The cytosolic sialidase Neu2 is degraded by autophagy during myoblast atrophy.
暂无分享,去创建一个
F. Colombo | W. Martinet | P. Costelli | R. Giuliani | A. Fanzani | A. Bonetto | S. Rossi | E. Stoppani | A. Preti | S. Marchesini
[1] M. A. Chambers,et al. TNF induction of atrogin-1/MAFbx mRNA depends on Foxo4 expression but not AKT-Foxo1/3 signaling. , 2008, American journal of physiology. Cell physiology.
[2] F. Colombo,et al. The enzymatic activity of sialidase Neu2 is inversely regulated during in vitro myoblast hypertrophy and atrophy. , 2008, Biochemical and biophysical research communications.
[3] A. Goldberg,et al. FoxO3 coordinately activates protein degradation by the autophagic/lysosomal and proteasomal pathways in atrophying muscle cells. , 2007, Cell metabolism.
[4] A. Goldberg,et al. FoxO3 controls autophagy in skeletal muscle in vivo. , 2007, Cell metabolism.
[5] C. Scuoppo,et al. Conditional Activation of MET in Differentiated Skeletal Muscle Induces Atrophy* , 2007, Journal of Biological Chemistry.
[6] C. Stewart,et al. Waste management - cytokines, growth factors and cachexia. , 2006, Cytokine & growth factor reviews.
[7] F. Colombo,et al. Insulin‐like growth factor 1 signaling regulates cytosolic sialidase Neu2 expression during myoblast differentiation and hypertrophy , 2006, The FEBS journal.
[8] M. Tisdale,et al. Mechanism of induction of muscle protein degradation by angiotensin II. , 2006, Cellular signalling.
[9] E. Clementi,et al. Follistatin induction by nitric oxide through cyclic GMP: a tightly regulated signaling pathway that controls myoblast fusion , 2006, The Journal of cell biology.
[10] D. Taillandier,et al. Lysosomal proteolysis in skeletal muscle. , 2005, The international journal of biochemistry & cell biology.
[11] S. Lecker,et al. Ubiquitin-protein ligases in muscle wasting. , 2005, The international journal of biochemistry & cell biology.
[12] P. Costelli,et al. Ca(2+)-dependent proteolysis in muscle wasting. , 2005, The international journal of biochemistry & cell biology.
[13] Mark M. Kockx,et al. Amino Acid Deprivation Induces Both Apoptosis and Autophagy in Murine C2C12 Muscle Cells , 2005, Biotechnology Letters.
[14] M. Tisdale,et al. Angiotensin II directly induces muscle protein catabolism through the ubiquitin–proteasome proteolytic pathway and may play a role in cancer cachexia , 2005, British Journal of Cancer.
[15] D. Mann,et al. TNF-acts via p 38 MAPK to stimulate expression of the ubiquitin ligase atrogin 1 / MAFbx in skeletal muscle , 2005 .
[16] N. Mizushima. Methods for monitoring autophagy. , 2004, The international journal of biochemistry & cell biology.
[17] A. Goldberg,et al. IGF-I stimulates muscle growth by suppressing protein breakdown and expression of atrophy-related ubiquitin ligases, atrogin-1 and MuRF1. , 2004, American journal of physiology. Endocrinology and metabolism.
[18] V. Sartorelli,et al. Molecular and Cellular Determinants of Skeletal Muscle Atrophy and Hypertrophy , 2004, Science's STKE.
[19] S. Furuta,et al. Ras is involved in the negative control of autophagy through the class I PI3-kinase , 2004, Oncogene.
[20] Eric P Hoffman,et al. Deacetylase inhibitors increase muscle cell size by promoting myoblast recruitment and fusion through induction of follistatin. , 2004, Developmental cell.
[21] Marco Sandri,et al. Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle Atrophy , 2004, Cell.
[22] G. Borsani,et al. Properties of Recombinant Human Cytosolic Sialidase HsNEU2 , 2004, Journal of Biological Chemistry.
[23] W. Mitch,et al. Activation of caspase-3 is an initial step triggering accelerated muscle proteolysis in catabolic conditions. , 2004, The Journal of clinical investigation.
[24] D. Glass,et al. Molecular mechanisms modulating muscle mass. , 2003, Trends in molecular medicine.
[25] F. Colombo,et al. Overexpression of cytosolic sialidase Neu2 induces myoblast differentiation in C2C12 cells , 2003, FEBS letters.
[26] D. E. Goll,et al. The calpain system. , 2003, Physiological reviews.
