Effect of verapamil on infarct size in dogs subjected to coronary artery occlusion with transient reperfusion.

[1]  W. Ganz,et al.  The thrombolysis in myocardial infarction (TIMI) trial. , 1985, The New England journal of medicine.

[2]  E. Braunwald,et al.  Effect of intracoronary verapamil on infarct size in the ischemic, reperfused canine heart: critical importance of the timing of treatment. , 1985, The American journal of cardiology.

[3]  Reimer Ka,et al.  Verapamil in two reperfusion models of myocardial infarction. Temporary protection of severely ischemic myocardium without limitation of ultimate infarct size. , 1984 .

[4]  B E Sobel,et al.  Coronary thrombolysis with recombinant human tissue-type plasminogen activator: a prospective, randomized, placebo-controlled trial. , 1984, Circulation.

[5]  E. Braunwald,et al.  Preservation of high-energy phosphates by verapamil in reperfused myocardium. , 1984, Circulation.

[6]  E. Braunwald,et al.  Thrombolytic therapy. A new strategy for the treatment of acute myocardial infarction (1). , 1984, The New England journal of medicine.

[7]  R. Kloner,et al.  The effect of streptokinase on intramyocardial hemorrhage, infarct size, and the no-reflow phenomenon during coronary reperfusion. , 1984, Circulation.

[8]  A. Ross,et al.  A randomized, angiographically controlled trial of intracoronary streptokinase in acute myocardial infarction. , 1984, The American journal of cardiology.

[9]  J L Ritchie,et al.  Western Washington randomized trial of intracoronary streptokinase in acute myocardial infarction. , 1983, The New England journal of medicine.

[10]  E. Braunwald,et al.  Studies of experimental coronary artery reperfusion. Effects on infarct size, myocardial function, biochemistry, ultrastructure and microvascular damage. , 1983, Circulation.

[11]  I. Palacios,et al.  Coronary reocclusion after selective administration of streptokinase. , 1983, Circulation.

[12]  W. O’Neill,et al.  Intracoronary fibrinolytic therapy in acute myocardial infarction. Report of a prospective randomized trial. , 1983, The New England journal of medicine.

[13]  P. R. Frederick,et al.  A randomized trial of intracoronary streptokinase in the treatment of acute myocardial infarction. , 1983, The New England journal of medicine.

[14]  E. Braunwald,et al.  Time course of functional and biochemical recovery of myocardium salvaged by reperfusion. , 1983, Journal of the American College of Cardiology.

[15]  A. DeMaria,et al.  Importance of follow-up medical and surgical approaches to prevent reinfarction, reocclusion, and recurrent angina following intracoronary thrombolysis with streptokinase in acute myocardial infarction. , 1982, American heart journal.

[16]  M. Fishbein,et al.  Early phase acute myocardial infarct size quantification: validation of the triphenyl tetrazolium chloride tissue enzyme staining technique. , 1981, American heart journal.

[17]  Y. Ikeda,et al.  Inhibition of Human Platelet Functions by Verapamil , 1981, Thrombosis and Haemostasis.

[18]  R. Jennings,et al.  Prolonged depletion of ATP and of the adenine nucleotide pool due to delayed resynthesis of adenine nucleotides following reversible myocardial ischemic injury in dogs. , 1981, Journal of molecular and cellular cardiology.

[19]  W. Boden,et al.  The Effect of Verapamil on Mechanical Performance of Acutely Ischemic and Reperfused Myocardium in the Conscious Dog , 1981, Circulation research.

[20]  E. Antman,et al.  Calcium channel blocking agents in the treatment of cardiovascular disorders. Part II: Hemodynamic effects and clinical applications. , 1980, Annals of internal medicine.

[21]  E. Antman,et al.  Calcium channel blocking agents in the treatment of cardiovascular disorders. Part I: Basic and clinical electrophysiologic effects. , 1980, Annals of internal medicine.

[22]  E. Braunwald,et al.  Autoradiographic method for measuring the ischemic myocardium at risk: effects of verapamil on infarct size aftr experimental coronary artery occlusion. , 1980, Proceedings of the National Academy of Sciences of the United States of America.

[23]  E. Braunwald,et al.  Prolonged derangements of canine myocardial purine metabolism after a brief coronary artery occlusion not associated with anatomic evidence of necrosis. , 1980, Proceedings of the National Academy of Sciences of the United States of America.

[24]  S. Vatner.  Correlation between Acute Reductions in Myocardial Blood Flow and Function in Conscious Dogs , 1980, Circulation research.

[25]  J. Fleiss,et al.  Some Statistical Methods Useful in Circulation Research , 1980, Circulation research.

[26]  E. Braunwald,et al.  Review: Observations on experimental myocardial ischaemia , 1980 .

[27]  L. Décourt,et al.  Effect of verapamil on regional coronary and myocardial perfusion during acute coronary occlusion. , 1980, The American journal of cardiology.

[28]  J. Lowe,et al.  The Wavefront Phenomenon of Ischemic Cell Death: 1. Myocardial Infarct Size vs Duration of Coronary Occlusion in Dogs , 1977, Circulation.

[29]  J. Lowe,et al.  Effect of the Calcium Antagonist Verapamil on Necrosis following Temporary Coronary Artery Occlusion in Dogs , 1977, Circulation.

[30]  M. Noble,et al.  Total and Regional Coronary Blood Flow Measured by Radioactive Microspheres in Conscious and Anesthetized Dogs , 1969, Circulation research.

[31]  H. S. Mueller,et al.  The Thrombolysis in Myocardial Infarction (TIMI) trial. Phase I findings. , 1985, The New England journal of medicine.