CNS plasticity and assessment of forelimb sensorimotor outcome in unilateral rat models of stroke, cortical ablation, parkinsonism and spinal cord injury
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[1] T. Schallert,et al. Intervention Strategies for Degeneration of Dopamine Neurons in Parkinsonism , 2000 .
[2] P. Sanberg,et al. Central Nervous System Diseases , 2000, Contemporary Neuroscience.
[3] B. Davidson,et al. Differential effects of glial cell line-derived neurotrophic factor (GDNF) in the striatum and substantia nigra of the aged Parkinsonian rat , 1999, Gene Therapy.
[4] T. Jones,et al. Motor Skills Training Enhances Lesion-Induced Structural Plasticity in the Motor Cortex of Adult Rats , 1999, The Journal of Neuroscience.
[5] T. Schallert,et al. Use-Dependent Exaggeration of Brain Injury: Is Glutamate Involved? , 1999, Experimental Neurology.
[6] M. Murray,et al. Transplants of Fibroblasts Genetically Modified to Express BDNF Promote Regeneration of Adult Rat Rubrospinal Axons and Recovery of Forelimb Function , 1999, The Journal of Neuroscience.
[7] I. Whishaw,et al. Complete Compensation in Skilled Reaching Success with Associated Impairments in Limb Synergies, after Dorsal Column Lesion in the Rat , 1999, The Journal of Neuroscience.
[8] O. Witte,et al. Lesion-induced plasticity as a potential mechanism for recovery and rehabilitative training. , 1998, Current opinion in neurology.
[9] J. Qian,et al. Behavioral and Cellular Protection of Rat Dopaminergic Neurons by an Adenoviral Vector Encoding Glial Cell Line-Derived Neurotrophic Factor , 1998, Experimental Neurology.
[10] M. Chesselet,et al. Motor and Somatosensory Deficits Following Uni- and Bilateral Lesions of the Cortex Induced by Aspiration or Thermocoagulation in the Adult Rat , 1998, Experimental Neurology.
[11] S. Finklestein,et al. Intracisternal osteogenic protein‐1 enhances functional recovery following focal stroke , 1998, Neuroreport.
[12] Dale Corbett,et al. The problem of assessing effective neuroprotection in experimental cerebral ischemia , 1998, Progress in Neurobiology.
[13] T. Schallert,et al. Use-dependent exacerbation of brain damage occurs during an early post-lesion vulnerable period , 1998, Brain Research.
[14] T. Jones,et al. Brain damage, behavior, rehabilitation, recovery, and brain plasticity , 1998 .
[15] T. Robinson,et al. Brain Plasticity and Behavior , 2003, Annual review of psychology.
[16] S. Pellis,et al. Analysis of limb use by control rats and unilateral DA-depleted rats in the Montoya staircase test: movements, impairments and compensatory strategies , 1997, Behavioural Brain Research.
[17] S. Finklestein,et al. Intracisternal basic fibroblast growth factor enhances functional recovery and up-regulates the expression of a molecular marker of neuronal sprouting following focal cerebral infarction. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[18] T. Schallert,et al. Use-dependent structural events in recovery of function. , 1997, Advances in neurology.
[19] T. Schallert,et al. Use-Dependent Exaggeration of Neuronal Injury after Unilateral Sensorimotor Cortex Lesions , 1996, The Journal of Neuroscience.
[20] J. Grotta,et al. An Alternative Method for the Quantitation of Neuronal Damage after Experimental Middle Cerebral Artery Occlusion in Rats: Analysis of Behavioral Deficit , 1996, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[21] Dennis Higgins,et al. Osteogenic protein-1 induces dendritic growth in rat sympathetic neurons , 1995, Neuron.
[22] Shelley R. Winn,et al. Implantation of encapsulated catecholamine and GDNF-producing cells in rats with unilateral dopamine depletions and parkinsonian symptoms , 1995, Experimental Neurology.
[23] T. Jones,et al. Use-dependent growth of pyramidal neurons after neocortical damage , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[24] T. Jones,et al. "Exuberant" Neuronal Growth After Brain Damage in Adult Rats: The Essential Role of Behavioral Experience , 1993, Journal of neural transplantation & plasticity.
[25] T. Jones,et al. Subcortical deterioration after cortical damage: effects of diazepam and relation to recovery of function , 1992, Behavioural Brain Research.
[26] Theresa A. Jones,et al. Overgrowth and pruning of dendrites in adult rats recovering from neocortical damage , 1992, Brain Research.
[27] E. J. Green,et al. Sensorimotor and cognitive consequences of middle cerebral artery occlusion in rats , 1992, Brain Research.
[28] B. Will,et al. Environmental approaches to recovery of function from brain damage: a review of animal studies (1981 to 1991). , 1992, Advances in experimental medicine and biology.
[29] Ian Q. Whishaw,et al. The impairments in reaching and the movements of compensation in rats with motor cortex lesions: an endpoint, videorecording, and movement notation analysis , 1991, Behavioural Brain Research.
[30] T. Schallert,et al. Effects of MK-801 on recovery from sensorimotor cortex lesions. , 1990, Stroke.
[31] T. Jones,et al. Functional subdivisions of the rat somatic sensorimotor cortex , 1990, Behavioural Brain Research.
[32] T. Schallert,et al. Rescuing neurons from trans-synaptic degeneration after brain damage: helpful, harmful, or neutral in recovery of function? , 1990, Canadian journal of psychology.
[33] D. Wiebers,et al. Animal models of stroke: are they relevant to human disease? , 1990, Stroke.
[34] Timothy Schallert,et al. Seizures and recovery from experimental brain damage , 1988, Experimental Neurology.
[35] T. E. Levere,et al. Neural System Imbalances and the Consequence of Large Brain Injuries , 1988 .
[36] Stanley Finger,et al. Brain injury and recovery : theoretical and controversial issues , 1988 .
[37] T. Schallert,et al. Recovery of function after brain damage: Severe and chronic disruption by diazepam , 1986, Brain Research.
[38] I Q Whishaw,et al. Bilateral cutaneous stimulation of the somatosensory system in hemidecorticate rats. , 1984, Behavioral neuroscience.
[39] M. Sarter,et al. Behavioral and neuronal reorganization after unilateral substantia nigra lesions: Evidence for increased interhemispheric nigrostriatal projections , 1983, Neuroscience.
[40] T. Schallert,et al. Posture-independent sensorimotor analysis of inter-hemispheric receptor asymmetries in neostriatum , 1983, Pharmacology Biochemistry and Behavior.
[41] N. Lobaugh,et al. Tactile extinction: Distinguishing between sensorimotor and motor asymmetries in rats with unilateral nigrostriatal damage , 1982, Pharmacology Biochemistry and Behavior.
[42] D. Graham,et al. Focal Cerebral Ischaemia in the Rat: 1. Description of Technique and Early Neuropathological Consequences following Middle Cerebral Artery Occlusion , 1981, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[43] R. Hall,et al. Organization of motor and somatosensory neocortex in the albino rat , 1974 .
[44] A. Castro,et al. Motor performance in rats. The effects of pyramidal tract section. , 1972, Brain research.
[45] P. Teitelbaum,et al. The lateral hypothalamic syndrome: recovery of feeding and drinking after lateral hypothalamic lesions. , 1962, Psychological review.