Intimal proliferation of smooth muscle cells as an explanation for recurrent coronary artery stenosis after percutaneous transluminal coronary angioplasty.
暂无分享,去创建一个
N. Ratliff | J. Hollman | D. Phillips | G. Austin | S. Tabei | G. Austin | Garth E. Austin | Garth E. Austin
[1] J. Douglas,et al. Coronary artery spasm at the site of angioplasty in the first 2 months after successful percutaneous transluminal coronary angioplasty. , 1983, Journal of the American College of Cardiology.
[2] T. Ryan,et al. The mechanism of transluminal angioplasty: evidence for formation of aneurysms in experimental atherosclerosis. , 1983, Circulation.
[3] A E Becker,et al. Transluminal coronary angioplasty and early restenosis. Fibrocellular occlusion after wall laceration. , 1983, British heart journal.
[4] B. McManus,et al. Status of the major epicardial coronary arteries 80 to 150 days after percutaneous transluminal coronary angioplasty. Analysis of 3 necropsy patients. , 1983, The American journal of cardiology.
[5] A. Gruentzig,et al. Results from coronary angioplasty and implications for the future. , 1982, American heart journal.
[6] R. Bonow,et al. Improved myocardial function during exercise after successful percutaneous transluminal coronary angioplasty. , 1982, The New England journal of medicine.
[7] J. Fallon,et al. Morphology after transluminal angioplasty in human beings. , 1981, The New England journal of medicine.
[8] T. Cr. Immunoperoxidase techniques: practical and theoretical aspects. , 1978, Archives of pathology & laboratory medicine.
[9] C. Taylor. Immunoperoxidase techniques: practical and theoretical aspects. , 1978, Archives of pathology & laboratory medicine.