Molecular analysis of C3 allotypes in Chinese patients with immunoglobulin A nephropathy.

[1]  Mathieson Pw,et al.  Deficiency and depletion of complement in the pathogenesis of nephritis and vasculitis. , 1993 .

[2]  P. Mathieson,et al.  Molecular analysis of C3 allotypes in patients with nephritic factor , 1993, Clinical and experimental immunology.

[3]  A. Rees,et al.  Familial IgA nephropathy: a study of HLA class II allogenotypes in a Chinese kindred. , 1992, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[4]  M. Endoh,et al.  Increased frequency of the heterozygous switch region of IgA2 in Japanese patients with IgA nephropathy. , 1992, Internal medicine.

[5]  X. Chen.  [HLA-DR gene frequencies in IgA nephropathy patients obtained by oligonucleotide genotyping]. , 1992, Zhonghua yi xue za zhi.

[6]  J. Feehally,et al.  The DQw7 allele at the HLA-DQB locus is associated with susceptibility to IgA nephropathy in Caucasians. , 1991, Kidney international.

[7]  B. Julian,et al.  Role for specific complement phenotypes and deficiencies in the clinical expression of IgA nephropathy. , 1991, The American journal of the medical sciences.

[8]  M. Walport,et al.  Molecular basis of polymorphisms of human complement component C3 , 1990, The Journal of experimental medicine.

[9]  G. Hitman,et al.  HLA DQ region gene polymorphism associated with primary IgA nephropathy. , 1990, Kidney international.

[10]  I. Nakanishi,et al.  Complement C6 and C7 polymorphisms in Japanese patients with chronic glomerulonephritis. , 1989, Human heredity.

[11]  C Summers,et al.  Analysis of any point mutation in DNA. The amplification refractory mutation system (ARMS). , 1989, Nucleic acids research.

[12]  B. Julian,et al.  Complement phenotypes in glomerulonephritis: increased frequency of homozygous null C4 phenotypes in IgA nephropathy and Henoch-Schönlein purpura. , 1984, Kidney international.

[13]  J. Winkelstein,et al.  Genetically determined variation in the complement system: relationship to disease. , 1984, The Journal of pediatrics.

[14]  Z. Tongmao.  Genetic polymorphisms of C3 and Bf in the Chinese population. , 1983, Human heredity.

[15]  D. Graham,et al.  The isolation of high molecular weight DNA from whole organisms or large tissue masses. , 1978, Analytical biochemistry.

[16]  H. Arvilommi.  Capacity of complement C3 phenotypes to bind on to mononuclear cells in man , 1974, Nature.

[17]  P. Teisberg.  High Voltage Agarose Gel Electrophoresis in the Study of C 3 Polymorphism , 1970, Vox sanguinis.

[18]  C. Alper,et al.  Genetic polymorphism of the third component of human complement (C'3). , 1968, The Journal of clinical investigation.

[19]  T. Kohsaka,et al.  Complement 4 gene deletion in patients with IgA nephropathy and Henoch-Schönlein nephritis. , 1992, Child nephrology and urology.

[20]  S. A. Hu,et al.  HLA and Chinese IgA nephropathy in Taiwan. , 1989, Tissue antigens.

[21]  M. Rambausek,et al.  Genetic polymorphism of C3 and Bf in IgA nephropathy. , 1987, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[22]  L. M. Srivastava,et al.  Association between C3 complement types and Indian childhood cirrhosis. , 1985, Human heredity.

[23]  R. Brönnestam.  Studies on the C3 polymorphism. Relationship between phenotype and quantitative immunochemical measurements. , 1973, Human heredity.