Histological diversity and molecular characteristics in gastric cancer: relation of cancer stem cell-related molecules and receptor tyrosine kinase molecules to mixed histological type and more histological patterns
暂无分享,去创建一个
[1] W. Lu,et al. Epithelial-Mesenchymal Plasticity in Cancer Progression and Metastasis. , 2019, Developmental cell.
[2] Katsuharu Saito,et al. Prognostic role of ARID1A negative expression in gastric cancer , 2019, Scientific Reports.
[3] Dakeun Lee,et al. Functional loss of ARID1A is tightly associated with high PD‐L1 expression in gastric cancer , 2019, International journal of cancer.
[4] T. Hirasawa,et al. Undifferentiated‐type component mixed with differentiated‐type early gastric cancer is a significant risk factor for endoscopic non‐curative resection , 2018, Digestive endoscopy : official journal of the Japan Gastroenterological Endoscopy Society.
[5] Marco Mina,et al. Pan-cancer inference of intra-tumor heterogeneity reveals associations with different forms of genomic instability , 2018, PLoS genetics.
[6] D. Tripathy,et al. Evaluation of cMET aberration by immunohistochemistry and fluorescence in situ hybridization (FISH) in triple negative breast cancers. , 2018, Annals of diagnostic pathology.
[7] W. Xu,et al. Tumor heterogeneity of gastric cancer: From the perspective of tumor-initiating cell , 2018, World journal of gastroenterology.
[8] Kyungeun Kim,et al. Acquisition of histologic diversity contributes to not only invasiveness but also lymph node metastasis in early gastric cancer. , 2017, Pathology, research and practice.
[9] Carla Oliveira,et al. Heterogeneity in Gastric Cancer: From Pure Morphology to Molecular Classifications , 2017, Pathobiology.
[10] I. Lebedev,et al. Clinically relevant morphological structures in breast cancer represent transcriptionally distinct tumor cell populations with varied degrees of epithelial-mesenchymal transition and CD44+CD24- stemness , 2017, Oncotarget.
[11] I. Lebedev,et al. Clinically relevant morphological structures in breast cancer represent transcriptionally distinct tumor cell populations with varied degrees of epithelial-mesenchymal transition and CD44+CD24- stemness , 2017, Oncotarget.
[12] M. Alsina,et al. Intratumoral heterogeneity in gastric cancer: a new challenge to face. , 2017, Annals of oncology : official journal of the European Society for Medical Oncology.
[13] W. Yasui,et al. Clinicopathological significance of SPC18 in colorectal cancer: SPC18 participates in tumor progression , 2017, Cancer science.
[14] Yihong Ma,et al. Reacquisition of E-cadherin expression in metastatic deposits of signet-ring cell carcinoma of the upper gastrointestinal system: a potential anchor for metastatic deposition , 2016, Journal of Clinical Pathology.
[15] Hans Clevers,et al. Tissue-specific mutation accumulation in human adult stem cells during life , 2016, Nature.
[16] S. Messing,et al. Immunohistochemistry as a surrogate for molecular subtyping of gastric adenocarcinoma. , 2016, Human pathology.
[17] Y. Hirokawa,et al. Significant intratumoral heterogeneity of human epidermal growth factor receptor 2 status in gastric cancer: A comparative study of immunohistochemistry, FISH, and dual‐color in situ hybridization , 2016, Cancer science.
[18] Bin Zhang,et al. Molecular classification and prediction in gastric cancer , 2015, Computational and structural biotechnology journal.
[19] R. Nusse,et al. Helicobacter pylori Activates and Expands Lgr5(+) Stem Cells Through Direct Colonization of the Gastric Glands. , 2015, Gastroenterology.
[20] H. Grabsch,et al. Five biopsy specimens from the proximal part of the tumor reliably determine HER2 protein expression status in gastric cancer , 2015, Gastric Cancer.
