Anti-inflammatory mechanism of PPARγ on LPS-induced pulp cells: role of the ROS removal activity.

[1]  Junliang Yang,et al.  Naringenin attenuates mucous hypersecretion by modulating reactive oxygen species production and inhibiting NF-κB activity via EGFR-PI3K-Akt/ERK MAPKinase signaling in human airway epithelial cells , 2011, Molecular and Cellular Biochemistry.

[2]  L. Biasucci,et al.  Inflammatory markers, cholesterol and statins: pathophysiological role and clinical importance , 2010, Clinical chemistry and laboratory medicine.

[3]  H. Yi,et al.  Attenuation of AH26-induced apoptosis by inhibition of SAPK/JNK pathway in MC-3T3 E1 cells. , 2010, Journal of endodontics.

[4]  H. Yi,et al.  PPARγ inhibits inflammatory reaction in oxidative stress induced human diploid fibloblast , 2010, Cell biochemistry and function.

[5]  N. Lee,et al.  Artemisia fukudo essential oil attenuates LPS-induced inflammation by suppressing NF-kappaB and MAPK activation in RAW 264.7 macrophages. , 2010, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[6]  M. Hasegawa,et al.  MAPK Signaling Is Required for LPS-induced VEGF in Pulp Stem Cells , 2010, Journal of dental research.

[7]  H. Yi,et al.  Anti-inflammatory effect of peroxisome proliferator activated receptor gamma on human dental pulp cells. , 2009, Journal of endodontics.

[8]  R. Vaillancourt,et al.  MAP3Ks as central regulators of cell fate during development , 2008, Developmental dynamics : an official publication of the American Association of Anatomists.

[9]  J. P. Issa,et al.  Action of nitric oxide on healthy and inflamed human dental pulp tissue. , 2008, Micron.

[10]  C. Lam,et al.  Tumor necrosis factor-alpha up-regulates the expression of CCL2 and adhesion molecules of human proximal tubular epithelial cells through MAPK signaling pathways. , 2008, Immunobiology.

[11]  Seung-Heon Hong,et al.  Involvement of mitogen-activated protein kinases and nuclear factor-kappa B activation in nitric oxide-induced interleukin-8 expression in human pulp cells. , 2008, Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics.

[12]  I. Yoo,et al.  Terrein reduces pulpal inflammation in human dental pulp cells. , 2008, Journal of endodontics.

[13]  Sang-Oh Yoon,et al.  Pro‐MMP‐2 activation by the PPARγ agonist, ciglitazone, induces cell invasion through the generation of ROS and the activation of ERK , 2007, FEBS letters.

[14]  Chuen-Mao Yang,et al.  Involvement of MAPKs and NF-kappaB in LPS-induced VCAM-1 expression in human tracheal smooth muscle cells. , 2007, Cellular signalling.

[15]  P. Pavasant,et al.  Transforming growth factor-beta1 up-regulates the expression of nerve growth factor through mitogen-activated protein kinase signaling pathways in dental pulp cells. , 2007, European journal of oral sciences.

[16]  Liduan Zheng,et al.  Hypoxia-Induced Mitogenic Factor Promotes Vascular Adhesion Molecule-1 Expression via the PI-3K/Akt–NF-κB Signaling Pathway , 2006 .

[17]  B. Kamińska.  MAPK signalling pathways as molecular targets for anti-inflammatory therapy--from molecular mechanisms to therapeutic benefits. , 2005, Biochimica et biophysica acta.

[18]  I. Keskiner,et al.  Reactive oxygen species scavenging activity during periodontal mucoperiosteal healing: an experimental study in dogs. , 2005, Archives of oral biology.

[19]  J. Atkinson,et al.  Pioglitazone Improves Aortic Wall Elasticity in a Rat Model of Elastocalcinotic Arteriosclerosis , 2005, Hypertension.

[20]  Jong-Suk Kim,et al.  PPAR‐gamma modulates allergic inflammation through up‐regulation of PTEN , 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[21]  B. P. Kota,et al.  An overview on biological mechanisms of PPARs. , 2005, Pharmacological research.

[22]  P C Trevilatto,et al.  Inhibition of human pulpal gelatinases (MMP-2 and MMP-9) by zinc oxide cements. , 2004, Journal of oral rehabilitation.

[23]  A. Lennon,et al.  MAP Kinase Cascades Are Activated in Astrocytes and Preadipocytes by 15-Deoxy-Δ12–14-prostaglandin J2 and the Thiazolidinedione Ciglitazone through Peroxisome Proliferator Activator Receptor γ-independent Mechanisms Involving Reactive Oxygenated Species* , 2002, The Journal of Biological Chemistry.

[24]  T. Mayumi,et al.  PPAR gamma ligands inhibit nitrotyrosine formation and inflammatory mediator expressions in adjuvant-induced rheumatoid arthritis mice. , 2002, European journal of pharmacology.

[25]  W. Pfister,et al.  Comparison of profiles of key periodontal pathogens in periodontium and endodontium. , 2000, Endodontics & dental traumatology.

[26]  G. Gebhart,et al.  Localization and Changes in NADPH-Diaphorase Reactivity and Nitric Oxide Synthase Immunoreactivity in Rat Pulp Following Tooth Preparation , 1999, Journal of dental research.

[27]  F. Sánchez‐Madrid,et al.  Adhesion Molecules in Inflammatory Diseases , 1998, Drugs.

[28]  B. Staels,et al.  Activation of Proliferator-activated Receptors α and γ Induces Apoptosis of Human Monocyte-derived Macrophages* , 1998, The Journal of Biological Chemistry.

[29]  Y. Sawa,et al.  Vascular endothelium of human dental pulp expresses diverse adhesion molecules for leukocyte emigration. , 1998, Tissue & cell.

[30]  B. Spiegelman,et al.  Stimulation of adipogenesis in fibroblasts by PPARγ2, a lipid-activated transcription factor , 1994, Cell.

[31]  S. Hamada,et al.  Induction of interleukin-1 and -6 in human gingival fibroblast cultures stimulated with Bacteroides lipopolysaccharides , 1991, Infection and immunity.

[32]  T. Moore,et al.  Mediators of inflammation. , 1985, Seminars in arthritis and rheumatism.

[33]  P. Jarolim,et al.  The dynamics of gene expression in human lung microvascular endothelial cells after stimulation with inflammatory cytokines. , 2006, Physiological research.

[34]  H. Northoff,et al.  Free radicals and oxidative stress in exercise--immunological aspects. , 1999, Exercise immunology review.