Ionic dependence of glutamate neurotoxicity
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[1] A. Kriegstein,et al. Glutamate neurotoxicity in cortical cell culture , 1987, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[2] T. Wieloch. Hypoglycemia-induced neuronal damage prevented by an N-methyl-D-aspartate antagonist. , 1985, Science.
[3] D. Choi,et al. Glutamate neurotoxicity in cortical cell culture is calcium dependent , 1985, Neuroscience Letters.
[4] S. Rothman. The neurotoxicity of excitatory amino acids is produced by passive chloride influx , 1985, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[5] J. Olney,et al. Calcium influx accompanies but does not cause excitotoxin-induced neuronal necrosis in retina , 1985, Brain Research Bulletin.
[6] B. Siesjö,et al. Cerebral metabolism in ischaemia: neurochemical basis for therapy. , 1985, British journal of anaesthesia.
[7] B. Meldrum,et al. Blockade of N-methyl-D-aspartate receptors may protect against ischemic damage in the brain. , 1984, Science.
[8] B. Meldrum,et al. Calcium Overload in Selectively Vulnerable Neurons of the Hippocampus during and after Ischemia: An Electron Microscopy Study in the Rat , 1984, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[9] L. Nowak,et al. Magnesium gates glutamate-activated channels in mouse central neurones , 1984, Nature.
[10] Coyle Jt. Neurotoxic action of kainic acid. , 1983 .
[11] F. Orrego,et al. Kainate, N-methylaspartate and other excitatory amino acids increase calcium influx into rat brain cortex cells in vitro , 1983, Neuroscience Letters.
[12] P. Usherwood,et al. Investigations into the mechanism of excitant amino acid cytotoxicity using a well-characterized glutamatergic system , 1983, Brain Research.
[13] F. Plum. What causes infarction in ischemic brain? , 1983, Neurology.
[14] J. Coyle. Neurotoxic action of kainic acid. , 1983, Journal of neurochemistry.
[15] J. Hablitz,et al. Excitation of hippocampal pyramidal cells by glutamate in the guinea‐pig and rat. , 1982, The Journal of physiology.
[16] J. Watkins,et al. Differential activation and blockade of excitatory amino acid receptors in the mammalian and amphibian central nervous systems. , 1982, Comparative biochemistry and physiology. C: Comparative pharmacology.
[17] R. Nicoll,et al. Synaptic excitation may activate a calcium-dependent potassium conductance in hippocampal pyramidal cells. , 1981, Science.
[18] J. Watkins,et al. 2-Amino-5-phosphonovalerate (2APV), a potent and selective antagonist of amino acid-induced and synaptic excitation , 1981, Neuroscience Letters.
[19] J. Farber,et al. Calcium dependence of toxic cell death: a final common pathway. , 1979, Science.
[20] M. Salpeter,et al. Agonist-induced myopathy at the neuromuscular junction is mediated by calcium , 1979, The Journal of cell biology.
[21] J. Olney,et al. Kainic acid: a powerful neurotoxic analogue of glutamate. , 1974, Brain research.
[22] R. Nicoll,et al. The pharmacology and ionic dependency of amino acid responses in the frog spinal cord , 1973, The Journal of physiology.
[23] D. Lucas,et al. The toxic effect of sodium L-glutamate on the inner layers of the retina. , 1957, A.M.A. archives of ophthalmology.