Long-term synaptic facilitation in the absence of short-term facilitation in Aplysia neurons.
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[1] D. A. Baxter,et al. Involvement of protein kinase C in serotonin-induced spike broadening and synaptic facilitation in sensorimotor connections of Aplysia. , 1992, Journal of neurophysiology.
[2] D. A. Baxter,et al. Differential effects of cAMP and serotonin on membrane current, action-potential duration, and excitability in somata of pleural sensory neurons of Aplysia. , 1990, Journal of neurophysiology.
[3] J. Byrne,et al. Long-term sensitization in Aplysia: biophysical correlates in tail sensory neurons. , 1987, Science.
[4] J. Byrne,et al. Associative conditioning analog selectively increases cAMP levels of tail sensory neurons in Aplysia. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[5] L. Squire,et al. The structure and organization of memory. , 1993, Annual review of psychology.
[6] D. A. Baxter,et al. Serotonergic modulation of two potassium currents in the pleural sensory neurons of Aplysia. , 1989, Journal of neurophysiology.
[7] T. Abrams,et al. cAMP modulates multiple K+ currents, increasing spike duration and excitability in Aplysia sensory neurons. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[8] E. Kandel,et al. Roles of PKA and PKC in facilitation of evoked and spontaneous transmitter release at depressed and nondepressed synapses in aplysia sensory neurons , 1992, Neuron.
[9] T. Crow,et al. Down-regulation of protein kinase C and kinase inhibitors dissociate short- and long-term enhancement produced by one-trial conditioning of Hermissenda. , 1993, Journal of neurophysiology.
[10] J. Byrne,et al. Intracellular injection of cAMP induces a long-term reduction of neuronal K+ currents. , 1988, Science.
[11] E R Kandel,et al. A critical period for macromolecular synthesis in long-term heterosynaptic facilitation in Aplysia. , 1986, Science.
[12] Eric R. Kandel,et al. Injection of the cAMP-responsive element into the nucleus of Aplysia sensory neurons blocks long-term facilitation , 1990, Nature.
[13] M. Hammer,et al. Serotonin acts in the synaptic region of sensory neruons in Aplysia to enhance transmitter release , 1989, Neuroscience Letters.
[14] E. Kandel,et al. Mechanoafferent neurons innervating tail of Aplysia. II. Modulation by sensitizing stimulation. , 1983, Journal of neurophysiology.
[15] E R Kandel,et al. Modulation of a transient K+ current in the pleural sensory neurons of Aplysia by serotonin and cAMP: implications for spike broadening. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[16] J. Byrne,et al. Long-term synaptic changes produced by a cellular analog of classical conditioning in Aplysia. , 1990, Science.
[17] C. H. Bailey,et al. Morphological Aspects of Synaptic Plasticity in Aplysia An Anatomical Substrate for Long‐Term Memory a , 1991, Annals of the New York Academy of Sciences.
[18] A. Baddeley,et al. Amnesia and the distinction between long- and short-term memory. , 1970 .
[19] J. Byrne,et al. Serotoninergic varicosities make synaptic contacts with pleural sensory neurons of Aplysia , 1991, The Journal of comparative neurology.
[20] J. Byrne,et al. cAMP induces long-term morphological changes in sensory neurons ofAplysia , 1991, Brain Research.
[21] E. Kandel,et al. cAMP evokes long-term facilitation in Aplysia sensory neurons that requires new protein synthesis. , 1988, Science.
[22] T. Carew,et al. Pharmacological dissociation of modulatory effects of serotonin in Aplysia sensory neurons. , 1991, Science.