Intercellular coupling through gap junctions masks M cells in the human heart.

[1]  Y. Rudy,et al.  Ionic Current Basis of Electrocardiographic Waveforms: A Model Study , 2002, Circulation research.

[2]  Stanley Nattel,et al.  Cellular electrophysiology of atrial fibrillation. , 2002, Cardiovascular research.

[3]  Jacques M T de Bakker,et al.  Is the apico-basal gradient larger than the transmural gradient? , 2002, Journal of cardiovascular pharmacology.

[4]  Akira Abe,et al.  Correlation between the effective refractory period and activation-recovery interval calculated from the intracardiac unipolar electrogram of humans with and without dl-sotalol treatment. , 2001, Japanese circulation journal.

[5]  P Taggart,et al.  Transmural repolarisation in the left ventricle in humans during normoxia and ischaemia. , 2001, Cardiovascular research.

[6]  C. Antzelevitch.  Transmural dispersion of repolarization and the T wave. , 2001, Cardiovascular research.

[7]  R Wilders,et al.  Gap junctions in cardiovascular disease. , 2000, Circulation research.

[8]  P. Taggart,et al.  Inhomogeneous transmural conduction during early ischaemia in patients with coronary artery disease. , 2000, Journal of molecular and cellular cardiology.

[9]  U. Gerlach,et al.  Rate- and site-dependent effects of propafenone, dofetilide, and the new I(Ks)-blocking agent chromanol 293b on individual muscle layers of the intact canine heart. , 1999, Circulation.

[10]  CHARLES ANTZELEVITCH,et al.  The M Cell: , 1999, Journal of cardiovascular electrophysiology.

[11]  K. Kamiya,et al.  Heterogeneous distribution of the two components of delayed rectifier K+ current: a potential mechanism of the proarrhythmic effects of methanesulfonanilideclass III agents. , 1999, Cardiovascular research.

[12]  P. C. Viswanathan,et al.  Effects of IKr and IKs heterogeneity on action potential duration and its rate dependence: a simulation study. , 1999, Circulation.

[13]  G. Tomaselli,et al.  Electrophysiological remodeling in hypertrophy and heart failure. , 1999, Cardiovascular research.

[14]  P. Boyden,et al.  Electrical remodeling in ischemia and infarction. , 1999, Cardiovascular research.

[15]  M. Rosen,et al.  The Controversial M Cell , 1999, Journal of cardiovascular electrophysiology.

[16]  H. Wellens,et al.  Repolarizing K+ currents ITO1 and IKs are larger in right than left canine ventricular midmyocardium. , 1999, Circulation.

[17]  C Antzelevitch,et al.  Cellular basis for the normal T wave and the electrocardiographic manifestations of the long-QT syndrome. , 1998, Circulation.

[18]  C. Antzelevitch,et al.  Characteristics and distribution of M cells in arterially perfused canine left ventricular wedge preparations. , 1998, Circulation.

[19]  M. Carrier,et al.  Transmural heterogeneity of action potentials and I to1 in myocytes isolated from the human right ventricle. , 1998, American journal of physiology. Heart and circulatory physiology.

[20]  J. Cook,et al.  EXPRESSION OF MAJOR GAP JUNCTION CONNEXIN TYPES IN THE WORKING MYOCARDIUM OF EIGHT CHORDATES , 1998, Cell biology international.

[21]  D. Beuckelmann,et al.  Simulation study of cellular electric properties in heart failure. , 1998, Circulation research.

[22]  M. Rosen,et al.  Regional differences in electrophysiological properties of epicardium, midmyocardium, and endocardium. In vitro and in vivo correlations. , 1996, Circulation.

[23]  F. Charpentier,et al.  Electrophysiologic characteristics of cells spanning the left ventricular wall of human heart: evidence for presence of M cells. , 1995, Journal of the American College of Cardiology.

[24]  C. Antzelevitch,et al.  Characteristics of the delayed rectifier current (IKr and IKs) in canine ventricular epicardial, midmyocardial, and endocardial myocytes. A weaker IKs contributes to the longer action potential of the M cell. , 1995, Circulation research.

[25]  C. Luo,et al.  A dynamic model of the cardiac ventricular action potential. I. Simulations of ionic currents and concentration changes. , 1994, Circulation research.

[26]  S Rees,et al.  Proarrhythmic effects of class III antiarrhythmic agents. , 1993, Cardiovascular research.

[27]  C Antzelevitch,et al.  Ionic bases for electrophysiological distinctions among epicardial, midmyocardial, and endocardial myocytes from the free wall of the canine left ventricle. , 1993, Circulation research.

[28]  C Antzelevitch,et al.  A subpopulation of cells with unique electrophysiological properties in the deep subepicardium of the canine ventricle. The M cell. , 1991, Circulation research.

[29]  A. Wilde,et al.  Ventricular tachycardia in the infarcted, Langendorff-perfused human heart: role of the arrangement of surviving cardiac fibers. , 1990, Journal of the American College of Cardiology.

[30]  W. Loewenstein,et al.  Junctional intercellular communication: the cell-to-cell membrane channel. , 1981, Physiological reviews.

[31]  A E Becker,et al.  Left ventricular fibre architecture in man. , 1981, British heart journal.

[32]  D. D. Streeter,et al.  Engineering Mechanics for Successive States in Canine Left Ventricular Myocardium: II. Fiber Angle and Sarcomere Length , 1973, Circulation research.

[33]  Hans Michael Piper,et al.  Cardiovascular Research , 1966, British medical bulletin.

[34]  P. Taggart,et al.  The transmural gradient of action potential duration in the vivo human heart and in model simulations: is there a contribution of M cells ? , 2002 .

[35]  M. Näbauer.  Electrical heterogeneity in the ventricular wall--and the M cell. , 1998, Cardiovascular research.

[36]  G. Steinbeck,et al.  Regional differences in current density and rate-dependent properties of the transient outward current in subepicardial and subendocardial myocytes of human left ventricle. , 1996, Circulation.

[37]  R. Lux,et al.  Correlation between in vivo transmembrane action potential durations and activation-recovery intervals from electrograms. Effects of interventions that alter repolarization time. , 1990, Circulation.