Opioid receptors influence spinal cord electrical activity and edema formation following spinal cord injury: experimental observations using naloxone in the rat

[1]  G. Siggins,et al.  Voltage-dependent effects of opioid peptides on hippocampal CA3 pyramidal neurons in vitro , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[2]  Y. Olsson,et al.  Topical application of dynorphin-A antibodies reduces edema and cell changes in traumatised rat spinal cord , 1994, Regulatory Peptides.

[3]  T. Holford,et al.  Effects of timing of methylprednisolone or naloxone administration on recovery of segmental and long-tract neurological function in NASCIS 2. , 1993, Journal of neurosurgery.

[4]  P. Headley,et al.  Functional evidence for multiple receptor activation by κ‐ligands in the inhibition of spinal nociceptive reflexes in the rat , 1993, British journal of pharmacology.

[5]  S. Lipton Molecular mechanisms of trauma-induced neuronal degeneration. , 1993, Current opinion in neurology and neurosurgery.

[6]  Y. Olsson,et al.  Met-Enkephalin-Arg6-Phe7 in spinal cord and brain following traumatic injury to the spinal cord: Influence of p-chlorophenylalanine. An experimental study in the rat using radioimmunoassay technique , 1993, Neuropharmacology.

[7]  R. Simpson,et al.  The effect of long-term high-dose naloxone infusion in experimental blunt spinal cord injury. , 1993, Journal of spinal disorders.

[8]  Y. Olsson,et al.  Dynorphin A content in the rat brain and spinal cord after a localized trauma to the spinal cord and its modification with p-chlorophenylalanine An experimental study using radioimmunoassay technique , 1992, Neuroscience Research.

[9]  R. Bakshi,et al.  N-methyl-d-aspartate (NMDA) and opioid receptors mediate dynorphin-induced spinal cord injury: behavioral and histological studies , 1992, Brain Research.

[10]  E. Benzel,et al.  Effects of naloxone and nalmefene in rat spinal cord injury induced by the ventral compression technique. , 1992, Journal of spinal disorders.

[11]  R. Vink,et al.  kappa-Opioid antagonist improves cellular bioenergetics and recovery after traumatic brain injury. , 1991, The American journal of physiology.

[12]  C H Tator,et al.  Review of the secondary injury theory of acute spinal cord trauma with emphasis on vascular mechanisms. , 1991, Journal of neurosurgery.

[13]  Tomas Winkler,et al.  Evaluation of traumatic spinal cord edema using evoked potentials recorded from the spinal epidural space An experimental study in the rat , 1991, Journal of the Neurological Sciences.

[14]  R. Traub,et al.  Effects of spinal kappa-opioid receptor agonists on the responsiveness of nociceptive superficial dorsal horn neurons , 1991, Pain.

[15]  A. Light,et al.  Effects of iontophoresed opioids on physiologically characterized laminae I and II dorsal horn neurons in the cat spinal cord , 1990, Brain Research.

[16]  E. Benzel,et al.  Effect of dosage and timing of administration of naloxone on outcome in the rat ventral compression model of spinal cord injury. , 1990, Neurosurgery.

[17]  S. Salzman Neural Monitoring: The Prevention of Intraoperative Injury , 1990 .

[18]  D. C. West,et al.  Spinal antinociceptive actions and naloxone reversibility of intravenous μ‐ and κ‐opioids in spinalized rats: potency mismatch with values reported for spinal administration , 1989, British journal of pharmacology.

[19]  M. Fehlings,et al.  The relationships among the severity of spinal cord injury, motor and somatosensory evoked potentials and spinal cord blood flow. , 1989, Electroencephalography and clinical neurophysiology.

[20]  Balentine Jd,et al.  Spinal cord trauma: in search of the meaning of granular axoplasm and vesicular myelin. , 1988, Journal of neuropathology and experimental neurology.

