Discrepancies in the clinical utility of the 14-3-3 protein for the diagnosis of sporadic Creutzfeldt-Jakob disease.
暂无分享,去创建一个
[1] J. Ahlskog,et al. Myelopathy due to copper deficiency following gastrointestinal surgery. , 2003, Archives of neurology.
[2] J. Gross,et al. Myelopathy due to copper deficiency , 2003, Neurology.
[3] N. Barbaro,et al. Challenging the clinical utility of the 14-3-3 protein for the diagnosis of sporadic Creutzfeldt-Jakob disease. , 2003, Archives of neurology.
[4] A. Aksamit. Cerebrospinal fluid 14-3-3 protein: variability of sporadic Creutzfeldt-Jakob disease, laboratory standards, and quantitation. , 2003, Archives of neurology.
[5] N. R. Holland,et al. CNS demyelination associated with copper deficiency and hyperzincemia , 2002, Neurology.
[6] H. Stuerenburg,et al. Copper deficiency-associated myelopathy in a 45-year-old woman , 2001, Journal of Neurology.
[7] J. Yagüe,et al. The impact of the introduction of the 14-3-3 protein assay in the surveillance of sporadic Creutzfeldt-Jakob disease in Catalonia , 2001, Journal of Neurology.
[8] H. Budka,et al. Analysis of EEG and CSF 14-3-3 proteins as aids to the diagnosis of Creutzfeldt–Jakob disease , 2000, Neurology.
[9] B. Steinhoff,et al. Current clinical diagnosis in Creutzfeldt‐Jakob disease: Identification of uncommon variants , 2000, Annals of neurology.
[10] E. Tolosa,et al. Detection of 14‐3‐3 brain protein in the cerebrospinal fluid of patients with paraneoplastic neurological disorders , 1999, Annals of neurology.
[11] P. Castaigne,et al. Creutzfeldt‐Jakob disease: Clinical analysis of a consecutive series of 230 neuropathologically verified cases , 1986, Annals of neurology.
[12] S. Jc,et al. Characterization of zinc-binding proteins of plasma in familial hyperzincemia. , 1982 .
[13] A. Prasad,et al. Hypocupremia induced by zinc therapy in adults. , 1978, JAMA.
[14] J. C. Smith,et al. Elevated plasmz zinc: a heritable anomaly. , 1976, Science.