CCR4+CD8+ T cells clonally expand to differentiated effectors in murine psoriasis and in human psoriatic arthritis
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F. Granucci | S. Volinia | P. Secchiero | G. Banfi | R. Guerrini | F. Mingozzi | F. Casciano | E. Marzola | E. Reali | Giulia Protti | L. Gornati | G. Montico
[1] D. Hartmann,et al. Lesional activation of Tc17 cells in Behçet disease and psoriasis supports HLA class I‐mediated autoimmune responses * , 2021, The British journal of dermatology.
[2] A. Balato,et al. Therapeutics targeting the IL-23 and IL-17 pathway in psoriasis , 2021, The Lancet.
[3] Raphael Gottardo,et al. Integrated analysis of multimodal single-cell data , 2020, Cell.
[4] Z. Kopecki,et al. Immunological Memory in Imiquimod-Induced Murine Model of Psoriasiform Dermatitis , 2020, International journal of molecular sciences.
[5] A. Petrovič,et al. Current knowledge on autoantigens and autoantibodies in psoriasis , 2020, Scandinavian journal of immunology.
[6] F. Granucci,et al. CCR4+ Skin-Tropic Phenotype as a Feature of Central Memory CD8+ T Cells in Healthy Subjects and Psoriasis Patients , 2020, Frontiers in Immunology.
[7] Matthew D. Young,et al. Single-cell sequencing reveals clonal expansions of pro-inflammatory synovial CD8 T cells expressing tissue-homing receptors in psoriatic arthritis , 2019, Nature Communications.
[8] Nicholas L. Bormann,et al. scRepertoire: An R-based toolkit for single-cell immune receptor analysis. , 2020, F1000Research.
[9] N. Frahm,et al. CXCR3 enables recruitment and site-specific bystander activation of memory CD8+ T cells , 2019, Nature Communications.
[10] Jia Liu,et al. Toll-Like Receptor 7 Activation Enhances CD8+ T Cell Effector Functions by Promoting Cellular Glycolysis , 2019, Front. Immunol..
[11] F. Granucci,et al. Increased frequency of activated CD8+ T cell effectors in patients with psoriatic arthritis , 2019, Scientific Reports.
[12] M. Betts,et al. Human CD4+CD103+ cutaneous resident memory T cells are found in the circulation of healthy individuals , 2019, Science Immunology.
[13] L. Eidsmo,et al. A skewed pool of resident T cells triggers psoriasis‐associated tissue responses in never‐lesional skin from patients with psoriasis , 2019, The Journal of allergy and clinical immunology.
[14] R. Satija,et al. Normalization and variance stabilization of single-cell RNA-seq data using regularized negative binomial regression , 2019, Genome Biology.
[15] A. Butte,et al. Reference-based analysis of lung single-cell sequencing reveals a transitional profibrotic macrophage , 2018, Nature Immunology.
[16] C. Conrad,et al. Psoriasis: Classical vs. Paradoxical. The Yin-Yang of TNF and Type I Interferon , 2018, Front. Immunol..
[17] Fan Zhang,et al. Fast, sensitive, and accurate integration of single cell data with Harmony , 2018, bioRxiv.
[18] J. Prinz. Human Leukocyte Antigen-Class I Alleles and the Autoreactive T Cell Response in Psoriasis Pathogenesis , 2018, Front. Immunol..
[19] M. Wittmann,et al. IL-36γ Is a Strong Inducer of IL-23 in Psoriatic Cells and Activates Angiogenesis , 2018, Front. Immunol..
[20] L. French,et al. TNF blockade induces a dysregulated type I interferon response without autoimmunity in paradoxical psoriasis , 2018, Nature Communications.
[21] F. Granucci,et al. Blood to skin recirculation of CD4+ memory T cells associates with cutaneous and systemic manifestations of psoriatic disease. , 2017, Clinical Immunology.
[22] F. Granucci,et al. A role for CCR5(+)CD4 T cells in cutaneous psoriasis and for CD103(+) CCR4(+) CD8 Teff cells in the associated systemic inflammation. , 2016, Journal of autoimmunity.
