Calcitonin gene-related peptide immunoreactivity in chronic human spinal cord injury

[1]  A. Krassioukov,et al.  Malignant autonomic dysreflexia in spinal cord injured men , 2006, Spinal Cord.

[2]  A William Sheel,et al.  Autonomic dysreflexia during sperm retrieval in spinal cord injury: influence of lesion level and sildenafil citrate. , 2005, Journal of applied physiology.

[3]  Jonas Frisén,et al.  Allodynia limits the usefulness of intraspinal neural stem cell grafts; directed differentiation improves outcome , 2005, Nature Neuroscience.

[4]  J. Borisoff,et al.  Deafferentation and neurotrophin‐mediated intraspinal sprouting: a central role for the p75 neurotrophin receptor , 2005, The European journal of neuroscience.

[5]  H. Bidmon,et al.  Influence of post-mortem delay and storage temperature on the immunohistochemical detection of antigens in the CNS of mice. , 2004, Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie.

[6]  L. T. McPhail,et al.  A soluble Nogo receptor differentially affects plasticity of spinally projecting axons , 2004, The European journal of neuroscience.

[7]  W. Tetzlaff,et al.  Peripherally–derived olfactory ensheathing cells do not promote primary afferent regeneration following dorsal root injury , 2004, Glia.

[8]  A. Krassioukov,et al.  Descending vasomotor pathways in humans: correlation between axonal preservation and cardiovascular dysfunction after spinal cord injury. , 2003, Journal of neurotrauma.

[9]  S. Ohtori,et al.  Calcitonin gene-related peptide immunoreactive neurons with dichotomizing axons projecting to the lumbar muscle and knee in rats , 2003, European Spine Journal.

[10]  C. Hulsebosch,et al.  Direct evidence of primary afferent sprouting in distant segments following spinal cord injury in the rat: colocalization of GAP-43 and CGRP , 2003, Experimental Neurology.

[11]  S. McMahon,et al.  Glial cell line‐derived neurotrophic factor increases calcitonin gene‐related peptide immunoreactivity in sensory and motoneurons in vivo , 2003, The European journal of neuroscience.

[12]  A. Krassioukov,et al.  Autonomic dysreflexia associated with intramedullary astrocytoma of the spinal cord. , 2003, The Lancet. Oncology.

[13]  A. Krassioukov,et al.  Autonomic dysreflexia in acute spinal cord injury: an under-recognized clinical entity. , 2003, Journal of neurotrauma.

[14]  Hideshige Moriya,et al.  Calcitonin gene-related peptide immunoreactive DRG neurons innervating the cervical facet joints show phenotypic switch in cervical facet injury in rats , 2003, European Spine Journal.

[15]  M. Boninger,et al.  Autonomic Dysreflexia: Incidence In Persons With Neurologically Complete And Incomplete Tetraplegia , 2003, The journal of spinal cord medicine.

[16]  A. Krassioukov,et al.  Sensitivity of sympathetically correlated spinal interneurons, renal sympathetic nerve activity, and arterial pressure to somatic and visceral stimuli after chronic spinal injury. , 2002, Journal of neurotrauma.

[17]  S. Meakin,et al.  Neutralizing intraspinal nerve growth factor with a trkA-IgG fusion protein blocks the development of autonomic dysreflexia in a clip-compression model of spinal cord injury. , 2002, Journal of neurotrauma.

[18]  S. Kirshblum,et al.  Silent autonomic dysreflexia during a routine bowel program in persons with traumatic spinal cord injury: a preliminary study. , 2002, Archives of physical medicine and rehabilitation.

[19]  S. Ohtori,et al.  Calcitonin gene-related peptide immunoreactive sensory DRG neurons innervating the cervical facet joints in rats , 2002, Journal of orthopaedic science : official journal of the Japanese Orthopaedic Association.

[20]  M. Fehlings,et al.  Autonomic dysreflexia and primary afferent sprouting after clip-compression injury of the rat spinal cord. , 2001, Journal of neurotrauma.

