Eosinophilic Fasciitis: Increased Collagen Production and Type I Procollagen Messenger RNA Levels in Fibroblasts Cultured From Involved Skin
暂无分享,去创建一个
L. Niskanen | J. Heino | J. Uitto | V. Kähäri | J. Fräki | Jorma E. Fräki | Jyrki Heino | Jouni Uitto | Leo Niskanen
[1] K. Sayama,et al. Morphea Profunda , 1991, International journal of dermatology.
[2] S. Jimenez,et al. Evaluation of transforming growth factor beta and type I procollagen gene expression in fibrotic skin diseases by in situ hybridization. , 1990, The Journal of investigative dermatology.
[3] S. Jimenez,et al. Development of diffuse fasciitis with eosinophilia during L-tryptophan treatment: demonstration of elevated type I collagen gene expression in affected tissues. A clinicopathologic study of four patients. , 1990, Annals of internal medicine.
[4] E. Vuorio,et al. Identification of fibroblasts responsible for increased collagen production in localized scleroderma by in situ hybridization. , 1988, The Journal of investigative dermatology.
[5] T. Krieg,et al. Localization of collagen mRNA in normal and scleroderma skin by in‐situ hybridization , 1988, European journal of clinical investigation.
[6] J. Seyer,et al. Modulation of fibroblast functions by interleukin 1: increased steady- state accumulation of type I procollagen messenger RNAs and stimulation of other functions but not chemotaxis by human recombinant interleukin 1 alpha and beta , 1988, The Journal of cell biology.
[7] S. Krane,et al. Modulation by recombinant interleukin 1 of synthesis of types I and III collagens and associated procollagen mRNA levels in cultured human cells. , 1987, The Journal of biological chemistry.
[8] J. Heino,et al. Interleukin-1 increases collagen production and mRNA levels in cultured skin fibroblasts. , 1987, Biochimica et biophysica acta.
[9] E. Bauer,et al. Regulation of collagen gene expression in cutaneous diseases with dermal fibrosis: evidence for pretranslational control. , 1987, The Journal of investigative dermatology.
[10] J. Massagué,et al. Regulation of fibronectin and type I collagen mRNA levels by transforming growth factor-beta. , 1987, The Journal of biological chemistry.
[11] E. Vuorio,et al. Elevated proα2(I) collagen mRNA levels in cultured scleroderma fibroblasts result from an increased transcription rate of the corresponding gene , 1987, FEBS letters.
[12] E. Vuorio,et al. Coordinated regulation of type I and type III collagen production and mRNA levels of pro alpha 1(I) and pro alpha 2(I) collagen in cultured morphea fibroblasts. , 1987, Archives of dermatological research.
[13] J. Rosenbloom,et al. Co-ordinate increase in the expression of type I and type III collagen genes in progressive systemic sclerosis fibroblasts. , 1986, The Biochemical journal.
[14] M. Sporn,et al. Transforming growth factor type beta: rapid induction of fibrosis and angiogenesis in vivo and stimulation of collagen formation in vitro. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[15] Shulman Le. Diffuse fasciitis with hypergammaglobulinemia and eosinophilia: a new syndrome? , 1984 .
[16] Doyle Ja. Eosinophilic fasciitis: extracutaneous manifestations and associations , 1984 .
[17] E. Vuorio,et al. Increased type I collagen mRNA levels in cultured scleroderma fibroblasts. , 1984, Biochimica et biophysica acta.
[18] A. Kornblihtt,et al. Isolation and characterization of cDNA clones for human and bovine fibronectins. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[19] R. Winkelmann,et al. Cutaneous and subcutaneous inflammatory sclerosis syndromes. , 1982, Archives of dermatology.
[20] R. Penttinen,et al. Enhanced fibroblast collagen production by a macrophage-derived factor (CEMF). , 1982, Biochemical and biophysical research communications.
[21] R. Moskowitz,et al. Eosinophilic fasciitis. Immunopathology, ultrastructure, literature review, and consideration of its pathogenesis and relation to scleroderma. , 1982, Archives of pathology & laboratory medicine.
[22] R. Timpl,et al. Variability in collagen and fibronectin synthesis by scleroderma fibroblasts in primary culture. , 1981, The Journal of investigative dermatology.
[23] P. Thomas,et al. Hybridization of denatured RNA and small DNA fragments transferred to nitrocellulose. , 1980, Proceedings of the National Academy of Sciences of the United States of America.
[24] W. Rutter,et al. Isolation of biologically active ribonucleic acid from sources enriched in ribonuclease. , 1979, Biochemistry.
[25] A. Eisen,et al. Scleroderma: increased biosynthesis of triple-helical type I and type III procollagens associated with unaltered expression of collagenase by skin fibroblasts in culture. , 1979, The Journal of clinical investigation.
[26] S. Wahl,et al. Lymphocyte-mediated activation of fibroblast proliferation and collagen production. , 1978, Journal of immunology.
[27] R. Fleischmajer,et al. Ultrastructure of cutaneous cellular infiltrates in scleroderma. , 1977, Archives of dermatology.
[28] J. R. Reeves,et al. Cellular infiltrates in scleroderma skin. , 1977, Arthritis and rheumatism.
[29] R. Johnson,et al. Lymphokine stimulation of collagen accumulation. , 1976, The Journal of clinical investigation.
[30] E. Leroy,et al. Increased collagen synthesis by scleroderma skin fibroblasts in vitro: a possible defect in the regulation or activation of the scleroderma fibroblast. , 1974, The Journal of clinical investigation.
[31] D. Prockop,et al. Modified procedure for the assay of H-3-or C-14-labeled hydroxyproline. , 1966, Analytical biochemistry.
[32] W. F. Lever,et al. Histopathology of the Skin , 1962 .