Assessment of pulmonary fibrosis induced by paraquat using Al^18F-NODA-FAPI-04 PET/CT

[1]  C. Qin,et al.  Noninvasive Monitoring of Reparative Fibrosis after Myocardial Infarction in Rats Using 68Ga-FAPI-04 PET/CT. , 2022, Molecular pharmaceutics.

[2]  P. Zhang,et al.  Gallium-68-labeled fibroblast activation protein inhibitor PET in gastrointestinal cancer: insights into diagnosis and management , 2022, European Journal of Nuclear Medicine and Molecular Imaging.

[3]  A. McMillan,et al.  [68 Ga]Ga-FAPI-46 PET for non-invasive detection of pulmonary fibrosis disease activity , 2022, European Journal of Nuclear Medicine and Molecular Imaging.

[4]  Chunjing Yu,et al.  Research progress on the role of fibroblast activation protein in diagnosis and treatment of cancer , 2022, Nuclear medicine communications.

[5]  W. Cai,et al.  Clinical summary of fibroblast activation protein inhibitor-based radiopharmaceuticals: cancer and beyond , 2022, European Journal of Nuclear Medicine and Molecular Imaging.

[6]  Xing Zhou,et al.  FAPI-04 PET/CT Using [18F]AlF Labeling Strategy: Automatic Synthesis, Quality Control, and In Vivo Assessment in Patient , 2021, Frontiers in Oncology.

[7]  G. Schett,et al.  68Ga-FAPI-04 PET-CT for molecular assessment of fibroblast activation and risk evaluation in systemic sclerosis-associated interstitial lung disease: a single-centre, pilot study. , 2021, The Lancet. Rheumatology.

[8]  Yue Chen,et al.  Value of [68Ga]Ga-FAPI-04 imaging in the diagnosis of renal fibrosis , 2021, European Journal of Nuclear Medicine and Molecular Imaging.

[9]  Nan Li,et al.  Clinical translational evaluation of Al18F-NOTA-FAPI for fibroblast activation protein-targeted tumour imaging , 2021, European Journal of Nuclear Medicine and Molecular Imaging.

[10]  B. Hutton,et al.  Pulmonary 18F-FDG uptake helps refine current risk stratification in idiopathic pulmonary fibrosis (IPF) , 2018, European Journal of Nuclear Medicine and Molecular Imaging.

[11]  Baishen Sun,et al.  [Paraquat poisoning mechanism and its clinical treatment progress]. , 2017, Zhonghua wei zhong bing ji jiu yi xue.

[12]  K. Togashi,et al.  18F-FDG Uptake in Less Affected Lung Field Provides Prognostic Stratification in Patients with Interstitial Lung Disease , 2016, The Journal of Nuclear Medicine.

[13]  B. Sun,et al.  Advances in the mechanism of paraquat-induced pulmonary injury. , 2016, European review for medical and pharmacological sciences.

[14]  J. Connett,et al.  Fibrotic extracellular matrix activates a profibrotic positive feedback loop. , 2014, The Journal of clinical investigation.

[15]  B. Hutton,et al.  Areas of normal pulmonary parenchyma on HRCT exhibit increased FDG PET signal in IPF patients , 2013, European Journal of Nuclear Medicine and Molecular Imaging.

[16]  E. Puré,et al.  Fibroblast activation protein in remodeling tissues. , 2012, Current molecular medicine.

[17]  T. Wynn,et al.  Mechanisms of fibrosis: therapeutic translation for fibrotic disease , 2012, Nature Medicine.

[18]  J. Duarte,et al.  Paraquat Poisonings: Mechanisms of Lung Toxicity, Clinical Features, and Treatment , 2008, Critical reviews in toxicology.

[19]  E. Puré,et al.  Fibroblast activation protein: a serine protease expressed at the remodeling interface in idiopathic pulmonary fibrosis. , 2006, Human pathology.