Delivery of GABAARs to Synapses Is Mediated by HAP1-KIF5 and Disrupted by Mutant Huntingtin
暂无分享,去创建一个
Zhen Yan | P. Rostaing | A. Triller | S. Humbert | F. Saudou | A. MacAskill | Alison E. Twelvetrees | I. Arancibia-Cárcamo | J. Kittler | E. Yuen | M. Lumb | Michael J. Lumb | I. Arancibia-Carcamo
[1] I. Arancibia-Cárcamo,et al. Regulation of GABA(A) receptor membrane trafficking and synaptic localization. , 2009, Pharmacology & therapeutics.
[2] D. Attwell,et al. Miro1 Is a Calcium Sensor for Glutamate Receptor-Dependent Localization of Mitochondria at Synapses , 2009, Neuron.
[3] Paul Chen,et al. Activation of 5-HT2A/C Receptors Counteracts 5-HT1A Regulation of N-Methyl-D-aspartate Receptor Channels in Pyramidal Neurons of Prefrontal Cortex , 2008, Journal of Biological Chemistry.
[4] S. Moss,et al. GABAA receptor trafficking and its role in the dynamic modulation of neuronal inhibition , 2008, Nature Reviews Neuroscience.
[5] P. Verstreken,et al. Suppression of Neurodegeneration and Increased Neurotransmission Caused by Expanded Full-Length Huntingtin Accumulating in the Cytoplasm , 2008, Neuron.
[6] G. Kreitzer,et al. Polarization-dependent selective transport to the apical membrane by KIF5B in MDCK cells. , 2007, Developmental cell.
[7] R. Khazipov,et al. GABA: a pioneer transmitter that excites immature neurons and generates primitive oscillations. , 2007, Physiological reviews.
[8] L. Raymond,et al. Altered NMDA Receptor Trafficking in a Yeast Artificial Chromosome Transgenic Mouse Model of Huntington's Disease , 2007, The Journal of Neuroscience.
[9] L. Raymond,et al. N-Methyl-d-aspartate (NMDA) receptor function and excitotoxicity in Huntington's disease , 2007, Progress in Neurobiology.
[10] Elina Ikonen,et al. When intracellular logistics fails - genetic defects in membrane trafficking , 2006, Journal of Cell Science.
[11] R. Sidman,et al. Toll-like receptor 8 functions as a negative regulator of neurite outgrowth and inducer of neuronal apoptosis , 2006, The Journal of cell biology.
[12] S. Moss,et al. The Dynamic Synapse : Molecular Methods in Ionotropic Receptor Biology , 2006 .
[13] Xiao-Jiang Li,et al. Interaction of Huntingtin-associated Protein-1 with Kinesin Light Chain , 2006, Journal of Biological Chemistry.
[14] Zhen Yan,et al. Phospho-dependent binding of the clathrin AP2 adaptor complex to GABAA receptors regulates the efficacy of inhibitory synaptic transmission. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[15] D. Naylor,et al. Trafficking of GABAA Receptors, Loss of Inhibition, and a Mechanism for Pharmacoresistance in Status Epilepticus , 2005, The Journal of Neuroscience.
[16] P. Brundin,et al. Synaptic dysfunction in Huntington’s disease: a new perspective , 2005, Cellular and Molecular Life Sciences CMLS.
[17] Paul Chen,et al. Serotonin 5-HT1A Receptors Regulate NMDA Receptor Channels through a Microtubule-Dependent Mechanism , 2005, The Journal of Neuroscience.
[18] I. Módy,et al. Pathological Cell-Cell Interactions Elicited by a Neuropathogenic Form of Mutant Huntingtin Contribute to Cortical Pathogenesis in HD Mice , 2005, Neuron.
[19] Nobutaka Hirokawa,et al. Molecular motors and mechanisms of directional transport in neurons , 2005, Nature Reviews Neuroscience.
[20] Yu Wang,et al. Insulin exerts neuroprotection by counteracting the decrease in cell‐surface GABAA receptors following oxygen–glucose deprivation in cultured cortical neurons , 2005, Journal of neurochemistry.
[21] S. Moss,et al. Huntingtin-associated protein 1 regulates inhibitory synaptic transmission by modulating gamma-aminobutyric acid type A receptor membrane trafficking. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[22] Fabrice P Cordelières,et al. Huntingtin Controls Neurotrophic Support and Survival of Neurons by Enhancing BDNF Vesicular Transport along Microtubules , 2004, Cell.