[21]  G. Savić,et al.  The relationship between neurological level of injury and symptomatic cardiovascular disease risk in the aging spinal injured , 2001, Spinal Cord.

[22]  S. Demirel,et al.  Risk factors for coronary heart disease in patients with spinal cord injury in Turkey , 2001, Spinal Cord.

[23]  K. Stiller,et al.  The prevalence of orthostatic hypotension during physiotherapy treatment in patients with an acute spinal cord injury , 2000, Spinal Cord.

[24]  G. Savić,et al.  Hospital readmissions in people with chronic spinal cord injury , 2000, Spinal Cord.

[25]  A. Krassioukov,et al.  Cardiovascular consequences of loss of supraspinal control of the sympathetic nervous system after spinal cord injury. , 2000, Archives of physical medicine and rehabilitation.

[26]  J. Silver.  Early autonomic dysreflexia , 2000, Spinal Cord.

[27]  A. Krassioukov,et al.  Neutralizing Intraspinal Nerve Growth Factor Blocks Autonomic Dysreflexia Caused By Spinal Cord Injury , 1999, The Journal of Neuroscience.

[28]  J. Silver.  Autonomic dysreflexia during urodynamics. A Giannantoni et al. Spinal Cord 1998; 36: 756–760 , 1999, Spinal Cord.

[29]  L. Weaver,et al.  Sprouting of primary afferent fibers after spinal cord transection in the rat , 1998, Neuroscience.

[30]  T. Fukuoka,et al.  Calcitonin gene-related peptide increase in the rat spinal dorsal horn and dorsal column nucleus following peripheral nerve injury: Up-regulation in a subpopulation of primary afferent sensory neurons , 1997, Neuroscience.

[31]  C. Hulsebosch,et al.  Spinal Cord Injury and Anti-NGF Treatment Results in Changes in CGRP Density and Distribution in the Dorsal Horn in the Rat , 1997, Experimental Neurology.

[32]  V. Dietz,et al.  Assessment of autonomic dysreflexia in patients with spinal cord injury. , 1997, Journal of neurology, neurosurgery, and psychiatry.

[33]  R. Waters,et al.  International Standards for Neurological and Functional Classification of Spinal Cord Injury , 1997, Spinal Cord.

[34]  B. Kakulas,et al.  Neuropathology of human spinal cord injury sustained in sports-related activities. , 1997, Journal of neurotrauma.

[35]  A. Krassioukov,et al.  Relationship between sympathetic activity and arterial pressure in conscious spinal rats. , 1997, The American journal of physiology.

[36]  K. Crutcher,et al.  Plasticity of mature sensory cerebrovascular axons following intracranial infusion of nerve growth factor , 1995, The Journal of comparative neurology.

[37]  S. McMahon,et al.  Immunocytochemical Localization of trkA Receptors in Chemically Identified Subgroups of Adult Rat Sensory Neurons , 1995, The European journal of neuroscience.

[38]  S. Stiens,et al.  Cardiac rehabilitation in patients with spinal cord injuries , 1995 .

[39]  M. Tuszynski,et al.  Fibroblasts Genetically Modified to Produce Nerve Growth Factor Induce Robust Neuritic Ingrowth after Grafting to the Spinal Cord , 1994, Experimental Neurology.

[40]  W. Donovan,et al.  The International Standards Booklet for Neurological and Functional Classification of Spinal Cord Injury , 1994, Paraplegia.

[41]  D. Troost,et al.  Tissue fixation methods alter the immunohistochemical demonstrability of neurofilament proteins, synaptophysin, and glial fibrillary acidic protein in human cerebellum. , 1993, Acta histochemica.

[42]  R. Kim,et al.  Fatal cerebral hemorrhage due to autonomic dysreflexia in a tetraplegic patient: case report and review , 1992, Paraplegia.

[43]  N. Sonty.  Physical medicine and rehabilitation: State of the art reviews. Musculoskeletal pain , 1991, Journal of Occupational Rehabilitation.

