Reduction of airway anion secretion via CFTR in sphingomyelin pathway.

[1]  M. Kondo,et al.  Apical and basolateral ATP-induced anion secretion in polarized human airway epithelia. , 2004, American journal of respiratory cell and molecular biology.

[2]  M. Kondo,et al.  Effects of Fluoranthene, a Polycyclic Aromatic Hydrocarbon, on cAMP-Dependent Anion Secretion in Human Airway Epithelia , 2004, Journal of Pharmacology and Experimental Therapeutics.

[3]  R. Boucher,et al.  An overview of the pathogenesis of cystic fibrosis lung disease. , 2002, Advanced drug delivery reviews.

[4]  M. Kondo,et al.  Bisphenol A inhibits Cl(-) secretion by inhibition of basolateral K+ conductance in human airway epithelial cells. , 2002, The Journal of pharmacology and experimental therapeutics.

[5]  P. Linsdell,et al.  Characterization of basolateral K+ channels underlying anion secretion in the human airway cell line Calu‐3 , 2002, The Journal of physiology.

[6]  Sheng-Nan Wu,et al.  Ceramide Inhibits the Inwardly Rectifying Potassium Current in GH3 Lactotrophs. , 2001, Endocrinology.

[7]  J. Kok,et al.  The Involvement of Sphingolipids in Multidrug Resistance , 2001, The Journal of Membrane Biology.

[8]  F. Lang,et al.  Physiology of apoptosis. , 2000, American journal of physiology. Renal physiology.

[9]  H. Kume,et al.  CFTR-Mediated anion conductance regulates Na(+)-K(+)-pump activity in Calu-3 human airway cells. , 2000, Biochemical and biophysical research communications.

[10]  J. Wang,et al.  Sphingomyelinase and ceramide analogs induce contraction and rises in [Ca(2+)](i) in canine cerebral vascular muscle. , 2000, American journal of physiology. Heart and circulatory physiology.

[11]  D. Devor,et al.  Kinase-dependent Regulation of the Intermediate Conductance, Calcium-dependent Potassium Channel, hIK1* , 2000, The Journal of Biological Chemistry.

[12]  L. Kleine,et al.  Advances in the signal transduction of ceramide and related sphingolipids. , 1999, Critical reviews in clinical laboratory sciences.

[13]  K. M. Nicholson,et al.  Preferential killing of multidrug-resistant KB cells by inhibitors of glucosylceramide synthase , 1999, British Journal of Cancer.

[14]  W. Campbell,et al.  Effect of ceramide on KCa channel activity and vascular tone in coronary arteries. , 1999, Hypertension.

[15]  Ashvani K. Singh,et al.  Bicarbonate and Chloride Secretion in Calu-3 Human Airway Epithelial Cells , 1999, The Journal of general physiology.

[16]  A. Gear,et al.  Sphingolipid metabolism during human platelet activation. , 1999, Thrombosis research.

[17]  R. Kolesnick,et al.  Stress signals for apoptosis: ceramide and c-Jun kinase , 1998, Oncogene.

[18]  B. Soliven,et al.  Ceramide Inhibits Inwardly Rectifying K+ Currents via a Ras- and Raf-1-Dependent Pathway in Cultured Oligodendrocytes , 1998, The Journal of Neuroscience.

[19]  C. Liedtke,et al.  Antisense oligonucleotide to PKC-ε alters cAMP-dependent stimulation of CFTR in Calu-3 cells. , 1998, American journal of physiology. Cell physiology.

[20]  J. Riordan,et al.  Differential regulation of single CFTR channels by PP2C, PP2A, and other phosphatases. , 1998, American journal of physiology. Cell physiology.

[21]  K. Williams,et al.  Human Vascular Endothelial Cells Are a Rich and Regulatable Source of Secretory Sphingomyelinase , 1998, The Journal of Biological Chemistry.

[22]  A. K. Singh,et al.  Tolbutamide causes open channel blockade of cystic fibrosis transmembrane conductance regulator Cl- channels. , 1996, Biophysical journal.

[23]  L. Tsui,et al.  Phosphatase inhibitors activate normal and defective CFTR chloride channels. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[24]  J. Wine,et al.  CFTR in Calu-3 human airway cells: channel properties and role in cAMP-activated Cl- conductance. , 1994, The American journal of physiology.

[25]  J. Wine,et al.  Calu-3: a human airway epithelial cell line that shows cAMP-dependent Cl- secretion. , 1994, The American journal of physiology.

[26]  P. Quinton Cystic fibrosis: a disease in electrolyte transport , 1990, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[27]  B D Schultz,et al.  Pharmacology of CFTR chloride channel activity. , 1999, Physiological reviews.