Bimodal modulation of store-operated Ca2+ channels by clozapine in astrocytes

[1]  M. Fleury,et al.  Comparative effectiveness and safety of antipsychotic drugs in schizophrenia treatment: a real‐world observational study , 2016, Acta psychiatrica Scandinavica.

[2]  R. Casagrande,et al.  Clozapine and olanzapine are better antioxidants than haloperidol, quetiapine, risperidone and ziprasidone in in vitro models. , 2016, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.

[3]  Jiwon Kim,et al.  Clozapine and olanzapine inhibit proton currents in BV2 microglial cells. , 2015, European journal of pharmacology.

[4]  Sang Ki Park,et al.  Disrupted-in-schizophrenia-1 (DISC1) Regulates Endoplasmic Reticulum Calcium Dynamics , 2015, Scientific Reports.

[5]  S. Kędracka-Krok,et al.  Clozapine influences cytoskeleton structure and calcium homeostasis in rat cerebral cortex and has a different proteomic profile than risperidone , 2015, Journal of neurochemistry.

[6]  Jin Hee Hong,et al.  Serotonin-2C receptor involved serotonin-induced Ca2+ mobilisations in neuronal progenitors and neurons in rat suprachiasmatic nucleus , 2014, Scientific Reports.

[7]  T. Yoshioka,et al.  Ca(2+) spiking activity caused by the activation of store-operated Ca(2+) channels mediates TNF-α release from microglial cells under chronic purinergic stimulation. , 2013, Biochimica et biophysica acta.

[8]  C. Gama,et al.  Evidences for a progressive microglial activation and increase in iNOS expression in rats submitted to a neurodevelopmental model of schizophrenia: Reversal by clozapine , 2013, Schizophrenia Research.

[9]  H. Meltzer Update on typical and atypical antipsychotic drugs. , 2013, Annual review of medicine.

[10]  S. Miyamoto,et al.  Pharmacological treatment of schizophrenia: a critical review of the pharmacology and clinical effects of current and future therapeutic agents , 2012, Molecular Psychiatry.

[11]  S. Yamamura,et al.  Clozapine, but not haloperidol, enhances glial d‐serine and L‐glutamate release in rat frontal cortex and primary cultured astrocytes , 2012, British journal of pharmacology.

[12]  H. Rhim,et al.  Inhibition of recombinant Ca(v)3.1 (alpha(1G)) T-type calcium channels by the antipsychotic drug clozapine. , 2010, European journal of pharmacology.

[13]  M. Arif,et al.  Suppressive effect of clozapine but not haloperidol on the increases of neuropeptide-degrading enzymes and glial cells in MK-801-treated rat brain regions , 2007, Neuroscience Research.

[14]  Se Hyun Kim,et al.  Clozapine, a neuroleptic agent, inhibits Akt by counteracting Ca2+/calmodulin in PTEN-negative U-87MG human glioblastoma cells. , 2006, Cellular signalling.

[15]  Andrew Farah,et al.  Atypicality of atypical antipsychotics. , 2005, Primary care companion to the Journal of clinical psychiatry.

[16]  Paul Ernsberger,et al.  H1-Histamine Receptor Affinity Predicts Short-Term Weight Gain for Typical and Atypical Antipsychotic Drugs , 2003, Neuropsychopharmacology.

[17]  D. Braus,et al.  Decreased gene expression of glial and neuronal glutamate transporters after chronic antipsychotic treatment in rat brain , 2003, Neuroscience Letters.

[18]  B. MacVicar,et al.  Expression of voltage‐gated Ca2+ channel subtypes in cultured astrocytes , 2003, Glia.

[19]  G. Rozenberg,et al.  The Ca(2+) channel antagonists mibefradil and pimozide inhibit cell growth via different cytotoxic mechanisms. , 2002, Molecular pharmacology.

[20]  K. Unsicker,et al.  Atypical Neuroleptic Drugs Downregulate Dopamine Sensitivity in Rat Cortical and Striatal Astrocytes , 2001, Molecular and Cellular Neuroscience.

[21]  S. Kapur,et al.  Does fast dissociation from the dopamine d(2) receptor explain the action of atypical antipsychotics?: A new hypothesis. , 2001, The American journal of psychiatry.

[22]  J. Swartz,et al.  Olanzapine treatment after clozapine-induced granulocytopenia in 3 patients. , 1999, The Journal of clinical psychiatry.

[23]  F. Strumwasser,et al.  Antipsychotic drugs block IP3-dependent Ca2+-release from rat brain microsomes , 1996, Biological Psychiatry.

[24]  B. Mlinar,et al.  Preferential block of T-type calcium channels by neuroleptics in neural crest-derived rat and human C cell lines. , 1992, Molecular pharmacology.

[25]  D. Grandy,et al.  Cyclic AMP regulates the calcium transients released from IP(3)-sensitive stores by activation of rat kappa-opioid receptors expressed in CHO cells. , 2001, Cell calcium.