Antigen presentation to CD8+ T cells: cross-priming in infectious diseases.
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[1] K. Rock,et al. Cell injury releases endogenous adjuvants that stimulate cytotoxic T cell responses. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[2] M. Bevan,et al. Cd8+ but Not Cd8− Dendritic Cells Cross-Prime Cytotoxic T Cells in Vivo , 2000, The Journal of experimental medicine.
[3] A. Rickinson,et al. The Importance of Exogenous Antigen in Priming the Human CD8+ T Cell Response: Lessons from the EBV Nuclear Antigen EBNA11 , 2000, The Journal of Immunology.
[4] S. Akira,et al. A Toll-like receptor recognizes bacterial DNA , 2000, Nature.
[5] P. Srivastava,et al. Cutting Edge: Heat Shock Protein gp96 Induces Maturation and Migration of CD11c+ Cells In Vivo1 , 2000, The Journal of Immunology.
[6] P. Romero,et al. OmpA targets dendritic cells, induces their maturation and delivers antigen into the MHC class I presentation pathway , 2000, Nature Immunology.
[7] R. Steinman,et al. Canarypox Virus-Induced Maturation of Dendritic Cells Is Mediated by Apoptotic Cell Death and Tumor Necrosis Factor Alpha Secretion , 2000, Journal of Virology.
[8] Matthew L. Albert,et al. αvβ5 integrin recruits the CrkII–Dock180–Rac1 complex for phagocytosis of apoptotic cells , 2000, Nature Cell Biology.
[9] J. Abastado,et al. Incoming Human Cytomegalovirus pp65 (UL83) Contained in Apoptotic Infected Fibroblasts Is Cross-Presented to CD8+ T Cells by Dendritic Cells , 2000, Journal of Virology.
[10] P. Srivastava,et al. Necrotic but not apoptotic cell death releases heat shock proteins, which deliver a partial maturation signal to dendritic cells and activate the NF-kappa B pathway. , 2000, International immunology.
[11] M. Bevan,et al. Requirements for Bone Marrow–Derived Antigen-Presenting Cells in Priming Cytotoxic T Cell Responses to Intracellular Pathogens , 2000, The Journal of experimental medicine.
[12] K. Rock,et al. Bone Marrow–Derived Antigen-Presenting Cells Are Required for the Generation of Cytotoxic T Lymphocyte Responses to Viruses and Use Transporter Associated with Antigen Presentation (Tap)-Dependent and -Independent Pathways of Antigen Presentation , 2000, The Journal of experimental medicine.
[13] C. Janeway,et al. The Toll receptor family and microbial recognition. , 2000, Trends in microbiology.
[14] B. Cookson,et al. Salmonella induces macrophage death by caspase‐1‐dependent necrosis , 2000, Molecular microbiology.
[15] N. Restifo. Building better vaccines: how apoptotic cell death can induce inflammation and activate innate and adaptive immunity. , 2000, Current opinion in immunology.
[16] P. Srivastava,et al. Saturation, Competition, and Specificity in Interaction of Heat Shock Proteins (hsp) gp96, hsp90, and hsp70 with CD11b+ Cells1 , 2000, The Journal of Immunology.
[17] A. Alcamí,et al. Viral mechanisms of immune evasion , 2000, Immunology Today.
[18] A. Aderem,et al. Toll-like receptors in the induction of the innate immune response , 2000, Nature.
[19] P. Srivastava,et al. CD91: a receptor for heat shock protein gp96 , 2000, Nature Immunology.
[20] H. Rammensee,et al. The heat shock protein gp96 induces maturation of dendritic cells and down‐regulation of its receptor , 2000, European journal of immunology.
[21] Y. Kwaik,et al. The modulation of host cell apoptosis by intracellular bacterial pathogens. , 2000, Trends in microbiology.
[22] H. Rammensee,et al. Cross-Presentation of Glycoprotein 96–Associated Antigens on Major Histocompatibility Complex Class I Molecules Requires Receptor-Mediated Endocytosis , 2000, The Journal of experimental medicine.
[23] J. Rothman,et al. Receptor-Mediated Uptake of Antigen/Heat Shock Protein Complexes Results in Major Histocompatibility Complex Class I Antigen Presentation via Two Distinct Processing Pathways , 2000, The Journal of experimental medicine.
[24] S. Akira,et al. Caspase-1 activation of IL-1beta and IL-18 are essential for Shigella flexneri-induced inflammation. , 2000, Immunity.
[25] A. Fischer,et al. Consequences of Fas-Mediated Human Dendritic Cell Apoptosis Induced by Measles Virus , 2000, Journal of Virology.
[26] M. Svensson,et al. Salmonella infection of bone marrow-derived macrophages and dendritic cells: influence on antigen presentation and initiating an immune response. , 2000, FEMS immunology and medical microbiology.
[27] Stuart K. Calderwood,et al. HSP70 stimulates cytokine production through a CD14-dependant pathway, demonstrating its dual role as a chaperone and cytokine , 2000, Nature Medicine.
