Neuropeptide release influences brain edema formation after diffuse traumatic brain injury.

[1]  H. Doods.  Development of CGRP antagonists for the treatment of migraine. , 2001, Current opinion in investigational drugs.

[2]  R. Hargreaves,et al.  Neurogenic inflammation in the context of migraine , 2001, Microscopy research and technique.

[3]  C. Culmsee,et al.  Adaptive Plasticity in Tachykinin and Tachykinin Receptor Expression after Focal Cerebral Ischemia Is Differentially Linked to GABAergic and Glutamatergic Cerebrocortical Circuits and Cerebrovenular Endothelium , 2001, The Journal of Neuroscience.

[4]  A. Marmarou,et al.  The permissive nature of blood brain barrier (BBB) opening in edema formation following traumatic brain injury. , 2000, Acta neurochirurgica. Supplement.

[5]  M S Kramer,et al.  Discovery of the antidepressant and anti-emetic efficacy of substance P receptor (NK1) antagonists. , 1999, Trends in pharmacological sciences.

[6]  A. Faden,et al.  Effect of traumatic brain injury on mouse spatial and nonspatial learning in the Barnes circular maze. , 1998, Journal of neurotrauma.

[7]  A. Marmarou,et al.  Contribution of vasogenic and cellular edema to traumatic brain swelling measured by diffusion-weighted imaging. , 1997, Journal of neurosurgery.

[8]  H Kashiba,et al.  Systemic capsaicin in the adult rat differentially affects gene expression for neuropeptides and neurotrophin receptors in primary sensory neurons , 1996, Neuroscience.

[9]  R. Vink,et al.  Impact acceleration-induced severe diffuse axonal injury in rats: characterization of phosphate metabolism and neurologic outcome. , 1995, Journal of neurotrauma.

[10]  S. Brain,et al.  An investigation into the mechanism of capsaicin‐induced oedema in rabbit skin , 1995, British journal of pharmacology.

[11]  A. Marmarou,et al.  A new model of diffuse brain injury in rats. Part II: Morphological characterization. , 1994, Journal of neurosurgery.

[12]  B. Pike,et al.  The rotarod test: an evaluation of its effectiveness in assessing motor deficits following traumatic brain injury. , 1994, Journal of neurotrauma.

[13]  R. Reed,et al.  Neurogenic inflammation in rat trachea is accompanied by increased negativity of interstitial fluid pressure. , 1993, Circulation research.

[14]  A. Dray.  Neuropharmacological mechanisms of capsaicin and related substances. , 1992, Biochemical pharmacology.

[15]  C. Barnes.  Memory deficits associated with senescence: a neurophysiological and behavioral study in the rat. , 1979, Journal of comparative and physiological psychology.