This information is current as through Extracellular UDP Release Protect Mice against Bacterial Infection TLR-Activated Gap Junction Channels
暂无分享,去创建一个
Xiaoyu Zhang | B. Tan | Mingyao Liu | Juliang Qin | Hua Ren | Guangxu Zhang | Min Qian | Dubeĭ Lv | Binghe Tan
[1] N. Feldman,et al. DAMPs as mediators of sterile inflammation in aging-related pathologies , 2015, Ageing Research Reviews.
[2] P. Benaroch,et al. Extracellular ATP induces the rapid release of HIV-1 from virus containing compartments of human macrophages , 2015, Proceedings of the National Academy of Sciences.
[3] B. Brannetti,et al. ATP-gated ionotropic P2X7 receptor controls follicular T helper cell numbers in Peyer's patches to promote host-microbiota mutualism. , 2014, Immunity.
[4] Guangquan Ji,et al. Atractylenolide I inhibits lipopolysaccharide-induced inflammatory responses via mitogen-activated protein kinase pathways in RAW264.7 cells , 2014, Immunopharmacology and immunotoxicology.
[5] Mingyao Liu,et al. Extracellular UDP and P2Y6 Function as a Danger Signal To Protect Mice from Vesicular Stomatitis Virus Infection through an Increase in IFN-β Production , 2014, The Journal of Immunology.
[6] Rongan Zhang,et al. P2Y6 Receptor Potentiates Pro-Inflammatory Responses in Macrophages and Exhibits Differential Roles in Atherosclerotic Lesion Development , 2014, PloS one.
[7] Mingyao Liu,et al. Toll-Like Receptor-Triggered Calcium Mobilization Protects Mice against Bacterial Infection through Extracellular ATP Release , 2014, Infection and Immunity.
[8] M. Hirata,et al. Pulmonary Infection with Hypervirulent Mycobacteria Reveals a Crucial Role for the P2X7 Receptor in Aggressive Forms of Tuberculosis , 2014, PLoS pathogens.
[9] M. Idzko,et al. Nucleotide signalling during inflammation , 2014, Nature.
[10] A. Prince,et al. Sessile alveolar macrophages communicate with alveolar epithelium to modulate immunity , 2014, Nature.
[11] S. Phipps,et al. RAGE and TLRs: relatives, friends or neighbours? , 2013, Molecular immunology.
[12] Jean X. Jiang,et al. ATP Is Required and Advances Cytokine-Induced Gap Junction Formation in Microglia In Vitro , 2013, Mediators of inflammation.
[13] Y. Moriyama,et al. Autocrine Regulation of Macrophage Activation via Exocytosis of ATP and Activation of P2Y11 Receptor , 2013, PloS one.
[14] Roger J. Thompson,et al. Activation of neuronal P2X7 receptor-Pannexin-1 mediates death of enteric neurons during colitis , 2012, Nature Medicine.
[15] Ji-Eun Kim,et al. The P2X7 receptor-pannexin-1 complex decreases muscarinic acetylcholine receptor-mediated seizure susceptibility in mice. , 2011, The Journal of clinical investigation.
[16] Mingyao Liu,et al. P2Y6 Agonist Uridine 5′-Diphosphate Promotes Host Defense against Bacterial Infection via Monocyte Chemoattractant Protein-1–Mediated Monocytes/Macrophages Recruitment , 2011, The Journal of Immunology.
[17] Sang Yoon Lee,et al. Uridine 5′-Diphosphate Induces Chemokine Expression in Microglia and Astrocytes through Activation of the P2Y6 Receptor , 2011, The Journal of Immunology.
[18] P. Insel,et al. Pannexin-1 hemichannel-mediated ATP release together with P2X1 and P2X4 receptors regulate T-cell activation at the immune synapse. , 2010, Blood.
[19] Michael R. Elliott,et al. Pannexin 1 channels mediate ‘find-me’ signal release and membrane permeability during apoptosis , 2010, Nature.
[20] J. Sévigny,et al. Concomitant activation of P2Y2 and P2Y6 receptors on monocytes is required for TLR1/2‐induced neutrophil migration by regulating IL‐8 secretion , 2009, European journal of immunology.
[21] Michael R. Elliott,et al. Nucleotides released by apoptotic cells act as a find-me signal to promote phagocytic clearance , 2009, Nature.
[22] K. Schalper,et al. Modulation of brain hemichannels and gap junction channels by pro-inflammatory agents and their possible role in neurodegeneration. , 2009, Antioxidants & redox signaling.
[23] S. Basta,et al. An efficient culture method for generating large quantities of mature mouse splenic macrophages. , 2008, Journal of immunological methods.
[24] J. Buer,et al. Purinergic Control of T Cell Activation by ATP Released Through Pannexin-1 Hemichannels , 2008, Science Signaling.
[25] Osamu Takeuchi,et al. Sequential control of Toll-like receptor–dependent responses by IRAK1 and IRAK2 , 2008, Nature Immunology.
[26] J. Neefjes,et al. Gap junction-mediated intercellular communication in the immune system. , 2007, Progress in biophysics and molecular biology.
[27] K. Jacobson,et al. UDP acting at P2Y6 receptors is a mediator of microglial phagocytosis , 2007, Nature.
[28] S. Akira,et al. Pathogen Recognition and Innate Immunity , 2006, Cell.
[29] K. Jacobson,et al. P2Y6 Nucleotide Receptor Activates PKC to Protect 1321N1 Astrocytoma Cells Against Tumor Necrosis Factor-Induced Apoptosis , 2003, Cellular and Molecular Neurobiology.
[30] Y. Shibata,et al. Gap junctions in the cardiac muscle cells of the lamprey , 1977, Cell and Tissue Research.
[31] B. Nicholson. Gap junctions – from cell to molecule , 2003, Journal of Cell Science.
[32] J. Berman,et al. TNF-α Plus IFN-γ Induce Connexin43 Expression and Formation of Gap Junctions Between Human Monocytes/Macrophages That Enhance Physiological Responses 1 , 2003, The Journal of Immunology.
[33] M. Rosendaal,et al. Direct cell/cell communication in the lymphoid germinal center: connexin43 gap junctions functionally couple follicular dendritic cells to each other and to B lymphocytes , 1997, European journal of immunology.
[34] B L Langille,et al. Cardiac malformation in neonatal mice lacking connexin43. , 1995, Science.
[35] D. Erlinge,et al. The stable pyrimidines UDPbS and UTPgS discriminate between the P2 receptors that mediate vascular contraction and relaxation of the rat mesenteric artery , 2022 .