[21]  R. Hayes,et al.  Endogenous opioids may mediate secondary damage after experimental brain injury. , 1987, The American journal of physiology.

[22]  D. Choi,et al.  High concentrations of naloxone attenuate N-methyl-D-aspartate receptor-mediated neurotoxicity. , 1987, European journal of pharmacology.

[23]  A. Faden,et al.  Traumatic injury alters opiate receptor binding in rat spinal cord , 1986, Annals of neurology.

[24]  A. Faden,et al.  Opiate antagonist WIN44, 441–3 stereospecifically improves neurologic recovery after ischemic spinal injury , 1985, Neurology.

[25]  A. Faden,et al.  Naloxone in experimental spinal cord ischemia: dose-response studies. , 1984, European journal of pharmacology.

[26]  W R Martin,et al.  Pharmacology of opioids. , 1983, Pharmacological reviews.

[27]  R. North,et al.  Electrophysiology of opioids. , 1983, Pharmacological reviews.

[28]  J. Holaday,et al.  Endorphins in Experimental Spinal Injury: Therapeutic Effects of Naloxone , 1982 .

[29]  J. Holaday,et al.  Comparison of early and late naloxone treatment in experimental spinal injury , 1982, Neurology.

[30]  J. Holaday,et al.  Endorphins in experimental spinal injury: Therapeutic effect of naloxone , 1981, Annals of neurology.

[31]  J. C. Torre,et al.  Spinal cord injury. Review of basic and applied research. , 1981 .

[32]  J. Holaday,et al.  Opiate antagonist improves neurologic recovery after spinal injury , 1981 .

[33]  C. Woolf,et al.  The stereospecific effect of naloxone on rat dorsal horn neurones; Inhibition in superficial laminae and excitation in deeper laminae , 1980, PAIN.

[34]  P. Nelson,et al.  Specific-opiate-induced depression of transmitter release from dorsal root ganglion cells in culture. , 1978, Science.

[35]  J. Van Gilder,et al.  Evoked cortical potentials in experimental spinal cord trauma. , 1973, Journal of neurosurgery.

[36]  Y. Olsson,et al.  Influence of dynorphin A antibodies on the formation of edema and cell changes in spinal cord trauma. , 1995, Progress in brain research.

[37]  J. Westman,et al.  The opioid receptor antagonist naloxone influences the pathophysiology of spinal cord injury. , 1995, Progress in brain research.

[38]  S. Fleetwood-Walker,et al.  Opioids and Sensory Processing in the Central Nervous System , 1993 .

[39]  T. Yaksh The Spinal Actions of Opioids , 1993 .

[40]  W. Young,et al.  Secondary injury mechanisms in acute spinal cord injury. , 1993, The Journal of emergency medicine.

[41]  A. Faden Role of Endogenous Opioids and Opioid Receptors in Central Nervous System Injury , 1993 .

[42]  A. Faden,et al.  Pharmacological strategies in CNS trauma. , 1992, Trends in pharmacological sciences.

[43]  W. Collins,et al.  Methylprednisolone or naloxone treatment after acute spinal cord injury: 1-year follow-up data. Results of the second National Acute Spinal Cord Injury Study. , 1992, Journal of neurosurgery.

[44]  Y. Olsson,et al.  Release of endogenous neurochemicals may increase vascular permeability, induce edema and influence cell changes in trauma to the spinal cord. , 1992, Progress in brain research.

[45]  S. Finkelstein,et al.  Naloxone and experimental spinal cord injury: effect of varying dose and intensity of injury. , 1991, Journal of neurotrauma.

[46]  T. Hökfelt,et al.  Neuropeptides in the CNS , 1990 .

[47]  A. Faden,et al.  κ-Selective Opiate Antagonist Nor-Binaltorphimine Improves Outcome After Traumatic Spinal Cord Injury in Rats , 1987 .

[48]  本間 三郎,et al.  Fundamentals and clinical application of spinal cord monitoring. , 1984 .