NF-κB activity is required for anagen maintenance in human hair follicles in vitro.

[1]  M. Plikus New activators and inhibitors in the hair cycle clock: targeting stem cells' state of competence. , 2012, The Journal of investigative dermatology.

[2]  P. Schneider,et al.  Identification of ectodysplasin target genes reveals the involvement of chemokines in hair development. , 2012, The Journal of investigative dermatology.

[3]  Claus Scheidereit,et al.  It takes two to tango: IκBs, the multifunctional partners of NF‐κB , 2012, Immunological reviews.

[4]  M. Pasparakis Role of NF‐κB in epithelial biology , 2012, Immunological reviews.

[5]  S. Ghosh,et al.  Celebrating 25 years of NF‐κB research , 2012, Immunological reviews.

[6]  J. Haycock,et al.  Human hair follicle dermal cells and skin fibroblasts show differential activation of NF‐κB in response to pro‐inflammatory challenge , 2012, Experimental dermatology.

[7]  P. Maini,et al.  Self-Organizing and Stochastic Behaviors During the Regeneration of Hair Stem Cells , 2011, Science.

[8]  B. Andersen,et al.  Clock genes, hair growth and aging , 2010, Aging.

[9]  Boris Jerchow,et al.  Reciprocal requirements for EDA/EDAR/NF-kappaB and Wnt/beta-catenin signaling pathways in hair follicle induction. , 2009, Developmental cell.

[10]  Ralf Paus,et al.  The Hair Follicle as a Dynamic Miniorgan , 2009, Current Biology.

[11]  J. Tobias,et al.  Activation of β-catenin signaling programs embryonic epidermis to hair follicle fate , 2008, Development.

[12]  S. Ghosh,et al.  Shared Principles in NF-κB Signaling , 2008, Cell.

[13]  Elaine Fuchs,et al.  Scratching the surface of skin development , 2007, Nature.

[14]  R Paus,et al.  The biology of hair follicles. , 1999, The New England journal of medicine.

[15]  J. Zonana,et al.  Mutations in the human homologue of mouse dl cause autosomal recessive and dominant hypohidrotic ectodermal dysplasia , 1999, Nature Genetics.