Peripheral blood mononuclear cells of patients with Indian visceral leishmaniasis suppress natural killer cell activity in vitro.

[1]  S. Adhya,et al.  Detection of Leishmania in the blood of early kala-azar patients with the aid of the polymerase chain reaction. , 1995, Transactions of the Royal Society of Tropical Medicine and Hygiene.

[2]  G. Trinchieri,et al.  IL-12 is required for natural killer cell activation and subsequent T helper 1 cell development in experimental leishmaniasis. , 1995, Journal of immunology.

[3]  S. Ghosh,et al.  Enzymatic amplification of mini-exon-derived RNA gene spacers of Leishmania donovani: primers and probes for DNA diagnosis , 1993, Parasitology.

[4]  S. Bandyopadhyay,et al.  Impairment of natural killer cell activity in Indian kala-azar: restoration of activity by interleukin 2 but not by alpha or gamma interferon , 1993, Infection and immunity.

[5]  S. Reed,et al.  Transforming growth factor beta as a virulence mechanism for Leishmania braziliensis. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[6]  S. Reed,et al.  Transforming growth factor-beta in leishmanial infection: a parasite escape mechanism. , 1992, Science.

[7]  J. Silva,et al.  Selective inability of spleen antigen presenting cells from Leishmania donovani infected hamsters to mediate specific T cell proliferation to parasite antigens , 1992, Parasite immunology.

[8]  K. Mukerji,et al.  Evaluation of direct agglutination test (dat) and elisa for serodiagnosis of visceral leishmaniasis in india , 1991, Journal of clinical laboratory analysis.

[9]  G. Harms,et al.  Natural killer cell activity in visceral leishmaniasis. , 1991, Transactions of the Royal Society of Tropical Medicine and Hygiene.

[10]  P. Das,et al.  Suppression of macrophage lysosomal enzymes after Leishmania donovani infection. , 1989, Biochemical medicine and metabolic biology.

[11]  A. B. Neogy,et al.  Modulation of the cell-mediated immune response in kala-azar and post-kala-azar dermal leishmaniasis in relation to chemotherapy. , 1988, Annals of tropical medicine and parasitology.

[12]  N. Reiner,et al.  Parasite-accessory cell interactions in murine leishmaniasis. II. Leishmania donovani suppresses macrophage expression of class I and class II major histocompatibility complex gene products. , 1987, Journal of immunology.

[13]  N. Reiner Parasite accessory cell interactions in murine leishmaniasis. I. Evasion and stimulus-dependent suppression of the macrophage interleukin 1 response by Leishmania donovani. , 1987, Journal of immunology.

[14]  G. Trinchieri,et al.  Requirement for HLA-DR+ accessory cells in natural killing of cytomegalovirus-infected fibroblasts , 1986, The Journal of experimental medicine.

[15]  C. E. Kirkpatrick,et al.  Splenic natural killer-cell activity in mice infected with Leishmania donovani. , 1984, Cellular immunology.

[16]  A. C. Ghose,et al.  Cell-mediated immune response in Indian kala-azar and post-kala-azar dermal leishmaniasis , 1983, Infection and immunity.

[17]  A. Bryceson,et al.  Immunosuppression in Kenyan visceral leishmaniasis. , 1983, Clinical and experimental immunology.

[18]  W. Johnson,et al.  Cell-mediated immunity in American visceral leishmaniasis: reversible immunosuppression during acute infection , 1981, Infection and immunity.

[19]  E. Reinherz,et al.  A monoclonal antibody reactive with human peripheral blood monocytes. , 1980, Journal of immunology.