Basophils play a pivotal role in immunoglobulin-G-mediated but not immunoglobulin-E-mediated systemic anaphylaxis.

[1]  F. Finkelman.  Anaphylaxis: lessons from mouse models. , 2007, The Journal of allergy and clinical immunology.

[2]  H. Karasuyama,et al.  Basophils are essential initiators of a novel type of chronic allergic inflammation. , 2007, Blood.

[3]  Zhiqian Yu,et al.  Induction of serum IL‐18 with Propionibacterium acnes and lipopolysaccharide in phagocytic macrophage‐inactivated mice , 2007, Journal of leukocyte biology.

[4]  F. Simons,et al.  Risk assessment in anaphylaxis: current and future approaches. , 2007, The Journal of allergy and clinical immunology.

[5]  J. Kinet,et al.  New developments in FcεRI regulation, function and inhibition , 2007, Nature Reviews Immunology.

[6]  朱丽琳,et al.  Anaphylaxis , 2006 .

[7]  F. Finkelman,et al.  IgG-blocking antibodies inhibit IgE-mediated anaphylaxis in vivo through both antigen interception and FcγRIIb cross-linking , 2006 .

[8]  D. Hepner,et al.  Second symposium on the definition and management of anaphylaxis: summary report--Second National Institute of Allergy and Infectious Disease/Food Allergy and Anaphylaxis Network symposium. , 2006, The Journal of allergy and clinical immunology.

[9]  C. Chen,et al.  Mast cell-deficient W-sash c-kit mutant Kit W-sh/W-sh mice as a model for investigating mast cell biology in vivo. , 2005, The American journal of pathology.

[10]  K. Hirokawa,et al.  Basophils play a critical role in the development of IgE-mediated chronic allergic inflammation independently of T cells and mast cells. , 2005, Immunity.

[11]  S. Galli.  Pathogenesis and management of anaphylaxis: current status and future challenges. , 2005, The Journal of allergy and clinical immunology.

[12]  W. Paul,et al.  Basophils Produce IL-4 and Accumulate in Tissues after Infection with a Th2-inducing Parasite , 2004, The Journal of experimental medicine.

[13]  D. Voehringer,et al.  Type 2 immunity reflects orchestrated recruitment of cells committed to IL-4 production. , 2004, Immunity.

[14]  A. Guttormsen,et al.  Screening for mast cell tryptase and serum IgE antibodies in 18 patients with anaphylactic shock during general anaesthesia , 2003, Acta anaesthesiologica Scandinavica.

[15]  T. Kuijpers,et al.  Regulation and kinetics of platelet‐activating factor and leukotriene C4 synthesis by activated human basophils , 2003, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[16]  S. Plevy,et al.  The Incidence and Management of Infusion Reactions to Infliximab: A Large Center Experience , 2003, American Journal of Gastroenterology.

[17]  Calman Prussin,et al.  IgE, mast cells, basophils, and eosinophils. , 2003, The Journal of allergy and clinical immunology.

[18]  R. Lockey,et al.  Anaphylaxis: a review of causes and mechanisms. , 2002, The Journal of allergy and clinical immunology.

[19]  P. Lieberman,et al.  Histamine and antihistamines in anaphylaxis. , 2002, Clinical allergy and immunology.

[20]  L. Schwartz.  Mast cells and basophils. , 2002, Clinical allergy and immunology.

[21]  F. Finkelman,et al.  Pathways of anaphylaxis in the mouse. , 2002, The Journal of allergy and clinical immunology.

[22]  Franco H Falcone,et al.  The human basophil: a new appreciation of its role in immune responses. , 2000, Blood.

[23]  H. Shindou,et al.  Roles of cytosolic phospholipase A(2) and platelet-activating factor receptor in the Ca-induced biosynthesis of PAF. , 2000, Biochemical and biophysical research communications.

[24]  R. Coffman,et al.  Cutting edge: mouse IgG1 antibodies comprise two functionally distinct types that are differentially regulated by IL-4 and IL-12. , 1999, Journal of immunology.

[25]  Jae Seung Park,et al.  Immunoglobulin E–dependent Active Fatal Anaphylaxis in Mast Cell–deficient Mice , 1998, The Journal of experimental medicine.

[26]  J. Park,et al.  Anti-IL-4 monoclonal antibody prevents antibiotics-induced active fatal anaphylaxis. , 1997, Journal of immunology.