[23] S. Krebs,et al. Melanocyte antigen triggers autoimmunity in human psoriasis , 2015, The Journal of experimental medicine.
[24] J. Nolz. Molecular mechanisms of CD8+ T cell trafficking and localization , 2015, Cellular and Molecular Life Sciences.
[25] B. Marinari,et al. The antimicrobial peptide LL37 is a T-cell autoantigen in psoriasis , 2014, Nature Communications.
[26] E. Wagner,et al. Specific roles for dendritic cell subsets during initiation and progression of psoriasis , 2014, EMBO molecular medicine.
[27] M. Ståhle,et al. Epidermal Th22 and Tc17 Cells Form a Localized Disease Memory in Clinically Healed Psoriasis , 2014, The Journal of Immunology.
[28] G. Banfi,et al. Differential expression of interleukin‐2 by anti‐CD3‐stimulated peripheral blood mononuclear cells in patients with psoriatic arthritis and patients with cutaneous psoriasis , 2014, Clinical and experimental dermatology.
[29] S. Kaech,et al. Lung airway-surveilling CXCR3(hi) memory CD8(+) T cells are critical for protection against influenza A virus. , 2013, Immunity.
[30] H. Stunnenberg,et al. BLUEPRINT: mapping human blood cell epigenomes , 2013, Haematologica.
[31] Gianfranco Altomare,et al. Inhibition of CCR7/CCL19 axis in lesional skin is a critical event for clinical remission induced by TNF blockade in patients with psoriasis. , 2013, The American journal of pathology.
[32] B. Kleuser,et al. Sphingosine-1-phosphate exhibits anti-proliferative and anti-inflammatory effects in mouse models of psoriasis. , 2013, Journal of dermatological science.
[33] Thomas Gebhardt,et al. Memory T cell subsets, migration patterns, and tissue residence. , 2013, Annual review of immunology.
[34] S. Bromley,et al. Recirculating Memory T Cells Are a Unique Subset of CD4+ T Cells with a Distinct Phenotype and Migratory Pattern , 2013, The Journal of Immunology.
[35] S. Devaraj,et al. Inflammation, Atherosclerosis, and Psoriasis , 2013, Clinical Reviews in Allergy & Immunology.
[36] ENCODEConsortium,et al. An Integrated Encyclopedia of DNA Elements in the Human Genome , 2012, Nature.
[37] D. Herr,et al. FTY720 (fingolimod) efficacy in an animal model of multiple sclerosis requires astrocyte sphingosine 1-phosphate receptor 1 (S1P1) modulation , 2010, Proceedings of the National Academy of Sciences.
[38] J. Harty,et al. Repetitive antigen stimulation induces stepwise transcriptome diversification but preserves a core signature of memory CD8(+) T cell differentiation. , 2010, Immunity.
[39] D. Gladman,et al. Update on Biomarkers in Psoriatic Arthritis , 2010, Current rheumatology reports.
[40] L. Boon,et al. Imiquimod-Induced Psoriasis-Like Skin Inflammation in Mice Is Mediated via the IL-23/IL-17 Axis1 , 2009, The Journal of Immunology.
[41] R. Winchester,et al. Nucleotide Sequencing of Psoriatic Arthritis Tissue before and during Methotrexate Administration Reveals a Complex Inflammatory T Cell Infiltrate with Very Few Clones Exhibiting Features That Suggest They Drive the Inflammatory Process by Recognizing Autoantigens1 , 2004, The Journal of Immunology.
[42] K. Yoshikawa,et al. Induction of T‐cell‐mediated skin disease specific for antigen transgenically expressed in keratinocytes , 2003, European journal of immunology.
[43] S. Miller,et al. Epitope spreading in immune-mediated diseases: implications for immunotherapy , 2002, Nature Reviews Immunology.
[44] D. Nelson,et al. In Vivo Cross-Presentation of a Soluble Protein Antigen: Kinetics, Distribution, and Generation of Effector CTL Recognizing Dominant and Subdominant Epitopes1 , 2000, The Journal of Immunology.