Proteomic and histopathological characterization of the interface between oral squamous cell carcinoma invasion fronts and non-cancerous epithelia.

[1]  Tadashi Yamamoto,et al.  Label-free quantitative proteomic analysis reveals strong involvement of complement alternative and terminal pathways in human glomerular sclerotic lesions. , 2015, Journal of proteomics.

[2]  Takanori Kobayashi,et al.  Keratin 17 is co-expressed with 14-3-3 sigma in oral carcinoma in situ and squamous cell carcinoma and modulates cell proliferation and size but not cell migration , 2015, Virchows Archiv.

[3]  M. Ishii,et al.  Filamin acts as a key regulator in epithelial defence against transformed cells , 2014, Nature Communications.

[4]  R. Takagi,et al.  Inflammatory histopathogenesis of nasopalatine duct cyst: a clinicopathological study of 41 cases. , 2013, Oral diseases.

[5]  S. Shingaki,et al.  Intraepithelially entrapped blood vessels in oral carcinoma in-situ , 2012, Virchows Archiv.

[6]  Sameh Magdeldin,et al.  Toward deciphering proteomes of formalin-fixed paraffin-embedded (FFPE) tissues , 2012, Proteomics.

[7]  Takanori Kobayashi,et al.  Loss of keratin 13 in oral carcinoma in situ: a comparative study of protein and gene expression levels using paraffin sections , 2012, Modern Pathology.

[8]  W. Deng,et al.  Cell competition and its implications for development and cancer. , 2011, Journal of genetics and genomics = Yi chuan xue bao.

[9]  Takanori Kobayashi,et al.  Nuclear translocation of β‐catenin synchronized with loss of E‐cadherin in oral epithelial dysplasia with a characteristic two‐phase appearance , 2011, Histopathology.

[10]  Takanori Kobayashi,et al.  Emergence of keratin 17 vs. loss of keratin 13: their reciprocal immunohistochemical profiles in oral carcinoma in situ. , 2011, Oral oncology.

[11]  Takanori Kobayashi,et al.  Enhanced Expression of Podoplanin in Oral Carcinomas in situ and Squamous Cell Carcinomas , 2011, Pathobiology.

[12]  T. Itoh,et al.  Mucin 21/Epiglycanin Modulates Cell Adhesion , 2010, The Journal of Biological Chemistry.

[13]  Jun Cheng,et al.  Histopathological varieties of oral carcinoma in situ: Diagnosis aided by immunohistochemistry dealing with the second basal cell layer as the proliferating center of oral mucosal epithelia , 2010, Pathology international.

[14]  Qiutang Li,et al.  14-3-3sigma controls corneal epithelial cell proliferation and differentiation through the Notch signaling pathway. , 2010, Biochemical and biophysical research communications.

[15]  Guillaume Charras,et al.  Interaction with surrounding normal epithelial cells influences signalling pathways and behaviour of Src-transformed cells , 2010, Journal of Cell Science.

[16]  G. Pitiyage,et al.  Molecular markers in oral epithelial dysplasia: review. , 2009, Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology.

[17]  Robert E. Brown,et al.  Field effect in cancer-an update. , 2009, Annals of clinical and laboratory science.

[18]  Tesshi Yamada,et al.  Quantitative proteomics using formalin‐fixed paraffin‐embedded tissues of oral squamous cell carcinoma , 2009, Cancer science.

[19]  L. Johnston,et al.  Competitive Interactions Between Cells: Death, Growth, and Geography , 2009, Science.

[20]  Daniel C. Liebler,et al.  Equivalence of Protein Inventories Obtained from Formalin-fixed Paraffin-embedded and Frozen Tissue in Multidimensional Liquid Chromatography-Tandem Mass Spectrometry Shotgun Proteomic Analysis* , 2009, Molecular & Cellular Proteomics.

[21]  E. Moreno,et al.  Super competition as a possible mechanism to pioneer precancerous fields. , 2009, Carcinogenesis.

[22]  Jean-Paul Vincent,et al.  Characterization of the interface between normal and transformed epithelial cells , 2009, Nature Cell Biology.

[23]  Wei Li,et al.  Cell competition and its possible relation to cancer. , 2008, Cancer research.

[24]  T. Irimura,et al.  Identification and expression of human epiglycanin/MUC21: a novel transmembrane mucin. , 2007, Glycobiology.

[25]  M. Mann,et al.  Exponentially Modified Protein Abundance Index (emPAI) for Estimation of Absolute Protein Amount in Proteomics by the Number of Sequenced Peptides per Protein*S , 2005, Molecular & Cellular Proteomics.

[26]  W. Arend,et al.  Intracellular IL-1 Receptor Antagonist Type 1 Inhibits IL-1-Induced Cytokine Production in Keratinocytes through Binding to the Third Component of the COP9 Signalosome1 , 2005, The Journal of Immunology.

[27]  L. David,et al.  Keratins 8, 10, 13, and 17 are useful markers in the diagnosis of human cervix carcinomas. , 2004, Human pathology.

[28]  Konrad Basler,et al.  dMyc Transforms Cells into Super-Competitors , 2004, Cell.

[29]  Qing‐Yu He,et al.  Identification of tumor‐associated proteins in oral tongue squamous cell carcinoma by proteomics , 2004, Proteomics.

[30]  B. Graveley.  Alternative splicing: increasing diversity in the proteomic world. , 2001, Trends in genetics : TIG.

[31]  W. Arend,et al.  Biological role of interleukin 1 receptor antagonist isoforms , 2000, Annals of the rheumatic diseases.

[32]  P. Chambon,et al.  Cellular retinol‐binding protein I is essential for vitamin A homeostasis , 1999, The EMBO journal.

[33]  H. Shimizu,et al.  97-kDa linear IgA bullous dermatosis (LAD) antigen localizes to the lamina lucida of the epidermal basement membrane. , 1996, The Journal of investigative dermatology.

[34]  R. Burgeson,et al.  LAD-1, the linear IgA bullous dermatosis autoantigen, is a novel 120-kDa anchoring filament protein synthesized by epidermal cells. , 1996, The Journal of investigative dermatology.

[35]  I. Freedberg,et al.  Regulation of keratin gene expression: the role of the nuclear receptors for retinoic acid, thyroid hormone, and vitamin D3. , 1992, The Journal of investigative dermatology.

[36]  Robert C. Thompson,et al.  Interleukin-1 receptor antagonist activity of a human interleukin-1 inhibitor , 1990, Nature.

[37]  G. Morata,et al.  Minutes: mutants of drosophila autonomously affecting cell division rate. , 1975, Developmental biology.

[38]  A. Wyllie,et al.  Apoptosis: A Basic Biological Phenomenon with Wide-ranging Implications in Tissue Kinetics , 1972, British Journal of Cancer.

[39]  D. Slaughter,et al.  “Field cancerization” in oral stratified squamous epithelium. Clinical implications of multicentric origin , 1953, Cancer.