[27] G. Pavlath,et al. IL-4 Acts as a Myoblast Recruitment Factor during Mammalian Muscle Growth , 2003, Cell.
[28] M. Drost,et al. Lysosomal dysfunction in muscle with special reference to glycogen storage disease type II. , 2003, Biochimica et biophysica acta.
[29] D. Glass. Signalling pathways that mediate skeletal muscle hypertrophy and atrophy , 2003, Nature Cell Biology.
[30] M. Nakagawa,et al. The first molecular evidence that autophagy relates rimmed vacuole formation in chloroquine myopathy. , 2002, Journal of biochemistry.
[31] M. Sandri. Apoptotic signaling in skeletal muscle fibers during atrophy , 2002, Current opinion in clinical nutrition and metabolic care.
[32] A. Goldberg,et al. Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[33] D. Attaix,et al. Identification of cathepsin L as a differentially expressed message associated with skeletal muscle wasting. , 2001, The Biochemical journal.
[34] G. Yancopoulos,et al. Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo , 2001, Nature Cell Biology.
[35] D J Glass,et al. Identification of Ubiquitin Ligases Required for Skeletal Muscle Atrophy , 2001, Science.
[36] M. Colombo,et al. A novel assay to study autophagy: regulation of autophagosome vacuole size by amino acid deprivation. , 2001, Journal of cell science.
[37] D. Turk,et al. Lysosomal cysteine proteases: facts and opportunities , 2001, The EMBO journal.
[38] J. Müthing,et al. Characterization of cytosolic sialidase from Chinese hamster ovary cells: part II. Substrate specificity for gangliosides. , 2001, Carbohydrate research.
[39] J. Müthing,et al. Characterization of cytosolic sialidase from Chinese hamster ovary cells: part I: cloning and expression of soluble sialidase in Escherichia coli. , 2001, Carbohydrate research.
[40] Antonio Musarò,et al. Localized Igf-1 transgene expression sustains hypertrophy and regeneration in senescent skeletal muscle , 2001, Nature Genetics.
[41] Yi-Ping Li,et al. NF-κB mediates the protein loss induced by TNF-α in differentiated skeletal muscle myotubes , 2000 .
[42] D. Figarella-Branger,et al. Linkage of X-linked myopathy with excessive autophagy (XMEA) to Xq28 , 2000, European Journal of Human Genetics.
[43] A. Musarò,et al. Maturation of the Myogenic Program Is Induced by Postmitotic Expression of Insulin-Like Growth Factor I , 1999, Molecular and Cellular Biology.
[44] A. Goldberg,et al. Muscle protein breakdown and the critical role of the ubiquitin-proteasome pathway in normal and disease states. , 1999, The Journal of nutrition.
[45] C. Guézennec,et al. Coordinate activation of lysosomal, Ca 2+-activated and ATP-ubiquitin-dependent proteinases in the unweighted rat soleus muscle. , 1996, The Biochemical journal.
[46] K. Sato,et al. Involvement of an endogenous sialidase in skeletal muscle cell differentiation. , 1996, Biochemical and biophysical research communications.
[47] D. Taillandier,et al. Sensitivity and protein turnover response to glucocorticoids are different in skeletal muscle from adult and old rats. Lack of regulation of the ubiquitin-proteasome proteolytic pathway in aging. , 1995, The Journal of clinical investigation.
[48] Robert J. Schwartz,et al. Myogenic Vector Expression of Insulin-like Growth Factor I Stimulates Muscle Cell Differentiation and Myofiber Hypertrophy in Transgenic Mice (*) , 1995, The Journal of Biological Chemistry.
[49] A. Goldberg,et al. Inhibitors of the proteasome block the degradation of most cell proteins and the generation of peptides presented on MHC class I molecules , 1994, Cell.
[50] H. Kawasaki,et al. Molecular cloning and expression of cDNA encoding rat skeletal muscle cytosolic sialidase. , 1993, The Journal of biological chemistry.
[51] M. Rechsteiner,et al. Degradation of microinjected proteins: Effects of lysosomotropic agents and inhibitors of autophagy , 1983, Journal of cellular physiology.
[52] P. Seglen,et al. 3-Methyladenine: specific inhibitor of autophagic/lysosomal protein degradation in isolated rat hepatocytes. , 1982, Proceedings of the National Academy of Sciences of the United States of America.
[53] S. Dimauro,et al. Lysosomal glycogen storage disease with normal acid maltase , 1981, Neurology.