[27]  S. Galli,et al.  Systemic anaphylaxis in the mouse can be mediated largely through IgG1 and Fc gammaRIII. Assessment of the cardiopulmonary changes, mast cell degranulation, and death associated with active or IgE- or IgG1-dependent passive anaphylaxis. , 1997, The Journal of clinical investigation.

[28]  S. Galli,et al.  Absence of Fc epsilonRI alpha chain results in upregulation of Fc gammaRIII-dependent mast cell degranulation and anaphylaxis. Evidence of competition between Fc epsilonRI and Fc gammaRIII for limiting amounts of FcR beta and gamma chains. , 1997, The Journal of clinical investigation.

[29]  R. Schmidt,et al.  Impaired IgG-Dependent Anaphylaxis and Arthus Reaction in FcγRIII (CD16) Deficient Mice , 1996 .

[30]  K. Kumagai,et al.  Effects of macrophage depletion on the induction of histidine decarboxylase by lipopolysaccharide, interleukin 1 and tumour necrosis factor , 1995, British journal of pharmacology.

[31]  N. Van Rooijen,et al.  Liposome mediated depletion of macrophages: mechanism of action, preparation of liposomes and applications. , 1994, Journal of immunological methods.

[32]  P. Leder,et al.  Active anaphylaxis in IgE-deficient mice , 1994, Nature.

[33]  C. Hirshman,et al.  High-titer protamine-specific IgG antibody associated with anaphylaxis: report of a case and quantitative analysis of antibody in vasectomized men. , 1993, Anesthesiology.

[34]  A. Arimura,et al.  Active and passive cutaneous anaphylaxis in WBB6F1 mouse, a mast cell-deficient strain. , 1990, Immunological investigations.

[35]  C. Hirshman,et al.  Association of protamine IgE and IgG antibodies with life-threatening reactions to intravenous protamine. , 1989, The New England journal of medicine.

[36]  P. Braquet,et al.  Human endothelial cells are target for platelet-activating factor. I. Platelet-activating factor induces changes in cytoskeleton structures. , 1987, Journal of immunology.

[37]  P. Cammarata,et al.  Anaphylaxis in mast cell-deficient mice. , 1984, The Journal of investigative dermatology.

[38]  M. Devey,et al.  Immunoglobulin Class and Subclass Distribution of Dextran‐Reactive Antibodies in Human Reactors and Non Reactors to Clinical Dextran , 1982, Allergy.

[39]  L. McManus,et al.  Vasoactive properties of acetyl glyceryl ether phosphorylcholine and analogues. , 1982, Laboratory investigation; a journal of technical methods and pathology.

[40]  Y. Kitamura,et al.  Decrease of mast cells in W/Wv mice and their increase by bone marrow transplantation. , 1978, Blood.

[41]  J. Benveniste,et al.  Platelet-activating factor, a new mediator of anaphylaxis and immune complex deposition from rabbit and human basophils , 1974, Nature.

[42]  W. J. Dyer,et al.  A rapid method of total lipid extraction and purification. , 1959, Canadian journal of biochemistry and physiology.

[43]  Michael Schwenk,et al.  Anaphylaxis , 1919, The Indian medical gazette.

[44]  P. Kam,et al.  : 4 , 1898, You Can Cross the Massacre on Foot.

[45]  小島 利之.  Mast cells and basophils are selectively activated in vitro and in vivo through CD200R3 in an IgE-independent manner , 2008 .

[46]  H. Brühl,et al.  Identification of Antigen-Capturing Cells as Basophils , 2004 .

[47]  D. Metcalfe,et al.  4. IgE, mast cells, basophils, and eosinophils. , 2003, The Journal of allergy and clinical immunology.

[48]  R. Schmidt,et al.  Impaired IgG-dependent anaphylaxis and Arthus reaction in Fc gamma RIII (CD16) deficient mice. , 1996, Immunity.

[49]  L. Lichtenstein,et al.  CURRENT CONCEPTS : ANAPHYLAXIS , 1991 .

[50]  F. Snyder.  Platelet-Activating Factor and Related Lipid Mediators , 1987, Springer US.

[51]  G. Köhler,et al.  Biological activities of antitrinitrophenyl and antidinitrophenyl mouse monoclonal antibodies. , 1982, Proceedings of the National Academy of Sciences of the United States of America.

[52]  M. Wabl,et al.  Thirteen hybridomas secreting hapten‐specific immunoglobulin E from mice with Iga or Igb heavy chain haplotype , 1981, European journal of immunology.

[53]  박귀태,et al.  A Type 2 Diabetes–Associated Functional Regulatory Variant in a Pancreatic Islet Enhancer at the ADCY5 Locus , 2017, Diabetes.