Increased RNA Transcription of Energy Source Transporters in Circulating White Blood Cells of Aged Mice
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S. Gul | M. Maeda | Y. Kataoka | J. Boltze | A. Taguchi | Y. Ogawa | Carsten Claussen | Y. Takeuchi | A. Kikuchi-Taura | Yuka Okinaka | O. Saino | Rie Akamatsu | Orie Saino
[1] M. Shimaoka,et al. Endothelial Connexin-Integrin Crosstalk in Vascular Inflammation. , 2021, Biochimica et biophysica acta. Molecular basis of disease.
[2] S. Gul,et al. Gap Junction-Mediated Cell-Cell Interaction Between Transplanted Mesenchymal Stem Cells and Vascular Endothelium in Stroke , 2021, Stem cells.
[3] A. Taguchi,et al. Intravenous Bone Marrow Mononuclear Cells Transplantation Improves the Effect of Training in Chronic Stroke Mice , 2020, Frontiers in Medicine.
[4] G. Ferrara,et al. Metabolic determinants of leukocyte pathogenicity in neurological diseases , 2020, Journal of neurochemistry.
[5] M. Mattson,et al. Brain energy rescue: an emerging therapeutic concept for neurodegenerative disorders of ageing , 2020, Nature Reviews Drug Discovery.
[6] Carly E. Whyte,et al. Microglia Require CD4 T Cells to Complete the Fetal-to-Adult Transition , 2020, Cell.
[7] S. Gul,et al. Intravenous Bone Marrow Mononuclear Cells Transplantation in Aged Mice Increases Transcription of Glucose Transporter 1 and Na+/K+-ATPase at Hippocampus Followed by Restored Neurological Functions , 2020, Frontiers in Aging Neuroscience.
[8] S. Gul,et al. Bone Marrow Mononuclear Cells Activate Angiogenesis via Gap Junction–Mediated Cell-Cell Interaction , 2020, Stroke.
[9] D. Bennett,et al. Human Hippocampal Neurogenesis Persists in Aged Adults and Alzheimer's Disease Patients. , 2019, Cell stem cell.
[10] Jesús Ávila,et al. Adult hippocampal neurogenesis is abundant in neurologically healthy subjects and drops sharply in patients with Alzheimer’s disease , 2019, Nature Medicine.
[11] Yanguo Xin,et al. Function of Connexins in the Interaction between Glial and Vascular Cells in the Central Nervous System and Related Neurological Diseases , 2018, Neural plasticity.
[12] S. Villeda,et al. The systemic environment: at the interface of aging and adult neurogenesis , 2017, Cell and Tissue Research.
[13] D. Cangelosi,et al. Immunohistochemical analysis of PDK1, PHD3 and HIF-1α expression defines the hypoxic status of neuroblastoma tumors , 2017, PloS one.
[14] J. Kipnis,et al. How and why do T cells and their derived cytokines affect the injured and healthy brain? , 2017, Nature Reviews Neuroscience.
[15] Gang-jian Luo,et al. Connexin 43 expressed in endothelial cells modulates monocyte‑endothelial adhesion by regulating cell adhesion proteins. , 2015, Molecular medicine reports.
[16] Wei Li,et al. HIF-1α pathway: role, regulation and intervention for cancer therapy , 2015, Acta pharmaceutica Sinica. B.
[17] A. Belousov,et al. Neuronal gap junction coupling as the primary determinant of the extent of glutamate-mediated excitotoxicity , 2014, Journal of Neural Transmission.
[18] D. Stern,et al. A highly reproducible model of cerebral ischemia/reperfusion with extended survival in CB-17 mice , 2013, Neuroscience Research.
[19] M. Suematsu,et al. Regulation of glycolysis by Pdk functions as a metabolic checkpoint for cell cycle quiescence in hematopoietic stem cells. , 2013, Cell stem cell.
[20] A. Doi,et al. Bone marrow mononuclear cells promote proliferation of endogenous neural stem cells through vascular niches after cerebral infarction , 2010, Neuroscience Research.
[21] H. Moriwaki,et al. Circulating CD34-Positive Cells Provide a Marker of Vascular Risk Associated with Cognitive Impairment , 2008, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[22] D. Laird,et al. Life cycle of connexins in health and disease. , 2006, The Biochemical journal.
[23] G. Semenza,et al. HIF-1-mediated expression of pyruvate dehydrogenase kinase: a metabolic switch required for cellular adaptation to hypoxia. , 2006, Cell metabolism.
[24] H. Naritomi,et al. Administration of CD34+ cells after stroke enhances neurogenesis via angiogenesis in a mouse model. , 2004, The Journal of clinical investigation.
[25] Takuya Hayashi,et al. Circulating CD34-Positive Cells Provide an Index of Cerebrovascular Function , 2004, Circulation.
[26] L. M. Roberts,et al. Regulation of Connexin Degradation as a Mechanism to Increase Gap Junction Assembly and Function* , 2000, The Journal of Biological Chemistry.
[27] R. Stockley,et al. Use of an enzyme-linked immunosorbent assay to assess penetration of amoxicillin into lung secretions , 1992, Antimicrobial Agents and Chemotherapy.
[28] P. Wallace,et al. Methodological Considerations for Implementation of Lymphocyte Subset Analysis in a Clinical Reference Laboratory , 1986, Annals of the New York Academy of Sciences.
[29] M. Chanson,et al. Connexins in atherosclerosis. , 2013, Biochimica et biophysica acta.
[30] P. Sorgen,et al. Gap junctions. , 2012, Comprehensive Physiology.
[31] D. Stern,et al. A Reproducible and Simple Model of Permanent Cerebral Ischemia in CB-17 and SCID Mice. , 2010, Journal of experimental stroke & translational medicine.
[32] M. Palacín,et al. Mechanisms regulating GLUT4 glucose transporter expression and glucose transport in skeletal muscle. , 2005, Acta physiologica Scandinavica.