and atherosclerotic vascular disease: does the evidence support an independent association?: a scientific statement from the American Heart Association. , 2012, Circulation.
[30] Jesse R. Dixon,et al. Topological Domains in Mammalian Genomes Identified by Analysis of Chromatin Interactions , 2012, Nature.
[31] Greg Gibson,et al. Rare and common variants: twenty arguments , 2012, Nature Reviews Genetics.
[32] Fred A. Wright,et al. seeQTL: a searchable database for human eQTLs , 2011, Bioinform..
[33] A. Uitterlinden,et al. Rationale and design of the B-PROOF study, a randomized controlled trial on the effect of supplemental intake of vitamin B12 and folic acid on fracture incidence , 2011, BMC geriatrics.
[34] Manolis Kellis,et al. HaploReg: a resource for exploring chromatin states, conservation, and regulatory motif alterations within sets of genetically linked variants , 2011, Nucleic Acids Res..
[35] W. Rathmann,et al. Cohort profile: the study of health in Pomerania. , 2011, International journal of epidemiology.
[36] Mark I. McCarthy,et al. A genome-wide association study in Europeans and South Asians identifies five new loci for coronary artery disease , 2011, Nature Genetics.
[37] Dmitri V Zaykin,et al. P‐value based analysis for shared controls design in genome‐wide association studies , 2010, Genetic epidemiology.
[38] M. Nothnagel,et al. CDKN2BAS is associated with periodontitis in different European populations and is activated by bacterial infection , 2010, Journal of Medical Genetics.
[39] L. Smeeth,et al. Invasive Dental Treatment and Risk for Vascular Events , 2010, Annals of Internal Medicine.
[40] Alexander Teumer,et al. Replication of the association of chromosomal region 9p21.3 with generalized aggressive periodontitis (gAgP) using an independent case-control cohort , 2010, BMC Medical Genetics.
[41] Jordan Wesolowski,et al. SNARE motif: A common motif used by pathogens to manipulate membrane fusion , 2010, Virulence.
[42] Laurent Gil,et al. Ensembl variation resources , 2010, BMC Genomics.
[43] Wei Zhang,et al. SCAN: SNP and copy number annotation , 2010, Bioinform..
[44] G. Romito,et al. Quantitative detection of periodontopathic bacteria in atherosclerotic plaques from coronary arteries. , 2009, Journal of medical microbiology.
[45] D. James,et al. Variations in the requirement for v-SNAREs in GLUT4 trafficking in adipocytes , 2009, Journal of Cell Science.
[46] Bruno G. Loos,et al. UvA-DARE ( Digital Academic Repository ) Identification of a shared genetic susceptibility locus for coronary heart disease and periodontitis , 2009 .
[47] M. Nilges,et al. SNARE Protein Mimicry by an Intracellular Bacterium , 2008, PLoS pathogens.
[48] Kari Stefansson,et al. A common variant on chromosome 9p21 affects the risk of myocardial infarction. , 2007, Science.
[49] C. Gieger,et al. Genomewide association analysis of coronary artery disease. , 2007, The New England journal of medicine.
[50] A. Gylfason,et al. A Common Variant on Chromosome 9p21 Affects the Risk of Myocardial Infarction , 2007, Science.
[51] Simon C. Potter,et al. Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls , 2007, Nature.
[52] Jonathan C. Cohen,et al. A Common Allele on Chromosome 9 Associated with Coronary Heart Disease , 2007, Science.
[53] Francesco D'Aiuto,et al. Treatment of periodontitis and endothelial function. , 2007, The New England journal of medicine.
[54] Hongmin Sun,et al. The interaction between pathogens and the host coagulation system. , 2006, Physiology.
[55] Michael Krawczak,et al. PopGen: Population-Based Recruitment of Patients and Controls for the Analysis of Complex Genotype-Phenotype Relationships , 2006, Public Health Genomics.
[56] G. Abecasis,et al. Joint analysis is more efficient than replication-based analysis for two-stage genome-wide association studies , 2006, Nature Genetics.
[57] G. Hansson. Inflammation, atherosclerosis, and coronary artery disease. , 2005, The New England journal of medicine.
[58] U. John,et al. Study of Health in Pomerania (SHIP): a health survey in an East German region. Objectives and design of the oral health section. , 2003, Quintessence international.
[59] G. Reed,et al. Vesicle-associated membrane protein 3 (VAMP-3) and VAMP-8 are present in human platelets and are required for granule secretion. , 2002, Blood.
[60] D. Grönemeyer,et al. Assessment of clinically silent atherosclerotic disease and established and novel risk factors for predicting myocardial infarction and cardiac death in healthy middle-aged subjects: rationale and design of the Heinz Nixdorf RECALL Study. Risk Factors, Evaluation of Coronary Calcium and Lifestyle. , 2002, American heart journal.
[61] T. Galli,et al. Soluble NSF Attachment Protein Receptors (SNAREs) in RBL-2H3 Mast Cells: Functional Role of Syntaxin 4 in Exocytosis and Identification of a Vesicle-Associated Membrane Protein 8-Containing Secretory Compartment1 , 2000, The Journal of Immunology.
[62] M. Leinonen,et al. Infections, inflammation, and the risk of coronary heart disease. , 2000, Circulation.
[63] Aric Invest. The Atherosclerosis Risk in Communities (ARIC) Study: design and objectives. The ARIC investigators , 1989 .
[64] A. Folsom,et al. The Atherosclerosis Risk in Communities (ARIC) Study: design and objectives. The ARIC investigators. , 1989, American journal of epidemiology.
[65] D. Tousoulis,et al. Atherosclerosis and Coronary Artery Disease , 2016 .
[66] Sidney C. Smith,et al. Periodontal Disease and Atherosclerotic Vascular Disease: Does the Evidence Support an Independent Association? A , 2012 .
[67] J. Kaur,et al. Periodontal Disease and Coronary Heart Disease , 2009 .
[68] U. John,et al. Study of Health in Pomerania (SHIP): A health examination survey in an east German region: Objectives and design , 2005, Sozial- und Präventivmedizin.
[69] V. Sambri,et al. Molecular detection of Treponema denticola and Porphyromonas gingivalis in carotid and aortic atheromatous plaques by FISH: report of two cases. , 2005, Journal of medical microbiology.
[70] D. Couper,et al. Periodontal Disease and Coronary Heart Disease: A Reappraisal of the Exposure , 2005, Circulation.
[71] A genome-wide association study identifies nucleotide variants at SIGLEC5 and DEFA1A3 as risk loci for periodontitis , 2022 .