Resveratrol promotes HSV-2 replication by increasing histone acetylation and activating NF-κB.
暂无分享,去创建一个
Shuwen Liu | C. Yang | Wanzhen Lu | Pingzheng Zhou | Xin-feng Xu | P. Jiang | Liqiong Ding
[1] S. Chanda,et al. SNW1, a Novel Transcriptional Regulator of the NF-κB Pathway , 2018, Molecular and Cellular Biology.
[2] G. Freeman,et al. Arming an Oncolytic Herpes Simplex Virus Type 1 with a Single-chain Fragment Variable Antibody against PD-1 for Experimental Glioblastoma Therapy , 2018, Clinical Cancer Research.
[3] N. Mukaida,et al. Resveratrol stimulation induces interleukin‐8 gene transcription via NF‐&kgr;B , 2018, Pharmacological research.
[4] Imran M. Shaikh,et al. Toxicity and Efficacy of a Novel GADD34-expressing Oncolytic HSV-1 for the Treatment of Experimental Glioblastoma , 2018, Clinical Cancer Research.
[5] W. Shao,et al. Bromodomain-containing protein 4–independent transcriptional activation by autoimmune regulator (AIRE) and NF-κB , 2018, The Journal of Biological Chemistry.
[6] Eui Tae Kim,et al. Time-resolved Global and Chromatin Proteomics during Herpes Simplex Virus Type 1 (HSV-1) Infection* , 2017, Molecular & Cellular Proteomics.
[7] Erguang Li,et al. An Epigenetic Compound Library Screen Identifies BET Inhibitors That Promote HSV-1 and -2 Replication by Bridging P-TEFb to Viral Gene Promoters through BRD4 , 2016, PLoS pathogens.
[8] T. Kristie. Dynamic modulation of HSV chromatin drives initiation of infection and provides targets for epigenetic therapies. , 2015, Virology.
[9] Peter Vickerman,et al. Global Estimates of Prevalent and Incident Herpes Simplex Virus Type 2 Infections in 2012 , 2015, PloS one.
[10] D. Hazuda,et al. Activation of HIV Transcription with Short-Course Vorinostat in HIV-Infected Patients on Suppressive Antiretroviral Therapy , 2014, PLoS pathogens.
[11] M. Hagiwara,et al. CDK9 inhibitor FIT-039 prevents replication of multiple DNA viruses. , 2014, The Journal of clinical investigation.
[12] M. Caligiuri,et al. Bortezomib-Induced Unfolded Protein Response Increases Oncolytic HSV-1 Replication Resulting in Synergistic Antitumor Effects , 2014, Clinical Cancer Research.
[13] R. Sandri-Goldin,et al. Inhibition of cdk9 during Herpes Simplex Virus 1 Infection Impedes Viral Transcription , 2013, PloS one.
[14] C. Busch,et al. Correction: Resveratrol as a Pan-HDAC Inhibitor Alters the Acetylation Status of Jistone Proteins in Human-Derived Hepatoblastoma Cells , 2013, PLoS ONE.
[15] C. Busch,et al. Resveratrol as a Pan-HDAC Inhibitor Alters the Acetylation Status of Jistone Proteins in Human-Derived Hepatoblastoma Cells , 2013, PloS one.
[16] Erguang Li,et al. Houttuynia cordata blocks HSV infection through inhibition of NF-κB activation , 2011, Antiviral Research.
[17] J. Pezzuto,et al. What Is New for an Old Molecule? Systematic Review and Recommendations on the Use of Resveratrol , 2011, PloS one.
[18] Craig S. Miller,et al. Histone deacetylase inhibitors induce reactivation of herpes simplex virus type 1 in a latency-associated transcript-independent manner in neuronal cells , 2005, Journal of NeuroVirology.
[19] Yu Liang,et al. Inhibition of the histone demethylase LSD1 blocks α-herpesvirus lytic replication and reactivation from latency , 2009, Nature Medicine.
[20] D. Knipe,et al. Herpes Simplex Virus ICP0 Promotes both Histone Removal and Acetylation on Viral DNA during Lytic Infection , 2008, Journal of Virology.
