Boosting the Exhausted Vasculature—SIRT3 (to the) Rescue
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[1] R. Tuder,et al. SIRT3 Is a Critical Regulator of Mitochondrial Function of Fibroblasts in Pulmonary Hypertension. , 2023, American journal of respiratory cell and molecular biology.
[2] P. Zimmer,et al. Tissue‐specific effects of exercise as NAD+‐boosting strategy: Current knowledge and future perspectives , 2023, Acta physiologica.
[3] C. Ruppert,et al. Epigenetic reactivation of transcriptional programs orchestrating fetal lung development in human pulmonary hypertension , 2022, Science Translational Medicine.
[4] J. Qu,et al. SIRT3 consolidates heterochromatin and counteracts senescence , 2021, Nucleic acids research.
[5] G. Kroemer,et al. Nicotinamide for the treatment of heart failure with preserved ejection fraction , 2021, Science Translational Medicine.
[6] S. Erzurum,et al. Metabolism in Pulmonary Hypertension. , 2021, Annual review of physiology.
[7] J. Barberà,et al. Standardized exercise training is feasible, safe, and effective in pulmonary arterial and chronic thromboembolic pulmonary hypertension: results from a large European multicentre randomized controlled trial. , 2020, European heart journal.
[8] R. Tuder,et al. Stable isotope metabolomics of pulmonary artery smooth muscle and endothelial cells in pulmonary hypertension and with TGF-beta treatment , 2020, Scientific Reports.
[9] F. Cheng,et al. Integrative proteomics and phosphoproteomics in pulmonary arterial hypertension , 2019, Scientific Reports.
[10] P. Carmeliet,et al. Identification of MicroRNA-124 as a Major Regulator of Enhanced Endothelial Cell Glycolysis in Pulmonary Arterial Hypertension via PTBP1 (Polypyrimidine Tract Binding Protein) and Pyruvate Kinase M2 , 2017, Circulation.
[11] P. Ježek,et al. Metabolic and Proliferative State of Vascular Adventitial Fibroblasts in Pulmonary Hypertension Is Regulated Through a MicroRNA-124/PTBP1 (Polypyrimidine Tract Binding Protein 1)/Pyruvate Kinase Muscle Axis , 2017, Circulation.
[12] James D. Thomas,et al. Metabolic and Functional Evaluation of the Heart and Lungs in Pulmonary Hypertension by Gated 2-[18F]-Fluoro-2-deoxy-D-glucose Positron Emission Tomography , 2017, Pulmonary circulation.
[13] J. Loscalzo,et al. Vascular stiffness mechanoactivates YAP/TAZ-dependent glutaminolysis to drive pulmonary hypertension. , 2016, The Journal of clinical investigation.
[14] S. Pullamsetti,et al. Constitutive Reprogramming of Fibroblast Mitochondrial Metabolism in Pulmonary Hypertension. , 2016, American journal of respiratory cell and molecular biology.
[15] S. Provencher,et al. Sirtuin 3 deficiency is associated with inhibited mitochondrial function and pulmonary arterial hypertension in rodents and humans. , 2014, Cell metabolism.
[16] L. Gleaves,et al. Evidence for right ventricular lipotoxicity in heritable pulmonary arterial hypertension. , 2014, American journal of respiratory and critical care medicine.
[17] J. Marks,et al. Sirtuin 3 deficiency does not augment hypoxia-induced pulmonary hypertension. , 2013, American journal of respiratory cell and molecular biology.
[18] S. Pullamsetti,et al. Heterogeneity in Lung 18FDG Uptake in Pulmonary Arterial Hypertension: Potential of Dynamic 18FDG Positron Emission Tomography With Kinetic Analysis as a Bridging Biomarker for Pulmonary Vascular Remodeling Targeted Treatments , 2013, Circulation.
[19] Enxuan Jing,et al. Sirtuin-3 (Sirt3) regulates skeletal muscle metabolism and insulin signaling via altered mitochondrial oxidation and reactive oxygen species production , 2011, Proceedings of the National Academy of Sciences.
[20] P. Harris. Editorials , 1911, Paraplegia.