[23] Scott T. Brady,et al. Neuropathogenic Forms of Huntingtin and Androgen Receptor Inhibit Fast Axonal Transport , 2003, Neuron.
[24] Richard G. Brusch,et al. Disruption of Axonal Transport by Loss of Huntingtin or Expression of Pathogenic PolyQ Proteins in Drosophila , 2003, Neuron.
[25] N. Hirokawa,et al. KIF17 Dynamics and Regulation of NR2B Trafficking in Hippocampal Neurons , 2003, The Journal of Neuroscience.
[26] ジャック アール. ワンズ. Suppression of neurodegeneration , 2002 .
[27] L. Raymond,et al. Increased Sensitivity to N-Methyl-D-Aspartate Receptor-Mediated Excitotoxicity in a Mouse Model of Huntington's Disease , 2002, Neuron.
[28] C. Ibáñez,et al. Coordinated Activation of Autophosphorylation Sites in the RET Receptor Tyrosine Kinase , 2002, The Journal of Biological Chemistry.
[29] M. Morris,et al. A peptide carrier for the delivery of biologically active proteins into mammalian cells , 2001, Nature Biotechnology.
[30] N. Hirokawa,et al. All kinesin superfamily protein, KIF, genes in mouse and human , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[31] M. MacDonald,et al. Dominant phenotypes produced by the HD mutation in STHdh(Q111) striatal cells. , 2000, Human molecular genetics.
[32] M. Ehlers,et al. Reinsertion or Degradation of AMPA Receptors Determined by Activity-Dependent Endocytic Sorting , 2000, Neuron.
[33] S. Moss,et al. Constitutive Endocytosis of GABAA Receptors by an Association with the Adaptin AP2 Complex Modulates Inhibitory Synaptic Currents in Hippocampal Neurons , 2000, The Journal of Neuroscience.
[34] S. Moss,et al. Analysis of GABAA Receptor Assembly in Mammalian Cell Lines and Hippocampal Neurons Using γ2 Subunit Green Fluorescent Protein Chimeras , 2000, Molecular and Cellular Neuroscience.
[35] N. Hirokawa,et al. Kinesin superfamily motor protein KIF17 and mLin-10 in NMDA receptor-containing vesicle transport. , 2000, Science.
[36] V. Allan,et al. Microtubule-based endoplasmic reticulum motility in Xenopus laevis: activation of membrane-associated kinesin during development. , 1999, Molecular biology of the cell.
[37] A. Triller,et al. Dendritic and Postsynaptic Protein Synthetic Machinery , 1999, The Journal of Neuroscience.
[38] P. Rostaing,et al. Localization of components of glycinergic synapses during rat spinal cord development , 1998, The Journal of comparative neurology.
[39] S. Hersch,et al. Interaction of Huntingtin-Associated Protein with Dynactin P150Glued , 1998, The Journal of Neuroscience.
[40] P. Worley,et al. Huntingtin-associated protein 1 (HAP1) interacts with the p150Glued subunit of dynactin. , 1997, Human molecular genetics.
[41] Guo-Qiang Bi,et al. Kinesin- and Myosin-driven Steps of Vesicle Recruitment for Ca2+-regulated Exocytosis , 1997, The Journal of cell biology.
[42] S. Snyder,et al. A huntingtin-associated protein enriched in brain with implications for pathology , 1995, Nature.
[43] J. Scholey,et al. Inhibition of kinesin-driven microtubule motility by monoclonal antibodies to kinesin heavy chains , 1988, The Journal of cell biology.
[44] Joseph B. Martin. Huntington's disease , 1984, Neurology.
[45] Zhen Yan,et al. Dopamine D3 Receptors Regulate GABAA Receptor Function through a Phospho-Dependent Endocytosis Mechanism in Nucleus Accumbens , 2006, The Journal of Neuroscience.
[46] J. Isaac,et al. Transfecting and Transducing Neurons with Synthetic Nucleic Acids and Biologically Active Macromolecules , 2006 .
[47] Moss Sj,et al. Transfecting and Transducing Neurons with Synthetic Nucleic Acids and Biologically Active Macromolecules -- The Dynamic Synapse: Molecular Methods in Ionotropic Receptor Biology , 2006 .
[48] Shihua Li,et al. HAP1 and intracellular trafficking. , 2005, Trends in pharmacological sciences.