[21] B. Roizman,et al. Role of cdk9 in the Optimization of Expression of the Genes Regulated by ICP22 of Herpes Simplex Virus 1 , 2008, Journal of Virology.
[22] R. Orlando,et al. Curcumin and resveratrol inhibit nuclear factor-kappaB-mediated cytokine expression in adipocytes , 2008, Nutrition & metabolism.
[23] D. Knipe,et al. Chromatin control of herpes simplex virus lytic and latent infection , 2008, Nature Reviews Microbiology.
[24] N. Fraser,et al. Temporal Association of the Herpes Simplex Virus Genome with Histone Proteins during a Lytic Infection , 2007, Journal of Virology.
[25] L. Kopelovich,et al. Resveratrol: a review of preclinical studies for human cancer prevention. , 2007, Toxicology and applied pharmacology.
[26] Joseph A. Baur,et al. Therapeutic potential of resveratrol: the in vivo evidence , 2006, Nature Reviews Drug Discovery.
[27] B. Marinari,et al. Herpes Simplex Virus Disrupts NF-κB Regulation by Blocking Its Recruitment on the IκBα Promoter and Directing the Factor on Viral Genes* , 2006, Journal of Biological Chemistry.
[28] S. Berger,et al. During Lytic Infection Herpes Simplex Virus Type 1 Is Associated with Histones Bearing Modifications That Correlate with Active Transcription , 2004, Journal of Virology.
[29] F. J. Herrera,et al. VP16-Dependent Association of Chromatin-Modifying Coactivators and Underrepresentation of Histones at Immediate-Early Gene Promoters during Herpes Simplex Virus Infection , 2004, Journal of Virology.
[30] B. Roizman,et al. Herpes Simplex Virus 1 Gene Expression Is Accelerated by Inhibitors of Histone Deacetylases in Rabbit Skin Cells Infected with a Mutant Carrying a cDNA Copy of the Infected-Cell Protein No. 0 , 2003, Journal of Virology.
[31] S. Pervaiz. Resveratrol: from grapevines to mammalian biology , 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[32] C. Amici,et al. NEW EMBO MEMBER’S REVIEW: NF-κB and virus infection: who controls whom , 2003 .
[33] J. Marier,et al. Metabolism and Disposition of Resveratrol in Rats: Extent of Absorption, Glucuronidation, and Enterohepatic Recirculation Evidenced by a Linked-Rat Model , 2002, Journal of Pharmacology and Experimental Therapeutics.
[34] C. Amici,et al. Activation of IκB Kinase by Herpes Simplex Virus Type 1 , 2001, The Journal of Biological Chemistry.
[35] C. Amici,et al. Activation of I kappa b kinase by herpes simplex virus type 1. A novel target for anti-herpetic therapy. , 2001, The Journal of biological chemistry.
[36] B. Peterlin,et al. NF-κB Binds P-TEFb to Stimulate Transcriptional Elongation by RNA Polymerase II , 2001 .
[37] S. Manna,et al. Resveratrol Suppresses TNF-Induced Activation of Nuclear Transcription Factors NF-κB, Activator Protein-1, and Apoptosis: Potential Role of Reactive Oxygen Intermediates and Lipid Peroxidation1 , 2000, The Journal of Immunology.
[38] J. Zeh,et al. Reactivation of genital herpes simplex virus type 2 infection in asymptomatic seropositive persons. , 2000, The New England journal of medicine.
[39] W. Miller,et al. Herpes Simplex Virus Type 1 Induction of Persistent NF-κB Nuclear Translocation Increases the Efficiency of Virus Replication , 1998 .
[40] J. Zeh,et al. Virologic characteristics of subclinical and symptomatic genital herpes infections. , 1995, The New England journal of medicine.
[41] T. Mosmann. Rapid colorimetric assay for cellular growth and survival: application to proliferation and cytotoxicity assays. , 1983, Journal of immunological methods.
[42] E. Kieff,et al. Characterization of herpes simplex virus strains differing in their effects on social behaviour of infected cells. , 1968, The Journal of general virology.