Functional impairment in isolated rat hearts induced by activated leukocytes: protective effect of oxygen free radical scavengers.

[1]  J. Repine,et al.  Granulocytes mediate acute edematous lung injury in rabbits and in isolated rabbit lungs perfused with phorbol myristate acetate: role of oxygen radicals. , 2015, The American review of respiratory disease.

[2]  R. Engler Consequences of activation and adenosine-mediated inhibition of granulocytes during myocardial ischemia. , 1987, Federation proceedings.

[3]  L. Williamson,et al.  Neutrophils are involved in the increased vascular permeability produced by activated complement in man , 1986, British journal of haematology.

[4]  D. Das,et al.  Enhanced prostaglandin synthesis due to phospholipid breakdown in ischemic-reperfused myocardium. Control of its production by a phospholipase inhibitor or free radical scavengers. , 1986, Journal of molecular and cellular cardiology.

[5]  R. Myklebust,et al.  Influence of oxygen radicals generated by xanthine oxidase in the isolated perfused rat heart. , 1986, Cardiovascular research.

[6]  G. Schmid-Schönbein,et al.  Role of leukocytes in response to acute myocardial ischemia and reflow in dogs. , 1986, The American journal of physiology.

[7]  L M Cruz-Orive,et al.  Estimation of surface area from vertical sections , 1986, Journal of microscopy.

[8]  C. Haslett,et al.  Neutrophil-mediated injury to endothelial cells. Enhancement by endotoxin and essential role of neutrophil elastase. , 1986, The Journal of clinical investigation.

[9]  M. Bernier,et al.  Reperfusion‐induced Arrhythmias and Oxygen‐derived Free Radicals: Studies with “Anti‐Free Radical” Interventions and a Free Radical‐generating System in the Isolated Perfused Rat Heart , 1986, Circulation research.

[10]  B. Pitt,et al.  Reduction of the size of infarction by allopurinol in the ischemic-reperfused canine heart. , 1986, Circulation.

[11]  R. Kloner,et al.  Superoxide Dismutase Plus Catalase Improve Contractile Function in the Canine Model of the “Stunned Myocardium” , 1986, Circulation research.

[12]  S. Weiss,et al.  Involvement of hydrogen peroxide and hydroxyl radical in the 'oxygen paradox': reduction of creatine kinase release by catalase, allopurinol or deferoxamine, but not by superoxide dismutase. , 1985, Journal of molecular and cellular cardiology.

[13]  B. Woodward,et al.  Effect of some free radical scavengers on reperfusion induced arrhythmias in the isolated rat heart. , 1985, Journal of molecular and cellular cardiology.

[14]  K. Burton Superoxide dismutase enhances recovery following myocardial ischemia. , 1985, The American journal of physiology.

[15]  E. Haber,et al.  The Role of Free Radical‐Mediated Processes in Oxygen‐Related Damage in Cultured Murine Myocardial Cells , 1985, Circulation research.

[16]  G. Ghai,et al.  Myocardial alterations due to free-radical generation. , 1984, The American journal of physiology.

[17]  M. Duvelleroy,et al.  Role of oxygen radicals in cardiac injury due to reoxygenation. , 1984, Journal of molecular and cellular cardiology.

[18]  J. Salmon,et al.  Role of leukocytes in acute myocardial infarction in anesthetized dogs: relationship to myocardial salvage by anti-inflammatory drugs. , 1984, The Journal of pharmacology and experimental therapeutics.

[19]  J. Repine,et al.  Hydroxyl Radical Scavengers Produce Similar Decreases in the Chemiluminescence Responses and Bactericidal Activities of Neutrophils , 1984, Infection and immunity.

[20]  M. Hess,et al.  Hydrogen peroxide and hydroxyl radical mediation of activated leukocyte depression of cardiac sarcoplasmic reticulum. Participation of the cyclooxygenase pathway. , 1983, Circulation research.

[21]  L. Greenfield,et al.  Inhibition of surgically induced ischemia/reperfusion injury by oxygen free radical scavengers. , 1983, The Journal of thoracic and cardiovascular surgery.

[22]  M. Schork,et al.  Reduction of the Extent of Ischemic Myocardial Injury by Neutrophil Depletion in the Dog , 1983, Circulation.

[23]  Weiss Sj,et al.  Phagocyte-generated oxygen metabolites and cellular injury , 1982 .

[24]  K. Arfors,et al.  Increase in microvascular permeability induced by enzymatically generated free radicals. I. In vivo study. , 1981, Microvascular research.

[25]  K. Arfors,et al.  Increase in microvascular permeability induced by enzymatically generated free radicals. II. Role of superoxide anion radical, hydrogen peroxide, and hydroxyl radical. , 1981, Microvascular research.

[26]  H. Jacob,et al.  Oxygen radicals mediate endothelial cell damage by complement-stimulated granulocytes. An in vitro model of immune vascular damage. , 1978, The Journal of clinical investigation.

[27]  L. Dechatelet,et al.  Effect of phorbol myristate acetate on the oxidative metabolism of human polymorphonuclear leukocytes , 1976 .

[28]  R. Allen,et al.  Phagocytic activation of a luminol-dependent chemiluminescence in rabbit alveolar and peritoneal macrophages. , 1976, Biochemical and biophysical research communications.

[29]  Lefer Am,et al.  Preservation of myocardial integrity by a protease inhibitor during acute myocardial ischemia. , 1974 .

[30]  Jennings Rb,et al.  EXPERIMENTAL ACUTE MYOCARDIAL INFARCTION; HISTOLOGIC AND HISTOCHEMICAL STUDIES OF EARLY MYOCARDIAL INFARCTS INDUCED BY TEMPORARY OR PERMANENT OCCLUSION OF A CORONARY ARTERY. , 1964 .

[31]  Seymour Geisser,et al.  Statistical Principles in Experimental Design , 1963 .

[32]  A. Semb,et al.  Oxygen free radicals decrease survival time of isolated rat hearts. , 1989, Scandinavian journal of thoracic and cardiovascular surgery.

[33]  R. Myklebust,et al.  Ultrastructural changes induced in the isolated rat heart by enzymatically generated oxygen radicals. , 1987, Journal of molecular and cellular cardiology.

[34]  J. Niedel,et al.  Phorbol diester receptor copurifies with protein kinase C. , 1983, Proceedings of the National Academy of Sciences of the United States of America.

[35]  P. Ward,et al.  Role of oxygen-derived free radicals and metabolites in leukocyte-dependent inflammatory reactions. , 1982, The American journal of pathology.