Metabolism of heterocyclic aromatic amines by human hepatocytes and cytochrome P4501A2.
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[1] A. Guillouzo,et al. Differential metabolism of 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine in rat and human hepatocytes. , 2002, Carcinogenesis.
[2] F. Guengerich,et al. Metabolism of 2-amino-3,8-dimethylimidazo[4,5-f]quinoxaline in human hepatocytes: 2-amino-3-methylimidazo[4,5-f]quinoxaline-8-carboxylic acid is a major detoxification pathway catalyzed by cytochrome P450 1A2. , 2001, Chemical research in toxicology.
[3] K. Kulp,et al. Identification of urine metabolites of 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine following consumption of a single cooked chicken meal in humans. , 2000, Carcinogenesis.
[4] T. Sugimura,et al. Nutrition and dietary carcinogens. , 2000, Carcinogenesis.
[5] S R Tannenbaum,et al. N-oxidative metabolism of 2-amino-3,8-dimethylimidazo[4,5-f]quinoxaline (MeIQx) in humans: excretion of the N2-glucuronide conjugate of 2-hydroxyamino-MeIQx in urine. , 1999, Cancer research.
[6] K. Turteltaub,et al. Macromolecular adduct formation and metabolism of heterocyclic amines in humans and rodents at low doses. , 1999, Cancer letters.
[7] K. Turteltaub,et al. DNA and protein adduct formation in the colon and blood of humans after exposure to a dietary-relevant dose of 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine. , 1999, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.
[8] F. Guengerich,et al. Activation of heterocyclic aromatic amines by rat and human liver microsomes and by purified rat and human cytochrome P450 1A2. , 1998, Chemical research in toxicology.
[9] A. Guillouzo,et al. Liver cell models in in vitro toxicology. , 1998, Environmental health perspectives.
[10] S. Leveson,et al. Metabolism of the food-borne mutagen 2-amino-3,8-dimethylimidazo[4,5-f]quinoxaline in humans. , 1998, Chemical research in toxicology.
[11] H. Yamazaki,et al. Activation of chemically diverse procarcinogens by human cytochrome P-450 1B1. , 1996, Cancer research.
[12] A. Ronco,et al. Meat, fat and risk of breast cancer: A case‐control study from Uruguay , 1996, International journal of cancer.
[13] W. Willett,et al. Diet, nutrition, and avoidable cancer. , 1995, Environmental health perspectives.
[14] J. Holme,et al. Metabolism of the food carcinogen 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) in the rat and other rodents. , 1995, Princess Takamatsu symposia.
[15] J. Shaddock,et al. Effect of glutathione depletion and inhibition of glucuronidation and sulfation on 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) metabolism, PhIP-DNA adduct formation and unscheduled DNA synthesis in primary rat hepatocytes. , 1994, Carcinogenesis.
[16] J. Groopman,et al. The direct glucuronidation of 2-amino-1-methyl-6-phenylimidazo[4,5-b] pyridine (PhIP) by human and rabbit liver microsomes. , 1993, Chemical research in toxicology.
[17] W. Trager,et al. Isoform-selective mechanism-based inhibition of human cytochrome P450 1A2 by furafylline. , 1993, Chemical research in toxicology.
[18] M. Knize,et al. Heterocyclic-Amine Mutagens/Carcinogens in Foods , 1990 .
[19] D. Kingston,et al. Isolation, Structure Elucidation, and Synthesis of the Major Anaerobic Bacterial Metabolite of the Dietary Carcinogen 2-Amino-3,4-dimethyl-3H-imidazo[4,5-f]quinoline (meIQ) , 1989 .
[20] S. Thorgeirsson,et al. Metabolic activation of mutagenic heterocyclic aromatic amines from protein pyrolysates. , 1986, Critical reviews in toxicology.