MicroRNA expression profiling and target genes study in congenital microtia.
暂无分享,去创建一个
Duan Ma | Hui-jun Wang | Lei Jin | Shao-juan Hao | Ping Zhang | Chen-long Li | Fengyun Zheng | Tianyu Zhang | Long Chen | Fenghua Qing
[1] Haitao Li,et al. MicroRNA array analysis of microRNAs related to systemic scleroderma , 2012, Rheumatology International.
[2] M. Cunningham,et al. Microtia: Epidemiology and genetics , 2012, American journal of medical genetics. Part A.
[3] A. Czeizel,et al. Maternal diseases and risk of isolated ear abnormalities in their children. , 2011, Central European journal of public health.
[4] Pierpaolo Mastroiacovo,et al. Microtia-anotia: a global review of prevalence rates. , 2011, Birth defects research. Part A, Clinical and molecular teratology.
[5] R. Rosti,et al. No association between DNA repair gene (XPD, XRCC1, and XRCC4) polymorphisms and nonsyndromic microtia in Turkish patients. , 2011, Plastic and reconstructive surgery.
[6] K. Shrey,et al. Micro RNAs: tiny sequences with enormous potential. , 2011, Biochemical and biophysical research communications.
[7] B. Pan,et al. Mutational analysis of PACT gene in Chinese patients with microtia , 2011, American journal of medical genetics. Part A.
[8] B. Morrow,et al. Mutational analysis of HOXA2 and SIX2 in a Bronx population with isolated microtia. , 2010, International journal of pediatric otorhinolaryngology.
[9] Stefano Piccolo,et al. MicroRNA control of signal transduction , 2010, Nature Reviews Molecular Cell Biology.
[10] W. Yin,et al. Pedigree and Genetic Study of a Bilateral Congenital Microtia Family , 2010, Plastic and reconstructive surgery.
[11] Jinfang Wu,et al. Epidemiological analysis of microtia: a retrospective study in 345 patients in China. , 2010, International journal of pediatric otorhinolaryngology.
[12] B. Pan,et al. [Study of methylation of promoter of EYA1 gene in microtia]. , 2009, Zhonghua zheng xing wai ke za zhi = Zhonghua zhengxing waike zazhi = Chinese journal of plastic surgery.
[13] Qingguo Zhang,et al. Environmental and Genetic Factors Associated with Congenital Microtia: A Case-Control Study in Jiangsu, China, 2004 to 2007 , 2009, Plastic and reconstructive surgery.
[14] Yibing Yin,et al. Microarray profile of micro‐ribonucleic acid in tumor tissue from cervical squamous cell carcinoma without human papillomavirus , 2009, The journal of obstetrics and gynaecology research.
[15] J. Seidman,et al. A classic twin study of external ear malformations, including microtia. , 2009, The New England journal of medicine.
[16] G. Peters,et al. The double-stranded RNA-binding protein, PACT, is required for postnatal anterior pituitary proliferation , 2009, Proceedings of the National Academy of Sciences.
[17] X. Chen,et al. Role of miR-143 targeting KRAS in colorectal tumorigenesis , 2009, Oncogene.
[18] Duan Ma,et al. The cell growth suppressor, mir-126, targets IRS-1. , 2008, Biochemical and biophysical research communications.
[19] Haoming Zhang,et al. miR-16 family induces cell cycle arrest by regulating multiple cell cycle genes , 2008, Nucleic acids research.
[20] M. Tekin,et al. Homozygous FGF3 mutations result in congenital deafness with inner ear agenesis, microtia, and microdontia , 2008, Clinical genetics.
[21] S. Amladi,et al. Goldenhar syndrome with unusual features. , 2008, Indian journal of dermatology, venereology and leprology.
[22] M. Farhadi,et al. A mutation in HOXA2 is responsible for autosomal-recessive microtia in an Iranian family. , 2008, American journal of human genetics.
[23] A. James,et al. Bifid tongue: A rare feature associated with infants of diabetic mother syndrome , 2007, American journal of medical genetics. Part A.
[24] Martha L Bulyk,et al. Microarray Analyses of Newborn Mouse Ovaries Lacking Nobox1 , 2007, Biology of reproduction.
[25] A. Feinberg. Phenotypic plasticity and the epigenetics of human disease , 2007, Nature.
[26] R. Kosaki,et al. Wide phenotypic variations within a family with SALL1 mutations: Isolated external ear abnormalities to Goldenhar syndrome , 2007, American journal of medical genetics. Part A.
[27] K. Hirose,et al. A role of the double-stranded RNA-binding protein PACT in mouse ear development and hearing. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[28] D. Wieczorek,et al. Histone acetylation dependent allelic expression imbalance of BAPX1 in patients with the oculo-auriculo-vertebral spectrum. , 2006, Human molecular genetics.
[29] H. Zhuang,et al. [Case control study on risk factors of congenital microtia]. , 2006, Zhonghua er bi yan hou tou jing wai ke za zhi = Chinese journal of otorhinolaryngology head and neck surgery.
[30] E. Cho,et al. Odd-skipped related 1 (Odd 1) is an essential regulator of heart and urogenital development. , 2005, Developmental biology.
[31] S. O’Gorman. Second branchial arch lineages of the middle ear of wild‐type and Hoxa2 mutant mice , 2005, Developmental dynamics : an official publication of the American Association of Anatomists.
[32] T. Roscioli,et al. Pallister–Hall syndrome: Unreported skeletal features of a GLI3 mutation , 2005, American journal of medical genetics. Part A.
[33] K. Gunsalus,et al. Combinatorial microRNA target predictions , 2005, Nature Genetics.
[34] S. Ishimoto,et al. Correlation between microtia and temporal bone malformation evaluated using grading systems. , 2005, Archives of otolaryngology--head & neck surgery.
[35] Anton J. Enright,et al. Human MicroRNA Targets , 2004, PLoS biology.
[36] M. Werler,et al. Vasoactive exposures, vascular events, and hemifacial microsomia. , 2004, Birth defects research. Clinical and molecular teratology.
[37] A. Hatzigeorgiou,et al. A combined computational-experimental approach predicts human microRNA targets. , 2004, Genes & development.
[38] J. M. Rogers,et al. Effects of gestational exposure to ethane dimethanesulfonate in CD-1 mice: microtia and preliminary hearing tests. , 2003, Birth defects research. Part B, Developmental and reproductive toxicology.
[39] K. Livak,et al. Real time quantitative PCR. , 1996, Genome research.
[40] S. Potter,et al. Dominant mutation of the murine Hox-2.2 gene results in developmental abnormalities. , 1992, The Journal of experimental zoology.