Improvement of fractional flow reserve and collateral flow by treatment with external counterpulsation (Art.Net.‐2 Trial)

Background  Arteriogenesis (collateral artery growth) is nature’s most efficient rescue mechanism to overcome the fatal consequences of arterial occlusion or stenosis. The goal of this trial was to investigate the effect of external counterpulsation (ECP) on coronary collateral artery growth.

[1]  A. Khaledifar,et al.  Evaluation of left ventricular systolic and diastolic regional function after enhanced external counter pulsation therapy using strain rate imaging. , 2009, European journal of echocardiography : the journal of the Working Group on Echocardiography of the European Society of Cardiology.

[2]  W. Howard,et al.  Optimal Medical Therapy with or without PCI for Stable Coronary Disease , 2008 .

[3]  Pascal Meier,et al.  Beneficial Effect of Recruitable Collaterals: A 10-Year Follow-Up Study in Patients With Stable Coronary Artery Disease Undergoing Quantitative Collateral Measurements , 2007, Circulation.

[4]  Yan Zhang,et al.  Enhanced External Counterpulsation Inhibits Intimal Hyperplasia by Modifying Shear Stress–Responsive Gene Expression in Hypercholesterolemic Pigs , 2007, Circulation.

[5]  C. Seiler.  The Coronary Collateral Circulation in Man , 2007 .

[6]  S. Windecker,et al.  Coronary collateral flow in response to endurance exercise training , 2007, European journal of cardiovascular prevention and rehabilitation : official journal of the European Society of Cardiology, Working Groups on Epidemiology & Prevention and Cardiac Rehabilitation and Exercise Physiology.

[7]  T. Henry,et al.  Primer: practical approach to the selection of patients for and application of EECP , 2006, Nature Clinical Practice Cardiovascular Medicine.

[8]  A. Simon,et al.  Effects of Enhanced External Counterpulsation on Carotid Circulation in Patients with Coronary Artery Disease , 2006, Cardiology.

[9]  Thomas Schmitz-Rixen,et al.  The Range of Adaptation by Collateral Vessels After Femoral Artery Occlusion , 2006, Circulation research.

[10]  R. de Caterina,et al.  Rapid decline of collateral circulation increases susceptibility to myocardial ischemia: the trade-off of successful percutaneous recanalization of chronic total occlusions. , 2006, Journal of the American College of Cardiology.

[11]  Antonio Colombo,et al.  Guidelines for percutaneous coronary interventions. The Task Force for Percutaneous Coronary Interventions of the European Society of Cardiology. , 2005, European heart journal.

[12]  Ganesh Manoharan,et al.  Epicardial Stenosis Severity Does Not Affect Minimal Microcirculatory Resistance , 2004, Circulation.

[13]  W. Schaper,et al.  Influence of Mechanical, Cellular, and Molecular Factors on Collateral Artery Growth (Arteriogenesis) , 2004, Circulation research.

[14]  R. Nohara,et al.  Improvement of oxygen metabolism in ischemic myocardium as a result of enhanced external counterpulsation with heparin pretreatment for patients with stable angina , 2004, Heart and Vessels.

[15]  G. Mitchell,et al.  Effects of shear stress and flow pulsatility on endothelial function: insights gleaned from external counterpulsation therapy. , 2003, Journal of the American College of Cardiology.

[16]  A. Yeung,et al.  Novel Index for Invasively Assessing the Coronary Microcirculation , 2003, Circulation.

[17]  D. Holmes,et al.  Enhanced external counterpulsation for ischemic heart disease: what's behind the curtain? , 2003, Journal of the American College of Cardiology.

[18]  R. Gibbons.  ACC/AHA guideline for management of patients with chronic stable angina , 2003 .

[19]  Sankey V. Williams,et al.  ACC/AHA 2002 guideline update for the management of patients with chronic stable angina--summary article: a report of the American College of Cardiology/American Heart Association Task Force on practice guidelines (Committee on the Management of Patients With Chronic Stable Angina). , 2003, Journal of the American College of Cardiology.

[20]  浦野 久.  Enhanced external counterpulsation improves exercise tolerance, reduces exercise-induced myocardial ischemia and improves left ventricular diastolic filling in patients with coronary artery disease , 2002 .

[21]  H. Figulla,et al.  Collateral Function in Chronic Total Coronary Occlusions Is Related to Regional Myocardial Function and Duration of Occlusion , 2001, Circulation.

[22]  F. Eberli,et al.  Frequency distribution of collateral flow and factors influencing collateral channel development. Functional collateral channel measurement in 450 patients with coronary artery disease. , 2001, Journal of the American College of Cardiology.

[23]  F. Eberli,et al.  Promotion of Collateral Growth by Granulocyte-Macrophage Colony-Stimulating Factor in Patients With Coronary Artery Disease: A Randomized, Double-Blind, Placebo-Controlled Study , 2001, Circulation.

[24]  R. Nohara,et al.  Enhanced external counterpulsation improved myocardial perfusion and coronary flow reserve in patients with chronic stable angina; evaluation by(13)N-ammonia positron emission tomography. , 2001, European heart journal.

[25]  H. Trappe,et al.  [Guidelines for ergometry. German Society of Cardiology--Heart and Cardiovascular Research]. , 2000, Zeitschrift fur Kardiologie.

[26]  G. Schuler,et al.  Effect of Exercise on Coronary Endothelial Function in Patients With Coronary Artery Disease , 2000 .

[27]  R. Nesto,et al.  The multicenter study of enhanced external counterpulsation (MUST-EECP): effect of EECP on exercise-induced myocardial ischemia and anginal episodes. , 1999, Journal of the American College of Cardiology.

[28]  B Meier,et al.  Coronary collateral quantitation in patients with coronary artery disease using intravascular flow velocity or pressure measurements. , 1998, Journal of the American College of Cardiology.

[29]  B. Meier,et al.  Long-term physical exercise and quantitatively assessed human coronary collateral circulation. , 1998, Journal of the American College of Cardiology.

[30]  P. H. van der Voort,et al.  Measurement of fractional flow reserve to assess the functional severity of coronary-artery stenoses. , 1996, The New England journal of medicine.

[31]  Thomas J. Ryan,et al.  Guidelines for Percutaneous Transluminal Coronary Angioplasty A Report of the American Heart Association/American College of Cardiology Task Force on Assessment of Diagnostic and Therapeutic Cardiovascular Procedures (Committee on Percutaneous Transluminal Coronary Angioplasty) , 1993, Circulation.

[32]  S. Kaul,et al.  An association between collateral blood flow and myocardial viability in patients with recent myocardial infarction. , 1992, The New England journal of medicine.

[33]  S. Chierchia,et al.  Coronary collaterals reduce the duration of exercise-induced ischemia by allowing a faster recovery. , 1992, American heart journal.

[34]  T. Ryan.  Guidelines for percutaneous transluminal coronary angioplasty. A report of the American College of Cardiology/American Heart Association Task Force on Assessment of Diagnostic and Therapeutic Cardiovascular Procedures (Subcommittee on Percutaneous Transluminal Coronary Angioplasty). , 1988, Journal of the American College of Cardiology.

[35]  F. Loop,et al.  Guidelines for percutaneous transluminal coronary angioplasty. A report of the American College of Cardiology/American Heart Association Task Force on Assessment of Diagnostic and Therapeutic Cardiovascular Procedures (Subcommittee on Percutaneous Transluminal Coronary Angioplasty). , 1988, Circulation.

[36]  R. Helfant,et al.  The coronary collateral circulation. , 1971, Annals of internal medicine.