Multivariate analysis of factors associated with early‐onset segmental and nonsegmental vitiligo: a prospective observational study of 213 patients

Background  Although mixed forms have been described recently, segmental (SV) and nonsegmental vitiligo (NSV) are considered as clinically distinct. However, limited epidemiological data are available to help distinguish associated factors, and recent genome‐wide association studies have been restricted to NSV. The higher prevalence of SV in children is helpful when comparing the two major presentations of the disease.

[1]  T. Jouary,et al.  Segmental vitiligo associated with generalized vitiligo (mixed vitiligo): a retrospective case series of 19 patients. , 2011, Journal of the American Academy of Dermatology.

[2]  R. Spritz,et al.  Comprehensive association analysis of candidate genes for generalized vitiligo supports XBP1, FOXP3, and TSLP. , 2011, The Journal of investigative dermatology.

[3]  C. Bodemer,et al.  Segmental and nonsegmental childhood vitiligo has distinct clinical characteristics: a prospective observational study. , 2010, Journal of the American Academy of Dermatology.

[4]  R. Clark,et al.  First histopathological and immunophenotypic analysis of early dynamic events in a patient with segmental vitiligo associated with halo nevi , 2010, Pigment cell & melanoma research.

[5]  G. Girolomoni,et al.  The Role of Innate Immunity in Vitiligo , 2010 .

[6]  M. Picardo,et al.  Epidemiology, Definitions and Classification , 2010 .

[7]  Meenu R Pillai,et al.  The development and function of regulatory T cells , 2009, Cellular and Molecular Life Sciences.

[8]  M. Picardo,et al.  Clinical practice. Vitiligo. , 2009, The New England journal of medicine.

[9]  T. Jouary,et al.  Segmental vitiligo as the possible expression of cutaneous somatic mosaicism: implications for common non‐segmental vitiligo , 2008, Pigment cell & melanoma research.

[10]  I. L. Le Poole,et al.  HSP70i accelerates depigmentation in a mouse model of autoimmune vitiligo. , 2008, The Journal of investigative dermatology.

[11]  J. Silverberg,et al.  Thyroid abnormalities in pediatric patients with vitiligo in New York City. , 2008, Cutis.

[12]  M. Picardo,et al.  The definition and assessment of vitiligo: a consensus report of the Vitiligo European Task Force. , 2007, Pigment cell research.

[13]  J. Pincemail,et al.  [Oxidative stress]. , 2007, Revue medicale de Liege.

[14]  U. Pajvani,et al.  The relationship between family medical history and childhood vitiligo. , 2006, Journal of the American Academy of Dermatology.

[15]  B. Klapp,et al.  Towards a “free radical theory of graying”: melanocyte apoptosis in the aging human hair follicle is an indicator of oxidative stress induced tissue damage , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[16]  G. Chrousos,et al.  Increased prevalence of chronic autoimmune (Hashimoto's) thyroiditis in children and adolescents with vitiligo. , 2005, Journal of the American Academy of Dermatology.

[17]  R. Spritz,et al.  Epidemiology of vitiligo and associated autoimmune diseases in Caucasian probands and their families. , 2003, Pigment cell research.

[18]  S. Dogra,et al.  Epidemiology of Childhood Vitiligo: A Study of 625 Patients from North India , 2003, Pediatric dermatology.

[19]  S. Prćić,et al.  [Vitiligo in childhood]. , 2002, Medicinski pregled.

[20]  A. Taïeb Intrinsic and extrinsic pathomechanisms in vitiligo. , 2000, Pigment cell research.

[21]  Soyun Cho,et al.  Characteristics of Vitiligo in Korean Children , 2000, Pediatric dermatology.

[22]  S. Handa,et al.  Vitiligo: Clinical Findings in 1436 Patients , 1999, The Journal of dermatology.

[23]  J. Lacour [Vitiligo in children]. , 1988, Annales de pediatrie.

[24]  P. Grimes,et al.  Childhood vitiligo. , 1987, Journal of the American Academy of Dermatology.

[25]  K. Thomsen,et al.  Prevalence of vitiligo. Epidemiological survey on the Isle of Bornholm, Denmark. , 1977, Archives of dermatology.