ABSTRACT Introduction : Malaria infection could activate T cell helper 2 CD4+ which release cytokines IL-4, IL-5, IL-10 was a kind of immunosuppressant cytokines and prevented a cerebral tissue damage from the expression intercellular adhesion molecule-1 (ICAM-1) by TNF-α, ended with a cerebral malaria. The red fruit was proved as antioxidant but its effect to reduce TNF-α and expression of ICAM-1 in cerebral tissue endothelial cell remained unknown. Objectives : to know the effect of red fruit ethanol extract toward the change of TNF-α secretion number and ICAM-1 in Swiss male mice’s cerebral endothelial cell infected with P.berghei . Methods : Sixty Swiss male mice were divided in to 6 groups, group I was not given the red fruit (BM) ethanol extract and not infected P.berghei , Group II was given BM 260mg/kg BB but not infected P.berghei , group III, IV, V were given BM 130, 260, and 520 mg/kg respectively and infected with P.berghei . Red fruit extract or carrier substance was given for 4 weeks, P.berghei was given in the early of third week. TNF-α level check was done in third and ninth day after the injection, with TNF-α ELISA kit mouse. ICAM-1 cerebral endotel cell expression check was done in third and nine day with immunohistochemistry (IHC) coloring. Results : TNF-α level in third day after infected to the groups given red fruit extract (group III, IV, and V) showed no significant difference (p=0,839) compared to group which was not given red fruit but infected (group VI). TNF-α level in ninth day after infected to groups given red fruit ( group III,IV,V) lower (p<0,05) compared to groups which was not given red fruit but infected. ICAM-1 expression in third and ninth day after treatment to group given red fruit extract was lower (p<0,05) compared to groups given red fruit extract but infected. Conclusion : Red fruit ethanol extract ( P conoideus Lam) could decrease TNF-α level and ICAM-I expression in Swiss mice’ endothelial cerebral cells after infected by P.berghei. Key word : Pandanus conoideus Lam, TNF-α, ICAM-1 expression, Plasmodium berghei , Swiss mice
[1]
S. Morse,et al.
Factors in the emergence of infectious diseases.
,
1995,
Emerging infectious diseases.
[2]
N. Mathew.
Drug-induced headache.
,
1988,
Neurologic clinics.
[3]
H. Lillywhite,et al.
Pulmonary blood flow regulation in an aquatic snake.
,
1989,
Science.
[4]
J. Manners,et al.
DDAVP and open heart surgery
,
1989,
Anaesthesia.
[5]
Martin Farrall,et al.
PREDISPOSING LOCUS FOR ALZHEIMER'S DISEASE ON CHROMOSOME 21
,
1989,
The Lancet.
[6]
F. Meyskens,et al.
Effect of oral isotretinoin on dysplastic nevi.
,
1989,
Journal of the American Academy of Dermatology.
[7]
A. Piccoli,et al.
Early steroid therapy in IgA nephropathy: still an open question.
,
1989,
Nephron.
[8]
F. Magni,et al.
BN-52021 protects guinea-pig from heart anaphylaxis.
,
1988,
Pharmacological research communications.
[9]
G. Gardos,et al.
The natural history of tardive dyskinesia.
,
1988,
Journal of clinical psychopharmacology.
[10]
Y. Kobayashi,et al.
Steroid therapy in IgA nephropathy: a retrospective study in heavy proteinuric cases.
,
1988,
Nephron.
[11]
J. D. Perry.
Sports Injuries and Their Treatment
,
1987
.
[12]
L. Massone,et al.
LOCALISATIONS PALMAIRES PURPURIQUES DE LA DERMATITE HERPETIFORME
,
1987
.
[13]
W. Chey,et al.
Electrogastrographic study of patients with unexplained nausea, bloating, and vomiting.
,
1980,
Gastroenterology.