An important role for Cholecystokinin, a CLOCK target gene, in the development and treatment of manic-like behaviors
暂无分享,去创建一个
C. McClung | C. Tamminga | S. Ghose | A. Ozburn | R. Arey | J. Enwright | E. Falcón | S. Spencer | C. Mcclung
[1] D. Kupfer,et al. Advances in bipolar disorder: selected sessions from the 2011 International Conference on Bipolar Disorder , 2011, Annals of the New York Academy of Sciences.
[2] J. Quevedo,et al. Behavioral and neurochemical effects of sodium butyrate in an animal model of mania , 2011, Behavioural pharmacology.
[3] L. Mahadevan,et al. Dynamic acetylation of all lysine-4 trimethylated histone H3 is evolutionarily conserved and mediated by p300/CBP , 2011, Proceedings of the National Academy of Sciences.
[4] A. Harvey. Sleep and circadian functioning: critical mechanisms in the mood disorders? , 2011, Annual review of clinical psychology.
[5] Michelle M Sidor,et al. Specific Role of VTA Dopamine Neuronal Firing Rates and Morphology in the Reversal of Anxiety-Related, but not Depression-Related Behavior in the ClockΔ19 Mouse Model of Mania , 2011, Neuropsychopharmacology.
[6] P. Thibault,et al. H3 Lysine 4 Is Acetylated at Active Gene Promoters and Is Regulated by H3 Lysine 4 Methylation , 2011, PLoS genetics.
[7] Paolo Sassone-Corsi,et al. The histone methyltransferase MLL1 permits the oscillation of circadian gene expression , 2010, Nature Structural &Molecular Biology.
[8] Laurent Coque,et al. Knockdown of Clock in the Ventral Tegmental Area Through RNA Interference Results in a Mixed State of Mania and Depression-Like Behavior , 2010, Biological Psychiatry.
[9] M. Schalling,et al. CRY2 Is Associated with Rapid Cycling in Bipolar Disorder Patients , 2010, PloS one.
[10] C. McClung,et al. ΔFosB indirectly regulates Cck promoter activity , 2010, Brain Research.
[11] S. Rotzinger,et al. Behavioral effects of neuropeptides in rodent models of depression and anxiety , 2010, Peptides.
[12] M. Gratacós,et al. Differential Association of Circadian Genes with Mood Disorders: CRY1 and NPAS2 are Associated with Unipolar Major Depression and CLOCK and VIP with Bipolar Disorder , 2010, Neuropsychopharmacology.
[13] M. Berk,et al. Is Lithium in a Class of Its Own? A Brief Profile of Its Clinical Use , 2009, The Australian and New Zealand journal of psychiatry.
[14] Matcheri Keshavan,et al. Association study of 21 circadian genes with bipolar I disorder, schizoaffective disorder, and schizophrenia. , 2009, Bipolar disorders.
[15] C. Nievergelt,et al. Circadian polymorphisms associated with affective disorders , 2009, Journal of circadian rhythms.
[16] J. Kleinman,et al. Metabotropic Glutamate Receptor 2 and 3 Gene Expression in The Human Prefrontal Cortex and Mesencephalon in Schizophrenia , 2008, The International journal of neuroscience.
[17] Diane B. Boivin,et al. The role of circadian clock genes in mental disorders , 2007, Dialogues in clinical neuroscience.
[18] Enrico Smeraldi,et al. Actimetric evidence that CLOCK 3111 T/C SNP influences sleep and activity patterns in patients affected by bipolar depression , 2007, American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics.
[19] C. McClung,et al. Circadian genes, rhythms and the biology of mood disorders. , 2007, Pharmacology & therapeutics.
[20] Kole T. Roybal,et al. Mania-like behavior induced by disruption of CLOCK , 2007, Proceedings of the National Academy of Sciences.
[21] Wen-Ning Zhao,et al. CIPC is a mammalian circadian clock protein without invertebrate homologues , 2007, Nature Cell Biology.
[22] Carol A. Tamminga,et al. Human postmortem tissue: What quality markers matter? , 2006, Brain Research.
[23] Paolo Sassone-Corsi,et al. Circadian Regulator CLOCK Is a Histone Acetyltransferase , 2006, Cell.
[24] C. Nievergelt,et al. Suggestive evidence for association of the circadian genes PERIOD3 and ARNTL with bipolar disorder , 2006, American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics.
[25] E. Nestler,et al. Sustained hippocampal chromatin regulation in a mouse model of depression and antidepressant action , 2006, Nature Neuroscience.
[26] D. Kupfer,et al. Association study of eight circadian genes with bipolar I disorder, schizoaffective disorder and schizophrenia , 2006, Genes, brain, and behavior.
[27] Cyrus Martin,et al. The diverse functions of histone lysine methylation , 2005, Nature Reviews Molecular Cell Biology.
[28] J. Takahashi,et al. Regulation of dopaminergic transmission and cocaine reward by the Clock gene. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[29] E. Nestler,et al. Histone Modifications at Gene Promoter Regions in Rat Hippocampus after Acute and Chronic Electroconvulsive Seizures , 2004, The Journal of Neuroscience.
[30] J. Burgunder,et al. Circadian patterns of neurotransmitter related gene expression in motor regions of the rat brain , 2004, Neuroscience Letters.
[31] A. Serretti,et al. Genetic dissection of psychopathological symptoms: Insomnia in mood disorders and CLOCK gene polymorphism , 2003 .
[32] V. Wiegant,et al. Regulation of cholecystokinin release from central nerve terminals , 2001, Peptides.
[33] T. Hansen. Cholecystokinin gene transcription: promoter elements, transcription factors and signaling pathways , 2001, Peptides.
[34] F. Vaccarino,et al. Cholecystokinergic Innervation of Nucleus Accumbens Subregions , 1998, Peptides.
[35] D. P. King,et al. The mouse Clock mutation behaves as an antimorph and maps within the W19H deletion, distal of Kit. , 1997, Genetics.
[36] J. Bellach,et al. Circadian rhythms of dopamine and cholecystokinin in nucleus accumbens and striatum of rats--influence on dopaminergic stimulation. , 1995, Chronobiology international.
[37] B. Roques,et al. The CCKB receptor antagonist, L-365,260, elicits antidepressant-type effects in the forced-swim test in mice. , 1994, European journal of pharmacology.
[38] D. P. King,et al. Mutagenesis and mapping of a mouse gene, Clock, essential for circadian behavior. , 1994, Science.
[39] S. Kito,et al. Cholecystokinin increases intracellular Ca2+ concentration in cultured striatal neurons , 1991, Neuropeptides.
[40] B. Costall,et al. Development of a class of selective cholecystokinin type B receptor antagonists having potent anxiolytic activity. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[41] Rex Y. Wang,et al. In vivo release of dopamine in the nucleus accumbens of the rat: modulation by cholecystokinin , 1984, Brain Research.
[42] T. Hökfelt,et al. Evidence for coexistence of dopamine and CCK in meso-limbic neurones , 1980, Nature.
[43] Ethan D Buhr,et al. Molecular components of the Mammalian circadian clock. , 2013, Handbook of experimental pharmacology.
[44] Nicolas Cermakian,et al. Circadian rhythms and clock genes in psychotic disorders. , 2010, The Israel journal of psychiatry and related sciences.
[45] J. Aubry,et al. Lithium: Updated Human Knowledge Using an Evidence-Based Approach , 2009, CNS drugs.
[46] V. J. Polidora. Behavioral Effects of , 1967 .