[28] R. Young,et al. A proposed mechanism for the induction of cytotoxic T lymphocyte production by heat shock fusion proteins. , 2000, Immunity.
[29] U. Yrlid,et al. Salmonella-Induced Apoptosis of Infected Macrophages Results in Presentation of a Bacteria-Encoded Antigen after Uptake by Bystander Dendritic Cells , 2000, The Journal of experimental medicine.
[30] Nina Bhardwaj,et al. Consequences of cell death: exposure to necrotic tumor cells , 2000 .
[31] A. Lanzavecchia,et al. Dendritic cell maturation is induced by mycoplasma infection but not by necrotic cells , 2000, European journal of immunology.
[32] H. Kolb,et al. Cutting Edge: Heat Shock Protein 60 Is a Putative Endogenous Ligand of the Toll-Like Receptor-4 Complex1 , 2000, The Journal of Immunology.
[33] Stefania Gallucci,et al. Natural adjuvants: Endogenous activators of dendritic cells , 1999, Nature Medicine.
[34] Sebastian Amigorena,et al. Selective transport of internalized antigens to the cytosol for MHC class I presentation in dendritic cells , 1999, Nature Cell Biology.
[35] J. Neefjes,et al. Recycling MHC class I molecules and endosomal peptide loading. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[36] P. Klenerman,et al. A comparison of T cell memory against the same antigen induced by virus versus intracellular bacteria. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[37] C. Rugarli,et al. Immunogenicity of apoptotic cells in vivo: role of antigen load, antigen-presenting cells, and cytokines. , 1999, Journal of immunology.
[38] H. Rammensee,et al. Cutting edge: receptor-mediated endocytosis of heat shock proteins by professional antigen-presenting cells. , 1999, Journal of immunology.
[39] R. Andino,et al. Cytotoxic T-cell immunity to virus-infected non-haematopoietic cells requires presentation of exogenous antigen , 1999, Nature.
[40] P. Srivastava,et al. Calreticulin, a Peptide-binding Chaperone of the Endoplasmic Reticulum, Elicits Tumor- and Peptide-specific Immunity , 1999, The Journal of experimental medicine.
[41] P. Ricciardi-Castagnoli,et al. Bystander apoptosis triggers dendritic cell maturation and antigen-presenting function. , 1998, Journal of immunology.
[42] David W. Marsh,et al. CD4+ T Cell Tolerance to Parenchymal Self-Antigens Requires Presentation by Bone Marrow–derived Antigen-presenting Cells , 1998, The Journal of experimental medicine.
[43] M. Albert,et al. Dendritic cells acquire antigen from apoptotic cells and induce class I-restricted CTLs , 1998, Nature.
[44] Jeff F. Miller,et al. Compartmentalization of Bacterial Antigens: Differential Effects on Priming of CD8 T Cells and Protective Immunity , 1998, Cell.
[45] L. Frappier,et al. Human CD8+ T cell responses to EBV EBNA1: HLA class I presentation of the (Gly-Ala)-containing protein requires exogenous processing. , 1997, Immunity.
[46] A. Poggi,et al. The selective engulfment of apoptotic bodies by dendritic cells is mediated by the αvβ3 integrin and requires intracellular and extracellular calcium , 1997 .
[47] C. Kurts,et al. Class I–restricted Cross-Presentation of Exogenous Self-Antigens Leads to Deletion of Autoreactive CD8+ T Cells , 1997, The Journal of experimental medicine.
[48] W. Heath,et al. Induction of a CD8+ Cytotoxic T Lymphocyte Response by Cross-priming Requires Cognate CD4+ T Cell Help , 1997, The Journal of experimental medicine.
[49] A. Iwasaki,et al. The dominant role of bone marrow-derived cells in CTL induction following plasmid DNA immunization at different sites. , 1997, Journal of immunology.
[50] D. Carson,et al. Gene vaccination with naked plasmid DNA: mechanism of CTL priming , 1996, The Journal of experimental medicine.
[51] J. Miller,et al. Constitutive class I-restricted exogenous presentation of self antigens in vivo , 1996, The Journal of experimental medicine.
[52] E. Jaffee,et al. Role of bone marrow-derived cells in presenting MHC class I-restricted tumor antigens. , 1994, Science.
[53] K P Lee,et al. Differential T cell costimulatory requirements in CD28-deficient mice. , 1993, Science.
[54] M. Bevan,et al. Class I-restricted processing and presentation of exogenous cell- associated antigen in vivo , 1990, The Journal of experimental medicine.
[55] M. Bevan,et al. Minor H antigens introduced on H-2 different stimulating cells cross-react at the cytotoxic T cell level during in vivo priming. , 1976, Journal of immunology.
[56] M. Bevan. Cross-priming for a secondary cytotoxic response to minor H antigens with H-2 congenic cells which do not cross-react in the cytotoxic assay , 1976, The Journal of experimental medicine.
[57] R. Steinman,et al. Brief Definitive Report Dendritic Cells Cross-present Latency Gene Products from Epstein-barr Virus–transformed B Cells and Expand Tumor-reactive Cd8 Ϩ Killer T Cells , 2022 .