[44]  T. Hökfelt.  Neuropeptides in perspective: The last ten years , 1991, Neuron.

[45]  J. Wrathall,et al.  Increase in nerve growth factor-like immunoreactivity and decrease in choline acetyltransferase following contusive spinal cord injury , 1991, Brain Research.

[46]  S. Lawson,et al.  Cell type and conduction velocity of rat primary sensory neurons with calcitonin gene-related peptide-like immunoreactivity , 1990, Neuroscience.

[47]  J. Sobrino,et al.  Visceral and somatic afferent origin of calcitonin gene-related peptide immunoreactivity in the lower thoracic spinal cord of the rat , 1989, Neuroscience.

[48]  M. Yekutiel,et al.  The prevalence of hypertension, ischaemic heart disease and diabetes in traumatic spinal cord injured patients and amputees , 1989, Paraplegia.

[49]  H. Takagi,et al.  Calcitonin gene-related peptide promotes mechanical nociception by potentiating release of substance P from the spinal dorsal horn in rats , 1987, Brain Research.

[50]  T. Hökfelt,et al.  Primary sensory neurons of the rat showing calcitonin gene-related peptide immunoreactivity and their relation to substance P-, somatostatin-, galanin-, vasoactive intestinal polypeptide- and cholecystokinin-immunoreactive ganglion cells , 1987, Cell and Tissue Research.

[51]  J. Mcgregor,et al.  Autonomic hyperreflexia: a mortal danger for spinal cord-damaged women in labor. , 1985, American journal of obstetrics and gynecology.

[52]  M. Ghatei,et al.  Calcitonin gene-related peptide immunoreactivity in the spinal cord of man and of eight other species , 1984, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[53]  T. Hökfelt,et al.  Immunoreactive calcitonin gene-related peptide and substance P coexist in sensory neurons to the spinal cord and interact in spinal behavioral responses of the rat , 1984, Neuroscience Letters.

[54]  L. Chui,et al.  AUTONOMIC DYSREFLEXIA , 1983, Rehabilitation nursing : the official journal of the Association of Rehabilitation Nurses.

[55]  A. Freehafer,et al.  Autonomic dysreflexia. A cause of morbidity and mortality in orthopedic patients with spinal cord injury. , 1982, Clinical orthopaedics and related research.

[56]  H. Head,et al.  THE AUTOMATIC BLADDER, EXCESSIVE SWEATING AND SOME OTHER REFLEX CONDITIONS, IN GROSS INJURIES OF THE SPINAL CORD , 1917 .

[57]  A. Krassioukov,et al.  Assessment of autonomic dysfunction following spinal cord injury: rationale for additions to International Standards for Neurological Assessment. , 2007, Journal of rehabilitation research and development.

[58]  R. Ruff,et al.  Directed rehabilitation reduces pain and depression while increasing independence and satisfaction with life for patients with paraplegia due to epidural metastatic spinal cord compression. , 2007, Journal of rehabilitation research and development.

[59]  A. Krassioukov,et al.  Autonomic Standards and SCI: Preliminary Considerations , 2006 .

[60]  B. Kakulas,et al.  A review of the neuropathology of human spinal cord injury with emphasis on special features. , 1999, The journal of spinal cord medicine.

[61]  R. Bunge,et al.  The changes in human spinal sympathetic preganglionic neurons after spinal cord injury , 1999, Spinal Cord.

[62]  A. Krassioukov,et al.  Reflex and morphological changes in spinal preganglionic neurons after cord injury in rats. , 1995, Clinical and experimental hypertension.

[63]  R. Bunge,et al.  Calcitonin gene-related peptide (CGRP)-like immunoreactivity in motoneurons of the human spinal cord following injury. , 1994, Journal of neurotrauma.

[64]  R. Quencer,et al.  Observations on the pathology of human spinal cord injury. A review and classification of 22 new cases with details from a case of chronic cord compression with extensive focal demyelination. , 1993, Advances in neurology.

[65]  G. Mitchell.  Anatomy of the autonomic nervous system , 1953 .