Induction of CTGF by TGF-beta1 in normal and radiation enteritis human smooth muscle cells: Smad/Rho balance and therapeutic perspectives.
暂无分享,去创建一个
J. Bourhis | C. Bourgier | J. Aigueperse | M. Vozenin-Brotons | Jalil Abdelali | V. Haydont | D. Mathé
[1] C. Stournaras,et al. LIM-kinase 2 and Cofilin Phosphorylation Mediate Actin Cytoskeleton Reorganization Induced by Transforming Growth Factor-β* , 2005, Journal of Biological Chemistry.
[2] J. Bourhis,et al. An unusual cause of upper gastrointestinal haemorrhage , 2005, Gut.
[3] M. Spiteri,et al. Connective tissue growth factor expression and induction by transforming growth factor-beta is abrogated by simvastatin via a Rho signaling mechanism. , 2004, American journal of physiology. Lung cellular and molecular physiology.
[4] J. Lefaix,et al. The radiation-induced fibroatrophic process: therapeutic perspective via the antioxidant pathway. , 2004, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.
[5] B. Fenton,et al. Effect of Administration of Lovastatin on the Development of Late Pulmonary Effects after Whole-Lung Irradiation in a Murine Model , 2004, Radiation research.
[6] J. Bourhis,et al. Gene Expression Profile in Human Late Radiation Enteritis Obtained by High-Density cDNA Array Hybridization , 2004, Radiation research.
[7] J. Overgaard,et al. Prediction of normal tissue radiosensitivity from polymorphisms in candidate genes. , 2003, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.
[8] M. Hauer-Jensen,et al. Bowel injury: current and evolving management strategies. , 2003, Seminars in radiation oncology.
[9] U. Malmqvist,et al. Stretch-induced contractile differentiation of vascular smooth muscle: sensitivity to actin polymerization inhibitors. , 2003, American journal of physiology. Cell physiology.
[10] C. Haie-meder,et al. Fibrogenic signals in patients with radiation enteritis are associated with increased connective tissue growth factor expression. , 2003, International journal of radiation oncology, biology, physics.
[11] D. Abraham,et al. Prostacyclin derivatives prevent the fibrotic response to TGFβ2 by inhibiting the Ras/MEK/ERK pathway , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[12] R. Goldschmeding,et al. Gene regulation of connective tissue growth factor: new targets for antifibrotic therapy? , 2002, Matrix biology : journal of the International Society for Matrix Biology.
[13] S. Sa,et al. CTGF expression in mesangial cells: involvement of SMADs, MAP kinase, and PKC. , 2002, Kidney international.
[14] E. Robertis,et al. Connective-tissue growth factor (CTGF) modulates cell signalling by BMP and TGF-β , 2002, Nature Cell Biology.
[15] D. Shegogue,et al. Role of p38 MAPK in transforming growth factor beta stimulation of collagen production by scleroderma and healthy dermal fibroblasts. , 2002, The Journal of investigative dermatology.
[16] S. Sebti,et al. Signaling events required for transforming growth factor-beta stimulation of connective tissue growth factor expression by cultured human lung fibroblasts. , 2001, Archives of biochemistry and biophysics.
[17] M. Goppelt‐Struebe,et al. Rho‐dependent inhibition of the induction of connective tissue growth factor (CTGF) by HMG CoA reductase inhibitors (statins) , 2001, British journal of pharmacology.
[18] S. Sa,et al. CTGF and SMADs, Maintenance of Scleroderma Phenotype Is Independent of SMAD Signaling* , 2001, The Journal of Biological Chemistry.
[19] M. Goppelt‐Struebe,et al. Induction of Connective Tissue Growth Factor by Activation of Heptahelical Receptors , 2000, The Journal of Biological Chemistry.
[20] J. Massagué,et al. TGFβ Signaling in Growth Control, Cancer, and Heritable Disorders , 2000, Cell.
[21] H. Moses,et al. Interdependent SMAD and JNK Signaling in Transforming Growth Factor-β-mediated Transcription* , 1999, The Journal of Biological Chemistry.
[22] T. Yamakawa,et al. Mechanotransduction of rat aortic vascular smooth muscle cells requires RhoA and intact actin filaments. , 1999, Circulation research.
[23] C. Phiel,et al. Identification of distinct molecular phenotypes in cultured gastrointestinal smooth muscle cells. , 1998, Gastroenterology.
[24] Gary R. Grotendorst,et al. A novel transforming growth factor beta response element controls the expression of the connective tissue growth factor gene. , 1996, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[25] M. Graham. Pathogenesis of Intestinal Strictures in Crohn's Disease—An Update , 1995, Inflammatory bowel diseases.
[26] M. Sporn,et al. Transforming growth factor type beta: rapid induction of fibrosis and angiogenesis in vivo and stimulation of collagen formation in vitro. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[27] Chul-woo Yang,et al. Pravastatin treatment attenuates interstitial inflammation and fibrosis in a rat model of chronic cyclosporine-induced nephropathy. , 2004, American journal of physiology. Renal physiology.
[28] A. Nicholson,et al. Regulation of TGF-β1-induced connective tissue growth factor expression in airway smooth muscle cells , 2004 .