[21] Nayoung Kim,et al. Helicobacter pylori-induced epithelial-mesenchymal transition, a potential role of gastric cancer initiation and an emergence of stem cells. , 2015, Carcinogenesis.
[22] Steven J. M. Jones,et al. Comprehensive molecular characterization of gastric adenocarcinoma , 2014, Nature.
[23] N. Tomita,et al. New molecular staging with G-factor supplements TNM classification in gastric cancer: a multicenter collaborative research by the Japan Society for Gastroenterological Carcinogenesis G-Project committee , 2014, Gastric Cancer.
[24] T. Nakajima,et al. Risk of lymph node metastases from intramucosal gastric cancer in relation to histological types: how to manage the mixed histological type for endoscopic submucosal dissection , 2013, Gastric Cancer.
[25] A. Yoshizawa,et al. HER2 expression and its clinicopathological features in resectable gastric cancer , 2013, Gastric Cancer.
[26] Joo Young Cho,et al. Optical biopsies by confocal endomicroscopy prevent additive endoscopic biopsies before endoscopic submucosal dissection in gastric epithelial neoplasias: a prospective, comparative study. , 2011, Gastrointestinal endoscopy.
[27] N. Cho,et al. Mixed-type gastric cancer and its association with high-frequency CpG island hypermethylation , 2010, Virchows Archiv.
[28] Pu Xia,et al. The pathobiological behaviors and prognosis associated with Japanese gastric adenocarcinomas of pure WHO histological subtypes. , 2010, Histology and histopathology.
[29] Hans Clevers,et al. Lgr5(+ve) stem cells drive self-renewal in the stomach and build long-lived gastric units in vitro. , 2010, Cell stem cell.
[30] F. Schmitt,et al. Association of ERBB2 gene status with histopathological parameters and disease-specific survival in gastric carcinoma patients , 2009, British Journal of Cancer.
[31] J. Gordon,et al. Helicobacter pylori evolution during progression from chronic atrophic gastritis to gastric cancer and its impact on gastric stem cells , 2008, Proceedings of the National Academy of Sciences.
[32] Hua-Chuan Zheng,et al. Mixed-type gastric carcinomas exhibit more aggressive features and indicate the histogenesis of carcinomas , 2008, Virchows Archiv.
[33] K. Takubo,et al. Clinicopathological and molecular characteristics of gastric and colorectal carcinomas in the elderly , 2007, Pathology international.
[34] H. Grabsch,et al. Mixed Gastric Carcinomas Show Similar Chromosomal Aberrations in Both their Diffuse and Glandular Components , 2006, Cellular oncology : the official journal of the International Society for Cellular Oncology.
[35] S. Kitano,et al. Pathology and prognosis of gastric carcinoma , 2000, Cancer.
[36] K. Hirokawa,et al. Pathological Characteristics of Gastric Carcinomas in the Very Old , 1998, Japanese journal of cancer research : Gann.
[37] K. Yamashita,et al. Assessing the Suitability of Gastric Carcinoma for Limited Resection: Histologic Differentiation of Endoscopic Biopsy , 1998, World Journal of Surgery.
[38] G. Ranzani,et al. Genetic pattern, histological structure, and cellular phenotype in early and advanced gastric cancers: evidence for structure-related genetic subsets and for loss of glandular structure during progression of some tumors. , 1998, Human pathology.
[39] S. Bianchi,et al. Reproducibility of histologic classification of gastric cancer. , 1991, British Journal of Cancer.
[40] Yutaka Naito,et al. Clinicopathological significance of MMP-7, laminin γ2 and EGFR expression at the invasive front of gastric carcinoma , 2013, Gastric Cancer.
[41] Y. Ohkura. Histological Diversity of Early Gastric Carcinoma , 2005 .
[42] G. Tamura. Promoter methylation status of tumor suppressor and tumor-related genes in neoplastic and non-neoplastic gastric epithelia. , 2004, Histology and histopathology.