Clinical characteristics of patients with increased urinary excretion of adrenaline in mild to moderate heart failure

Background: We have previously demonstrated that adrenaline (AD) is released into the circulation during acute myocardial infarction and is associated with a more severe clinical course. The role of elevated AD levels in congestive heart failure is not known.

[1]  R. Wolk,et al.  Hypomagnesemia in heart failure with ventricular arrhythmias. Beneficial effects of magnesium supplementation , 2000, Journal of internal medicine.

[2]  S. Anker,et al.  Catecholamine levels and treatment in chronic heart failure. , 1998, European heart journal.

[3]  G. Jennings,et al.  Adrenergic nervous system in heart failure. , 1997, The American journal of cardiology.

[4]  G. Eisenhofer,et al.  Cardiorenal epinephrine kinetics: evidence for neuronal release in the human heart. , 1997, American journal of physiology. Heart and circulatory physiology.

[5]  S. Dubner,et al.  Nonsustained ventricular tachycardia in severe heart failure. Independent marker of increased mortality due to sudden death. GESICA-GEMA Investigators. , 1996, Circulation.

[6]  M J Reiter,et al.  Effects of mechano-electrical feedback: potential arrhythmogenic influence in patients with congestive heart failure. , 1996, Cardiovascular research.

[7]  A. Quyyumi,et al.  Cardiac sympathetic nerve function in congestive heart failure. , 1996, Circulation.

[8]  G. Jennings,et al.  Adverse consequences of high sympathetic nervous activity in the failing human heart. , 1995, Journal of the American College of Cardiology.

[9]  G. Jennings,et al.  Regional epinephrine kinetics in human heart failure: evidence for extra-adrenal, nonneural release. , 1995, The American journal of physiology.

[10]  K. Okumura,et al.  Reduced cardiac extraction of norepinephrine and epinephrine in patients with heart failure--correlation with left ventricular function. , 1994, International journal of cardiology.

[11]  G. Jennings,et al.  Neurochemical evidence of cardiac sympathetic activation and increased central nervous system norepinephrine turnover in severe congestive heart failure. , 1994, Journal of the American College of Cardiology.

[12]  I. Meredith,et al.  Cardiac Sympathetic Nervous Activit in Congestive Heart Failure Evidence for Increased Neuronal Norepinephrine Release and Preserved Neuronal Uptake , 1993, Circulation.

[13]  F. Tristani,et al.  Ejection Fraction, Peak Exercise Oxygen Consumption, Cardiothoracic Ratio, Ventricular Arrhythmias, and Plasma Norepinephrine as Determinants of Prognosis in Heart Failure , 1993, Circulation.

[14]  L. Tavazzi,et al.  Prevalence and Prognostic Significance of Ventricular Arrhythmias After Acute Myocardial Infarction in the Fibrinolytic Era GISSI‐2 Results , 1993, Circulation.

[15]  H. Crijns,et al.  Electrophysiologic profile of ibopamine in patients with congestive heart failure and ventricular tachycardia and relation to its effects on hemodynamics and plasma catecholamines. , 1991, The American journal of cardiology.

[16]  M. A. Plant Hormones regulating cardiovascular function in patients with severe congestive heart failure and their relation to mortality , 1991 .

[17]  K. Swedberg,et al.  Hormones regulating cardiovascular function in patients with severe congestive heart failure and their relation to mortality. CONSENSUS Trial Study Group. , 1990, Circulation.

[18]  B. Massie,et al.  Congestive heart failure: Current controversies and future prospects , 1990 .

[19]  B. Pitt,et al.  Predictors of total mortality and sudden death in mild to moderate heart failure. Captopril-Digoxin Study Group. , 1989, Journal of the American College of Cardiology.

[20]  K. Swedberg,et al.  Endogenous catecholamine levels in chronic heart failure. Relation to the severity of hemodynamic abnormalities. , 1985, The American journal of medicine.

[21]  K. Swedberg,et al.  Comparison of myocardial catecholamine balance in chronic congestive heart failure and in angina pectoris without failure. , 1984, The American journal of cardiology.

[22]  J. Cohn,et al.  Plasma norepinephrine as a guide to prognosis in patients with chronic congestive heart failure. , 1984, The New England journal of medicine.

[23]  J. Cohn,et al.  Free and conjugated plasma catecholamines in patients with congestive heart failure. , 1984, The Journal of laboratory and clinical medicine.

[24]  J. Cohn,et al.  Activity of the sympathetic nervous system and renin-angiotensin system assessed by plasma hormone levels and their relation to hemodynamic abnormalities in congestive heart failure. , 1982, The American journal of cardiology.

[25]  J. Cohn,et al.  Response of plasma norepinephrine and epinephrine to dynamic exercise in patients with congestive heart failure. , 1982, The American journal of cardiology.

[26]  D. Goldstein,et al.  Plasma norepinephrine as an indicator of sympathetic neural activity in clinical cardiology. , 1981, The American journal of cardiology.

[27]  L. Ceremużyński,et al.  Cardiac rhythm disturbances and the release of catecholamines after acute coronary occlusion in dogs. , 1969, Cardiovascular research.

[28]  L. Ceremużyński,et al.  The continuous estimation of catecholamine release in the early stages of myocardial infarction in the dog. , 1968, Clinical science.

[29]  G. Eisenhofer,et al.  Cardiorenal epinephrine kinetics: evidence for neuronal release in the human heart. , 1997, The American journal of physiology.

[30]  P. Kligfield,et al.  Arrhythmias in ischemic and nonischemic dilated cardiomyopathy: prediction of mortality by ambulatory electrocardiography. , 1985, The American journal of cardiology.

[31]  J. Kuch,et al.  Blood and urine catecholamines in recent myocardial infarction, after attack of angina pectoris, and in non-cardiac pain (pleuritis). , 1973, Cor et Vasa.